T M Calderon
Affiliation: Albert Einstein College of Medicine
- Induction of IG9 monocyte adhesion molecule expression in smooth muscle and endothelial cells after balloon arterial injury in cholesterol-fed rabbitsT M Calderon
Department of Pathology, Albert Einstein College of Medicine, Bronx, NY, USA
Arterioscler Thromb Vasc Biol 20:1293-300. 2000..Thus, the expression of IG9 by SMCs as well as by ECs may be an important factor in the accumulation of foam cells in atherosclerotic plaque development after arterial injury...
- Mycophenolate mofetil treatment reduces atherosclerosis in the cholesterol-fed rabbitS M Greenstein
Department of Surgery, Department of Pathology, Montefiore Medical Center Albert Einstein College of Medicine, 111 East 210th Street, Bronx, New York 10467, USA
J Surg Res 91:123-9. 2000..Our results demonstrate that the administration of MMF significantly reduced neointimal SMC accumulation and plaque development in a hypercholesterolemic model of atherosclerosis...
- Infection of endothelial cells with Trypanosoma cruzi activates NF-kappaB and induces vascular adhesion molecule expressionH Huang
Department of Pathology, Albert Einstein College of Medicine, Bronx, New York 10461
Infect Immun 67:5434-40. 1999..cruzi infection may be an important factor in the inflammatory response and subsequent vascular injury and endothelial dysfunction that lead to chronic cardiomyopathy...
- Shedding of PECAM-1 during HIV infection: a potential role for soluble PECAM-1 in the pathogenesis of NeuroAIDSE A Eugenin
Department of Pathology, F727, Albert Einstein College of Medicine, 1300 Morris Park Ave, Bronx, NY 10461, USA
J Leukoc Biol 79:444-52. 2006....