Mauricio Alvarez

Summary

Affiliation: Albert Einstein College of Medicine
Country: USA

Publications

  1. pmc Cell-to-cell spread and massive vacuole formation after Cryptococcus neoformans infection of murine macrophages
    Mauricio Alvarez
    Department of Microbiology and Immunology, Albert Einstein College of Medicine, Bronx, NY 10461, USA
    BMC Immunol 8:16. 2007
  2. pmc The outcome of Cryptococcus neoformans intracellular pathogenesis in human monocytes
    Mauricio Alvarez
    Department of Microbiology and Immunology and Medicine, Albert Einstein College of Medicine, Bronx, NY 10461, USA
    BMC Microbiol 9:51. 2009
  3. pmc The outcome of phagocytic cell division with infectious cargo depends on single phagosome formation
    Yong Luo
    Department of Microbiology and Immunology, Albert Einstein College of Medicine, Bronx, New York, United States of America
    PLoS ONE 3:e3219. 2008
  4. ncbi request reprint Phagosome extrusion and host-cell survival after Cryptococcus neoformans phagocytosis by macrophages
    Mauricio Alvarez
    Department of Microbiology and Immunology, Albert Einstein College of Medicine, Bronx, New York, 10461, USA
    Curr Biol 16:2161-5. 2006
  5. pmc The relative susceptibility of mouse strains to pulmonary Cryptococcus neoformans infection is associated with pleiotropic differences in the immune response
    Oscar Zaragoza
    Departments of Microbiology and Immunology and Medicine, Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, NY 10461, USA
    Infect Immun 75:2729-39. 2007
  6. doi request reprint Antibody action after phagocytosis promotes Cryptococcus neoformans and Cryptococcus gattii macrophage exocytosis with biofilm-like microcolony formation
    Mauricio Alvarez
    Department of Medicine, Albert Einstein College of Medicine, 1300 Morris Park Ave, Bronx, NY 10461, USA
    Cell Microbiol 10:1622-33. 2008
  7. pmc Phagocytosis of Cryptococcus neoformans by, and nonlytic exocytosis from, Acanthamoeba castellanii
    Cara J Chrisman
    Department of Microbiology and Immunology, Albert Einstein College of Medicine, Bronx, New York 10461, USA
    Appl Environ Microbiol 76:6056-62. 2010
  8. ncbi request reprint An innate immune system cell is a major determinant of species-related susceptibility differences to fungal pneumonia
    Xiuping Shao
    Department of Pediatrics, Albert Einstein College of Medicine, Bronx, NY 10461, USA
    J Immunol 175:3244-51. 2005
  9. ncbi request reprint Structural and functional characterization of glycosylation in an immunoglobulin G1 to Cryptococcus neoformans glucuronoxylomannan
    Fang Wang
    Laboratory for Macromolecular Analysis and Proteomics, Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, NY 10461, USA
    Mol Immunol 43:987-98. 2006
  10. pmc Binding of the wheat germ lectin to Cryptococcus neoformans suggests an association of chitinlike structures with yeast budding and capsular glucuronoxylomannan
    Marcio L Rodrigues
    Instituto de Microbiologia Professor Paulo de Goes, Universidade Federal do Rio de Janeiro, 21941590, RJ, Brazil
    Eukaryot Cell 7:602-9. 2008

Collaborators

Detail Information

Publications11

  1. pmc Cell-to-cell spread and massive vacuole formation after Cryptococcus neoformans infection of murine macrophages
    Mauricio Alvarez
    Department of Microbiology and Immunology, Albert Einstein College of Medicine, Bronx, NY 10461, USA
    BMC Immunol 8:16. 2007
    ..In this report we have used time-lapse imaging to determine if this occurs with Cn...
  2. pmc The outcome of Cryptococcus neoformans intracellular pathogenesis in human monocytes
    Mauricio Alvarez
    Department of Microbiology and Immunology and Medicine, Albert Einstein College of Medicine, Bronx, NY 10461, USA
    BMC Microbiol 9:51. 2009
    ..In this study we extended studies of C. neoformans-macrophage cellular interaction/s to human peripheral blood monocytes...
  3. pmc The outcome of phagocytic cell division with infectious cargo depends on single phagosome formation
    Yong Luo
    Department of Microbiology and Immunology, Albert Einstein College of Medicine, Bronx, New York, United States of America
    PLoS ONE 3:e3219. 2008
    ..Post-mitotic infectious cargo distribution is a new parameter to consider in the study of intracellular pathogens since it could potentially define the outcome of phagocytic-microbial interactions...
  4. ncbi request reprint Phagosome extrusion and host-cell survival after Cryptococcus neoformans phagocytosis by macrophages
    Mauricio Alvarez
    Department of Microbiology and Immunology, Albert Einstein College of Medicine, Bronx, New York, 10461, USA
    Curr Biol 16:2161-5. 2006
    ....
  5. pmc The relative susceptibility of mouse strains to pulmonary Cryptococcus neoformans infection is associated with pleiotropic differences in the immune response
    Oscar Zaragoza
    Departments of Microbiology and Immunology and Medicine, Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, NY 10461, USA
    Infect Immun 75:2729-39. 2007
    ....
  6. doi request reprint Antibody action after phagocytosis promotes Cryptococcus neoformans and Cryptococcus gattii macrophage exocytosis with biofilm-like microcolony formation
    Mauricio Alvarez
    Department of Medicine, Albert Einstein College of Medicine, 1300 Morris Park Ave, Bronx, NY 10461, USA
    Cell Microbiol 10:1622-33. 2008
    ..This finding suggests that antibody agglutination effects persist in the phagosome to entangle nascent daughter cells and this phenomenon may contribute to antibody-mediated protection...
  7. pmc Phagocytosis of Cryptococcus neoformans by, and nonlytic exocytosis from, Acanthamoeba castellanii
    Cara J Chrisman
    Department of Microbiology and Immunology, Albert Einstein College of Medicine, Bronx, New York 10461, USA
    Appl Environ Microbiol 76:6056-62. 2010
    ..These results provide additional support for the notion that some mechanisms of virulence observed during mammalian infection originated, and were selected for, by environmental interactions...
  8. ncbi request reprint An innate immune system cell is a major determinant of species-related susceptibility differences to fungal pneumonia
    Xiuping Shao
    Department of Pediatrics, Albert Einstein College of Medicine, Bronx, NY 10461, USA
    J Immunol 175:3244-51. 2005
    ..In summary, differences in AM function in rats and mice suggest an explanation for the species differences in susceptibility to C. neoformans based on the inherent efficacy of a central effector cell of the innate immune system...
  9. ncbi request reprint Structural and functional characterization of glycosylation in an immunoglobulin G1 to Cryptococcus neoformans glucuronoxylomannan
    Fang Wang
    Laboratory for Macromolecular Analysis and Proteomics, Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, NY 10461, USA
    Mol Immunol 43:987-98. 2006
    ..The results imply that the contribution of N-glycan structure to immunoglobulin function varies depending on the Ag-Ab system...
  10. pmc Binding of the wheat germ lectin to Cryptococcus neoformans suggests an association of chitinlike structures with yeast budding and capsular glucuronoxylomannan
    Marcio L Rodrigues
    Instituto de Microbiologia Professor Paulo de Goes, Universidade Federal do Rio de Janeiro, 21941590, RJ, Brazil
    Eukaryot Cell 7:602-9. 2008
    ..neoformans, which may represent a new mechanism by which the capsular polysaccharide interacts with the cell wall and is rearranged during replication...
  11. pmc Vesicular polysaccharide export in Cryptococcus neoformans is a eukaryotic solution to the problem of fungal trans-cell wall transport
    Marcio L Rodrigues
    Laboratorio de Estudos Integrados em Bioquimica Microbiana, Instituto de Microbiologia Professor Paulo de Goes, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21941590, Brazil
    Eukaryot Cell 6:48-59. 2007
    ..The results imply a novel mechanism for the release of the major virulence factor of C. neoformans whereby polysaccharide packaged in lipid vesicles crosses the cell wall and the capsule network to reach the extracellular environment...