M Peet

Summary

Affiliation: University of Sheffield
Country: UK

Publications

  1. ncbi request reprint Association of the Ban I dimorphic site at the human cytosolic phospholipase A2 gene with schizophrenia
    M Peet
    University Department of Psychiatry, Northern General Hospital, Sheffield, UK
    Psychiatr Genet 8:191-2. 1998
  2. ncbi request reprint Two double-blind placebo-controlled pilot studies of eicosapentaenoic acid in the treatment of schizophrenia
    M Peet
    Academic Department of Psychiatry, Northern General Hospital, The Longley Centre, Norwood Grange Drive, S5 7JT, Sheffield, UK
    Schizophr Res 49:243-51. 2001
  3. ncbi request reprint Plasma homovanillic acid in untreated schizophrenia--relationship with symptomatology and sex
    Z J Zhang
    Department of Biomedical Science, University of Sheffield, S10 2TN, Sheffield, UK
    J Psychiatr Res 35:23-8. 2001

Detail Information

Publications3

  1. ncbi request reprint Association of the Ban I dimorphic site at the human cytosolic phospholipase A2 gene with schizophrenia
    M Peet
    University Department of Psychiatry, Northern General Hospital, Sheffield, UK
    Psychiatr Genet 8:191-2. 1998
    ..Genetically determined alterations in phospholipase activity may thus underlie the reported abnormalities of phospholipid metabolism in schizophrenia...
  2. ncbi request reprint Two double-blind placebo-controlled pilot studies of eicosapentaenoic acid in the treatment of schizophrenia
    M Peet
    Academic Department of Psychiatry, Northern General Hospital, The Longley Centre, Norwood Grange Drive, S5 7JT, Sheffield, UK
    Schizophr Res 49:243-51. 2001
    ..It is concluded that EPA may represent a new treatment approach to schizophrenia, and this requires investigation by large-scale placebo-controlled trials...
  3. ncbi request reprint Plasma homovanillic acid in untreated schizophrenia--relationship with symptomatology and sex
    Z J Zhang
    Department of Biomedical Science, University of Sheffield, S10 2TN, Sheffield, UK
    J Psychiatr Res 35:23-8. 2001
    ..Such neuronal deficits of developmental or degenerative origin may thus result in an elevation/disinhibition of central dopamine metabolism in schizophrenia...