Derya Unutmaz

Summary

Publications

  1. ncbi RORC2: the master of human Th17 cell programming
    Derya Unutmaz
    Department of Microbiology, Department of Pathology, NYU School of Medicine, NY, USA
    Eur J Immunol 39:1452-5. 2009
  2. ncbi Halofuginone inhibits TH17 cell differentiation by activating the amino acid starvation response
    Mark S Sundrud
    Department of Pathology, Harvard Medical School and Immune Disease Institute, Boston, MA 02115, USA
    Science 324:1334-8. 2009
  3. ncbi Cytokine signals through PI-3 kinase pathway modulate Th17 cytokine production by CCR6+ human memory T cells
    Qi Wan
    Department of Microbiology, New York University School of Medicine, New York, NY 10016, USA
    J Exp Med 208:1875-87. 2011
  4. ncbi CCR5 is a receptor for Staphylococcus aureus leukotoxin ED
    Francis Alonzo
    Department of Microbiology, New York University School of Medicine, New York, New York 10016, USA
    Nature 493:51-5. 2013
  5. ncbi Th17 cells and HIV infection
    Aimee Elhed
    Department of Microbiology, New York University School of Medicine, New York, NY 10016, USA
    Curr Opin HIV AIDS 5:146-50. 2010

Collaborators

  • Mark S Sundrud
  • Anjana Rao
  • Hanspeter Waldner
  • Aimee Elhed
  • Francis Alonzo
  • Lina Kozhaya
  • Qi Wan
  • Tamara Reyes-Robles
  • Ashley L Dumont
  • Victor J Torres
  • Nathaniel R Landau
  • Stephen A Rawlings
  • David G Myszka
  • Ivana M Djuretic
  • Radha Ramesh
  • Thaddeus J Carlson

Detail Information

Publications5

  1. ncbi RORC2: the master of human Th17 cell programming
    Derya Unutmaz
    Department of Microbiology, Department of Pathology, NYU School of Medicine, NY, USA
    Eur J Immunol 39:1452-5. 2009
    ..These findings support the notion that RORC2 is a master switch that initiates a wide range of phenotypic and functional programming during Th17 cell differentiation...
  2. ncbi Halofuginone inhibits TH17 cell differentiation by activating the amino acid starvation response
    Mark S Sundrud
    Department of Pathology, Harvard Medical School and Immune Disease Institute, Boston, MA 02115, USA
    Science 324:1334-8. 2009
    ..These results indicate that the AAR pathway is a potent and selective regulator of inflammatory T cell differentiation in vivo...
  3. ncbi Cytokine signals through PI-3 kinase pathway modulate Th17 cytokine production by CCR6+ human memory T cells
    Qi Wan
    Department of Microbiology, New York University School of Medicine, New York, NY 10016, USA
    J Exp Med 208:1875-87. 2011
    ..Furthermore, these findings suggest that ex vivo analysis of IL-17 expression may greatly underestimate the frequency and pathogenic potential of the human Th17 compartment...
  4. ncbi CCR5 is a receptor for Staphylococcus aureus leukotoxin ED
    Francis Alonzo
    Department of Microbiology, New York University School of Medicine, New York, New York 10016, USA
    Nature 493:51-5. 2013
    ..aureus infection, highlighting the importance of CCR5 targeting in S. aureus pathogenesis. Thus, depletion of CCR5(+) leukocytes by LukED suggests a new immune evasion mechanism of S. aureus that can be therapeutically targeted...
  5. ncbi Th17 cells and HIV infection
    Aimee Elhed
    Department of Microbiology, New York University School of Medicine, New York, NY 10016, USA
    Curr Opin HIV AIDS 5:146-50. 2010
    ..We discuss the recent findings on Th17 deficiency in HIV/simian immunodeficiency virus (SIV) infection and how this deficiency may impact the mucosal host defenses, potentially contributing to chronic immune activation...