Kehinde Ross

Summary

Affiliation: University of Newcastle
Country: UK

Publications

  1. pmc Agonist-induced calcium entry correlates with STIM1 translocation
    Kehinde Ross
    Dermatological Sciences, Institute of Cellular Medicine, Medical School, Newcastle University, Newcastle upon Tyne, United Kingdom
    J Cell Physiol 211:569-76. 2007
  2. pmc Inhibition of calcium-independent phospholipase A impairs agonist-induced calcium entry in keratinocytes
    K Ross
    Dermatological Sciences, Institute of Cellular Medicine, Medical School, Newcastle University, Newcastle upon Tyne, U K
    Br J Dermatol 158:31-7. 2008
  3. pmc Lysophosphatidic acid promotes cell migration through STIM1- and Orai1-mediated Ca2+(i) mobilization and NFAT2 activation
    Ralph Jans
    Dermatological Sciences, Institute of Cellular Medicine, Newcastle University, Newcastle upon Tyne, UK
    J Invest Dermatol 133:793-802. 2013
  4. ncbi request reprint Identification of threonine 66 as a functionally critical residue of the interleukin-1 receptor-associated kinase
    Kehinde Ross
    Division of Genomic Medicine, University of Sheffield, M Floor, Royal Hallamshire Hospital, Glossop Road, Sheffield S10 2JF, United Kingdom
    J Biol Chem 277:37414-21. 2002
  5. ncbi request reprint RelA control of IkappaBalpha phosphorylation: a positive feedback loop for high affinity NF-kappaB complexes
    Lin Yang
    Cell Biology Unit, Functional Genomics, Division of Genomic Medicine, The Medical School, University of Sheffield, Sheffield S10 2JF, United Kingdom
    J Biol Chem 278:30881-8. 2003

Collaborators

Detail Information

Publications5

  1. pmc Agonist-induced calcium entry correlates with STIM1 translocation
    Kehinde Ross
    Dermatological Sciences, Institute of Cellular Medicine, Medical School, Newcastle University, Newcastle upon Tyne, United Kingdom
    J Cell Physiol 211:569-76. 2007
    ..Thus by differentially regulating store-refilling after agonist-mediated depletion, LPA and UTP may exert distinct effects on the duration of STIM1 localization at the plasma membrane, and thus, on the magnitude and duration of ACE...
  2. pmc Inhibition of calcium-independent phospholipase A impairs agonist-induced calcium entry in keratinocytes
    K Ross
    Dermatological Sciences, Institute of Cellular Medicine, Medical School, Newcastle University, Newcastle upon Tyne, U K
    Br J Dermatol 158:31-7. 2008
    ..However, the mechanisms of agonist-induced calcium entry (ACE) in keratinocytes are not fully understood...
  3. pmc Lysophosphatidic acid promotes cell migration through STIM1- and Orai1-mediated Ca2+(i) mobilization and NFAT2 activation
    Ralph Jans
    Dermatological Sciences, Institute of Cellular Medicine, Newcastle University, Newcastle upon Tyne, UK
    J Invest Dermatol 133:793-802. 2013
    ..These data indicate that in conditions relevant to epidermal wound healing, LPA induces SOCE and NFAT activation through Orai1 channels and promotes cell migration through a calcineurin/NFAT2-dependent pathway...
  4. ncbi request reprint Identification of threonine 66 as a functionally critical residue of the interleukin-1 receptor-associated kinase
    Kehinde Ross
    Division of Genomic Medicine, University of Sheffield, M Floor, Royal Hallamshire Hospital, Glossop Road, Sheffield S10 2JF, United Kingdom
    J Biol Chem 277:37414-21. 2002
    ..These properties in turn determine key aspects of the signaling function of IRAK...
  5. ncbi request reprint RelA control of IkappaBalpha phosphorylation: a positive feedback loop for high affinity NF-kappaB complexes
    Lin Yang
    Cell Biology Unit, Functional Genomics, Division of Genomic Medicine, The Medical School, University of Sheffield, Sheffield S10 2JF, United Kingdom
    J Biol Chem 278:30881-8. 2003
    ..In addition, they suggest that regulation of both complex levels and affinities through phosphorylation, with effects on the system steady state, participate in selective activation of the NF-kappaB pathway...