M Lastowska

Summary

Affiliation: University of Newcastle
Country: UK

Publications

  1. ncbi Comprehensive genetic and histopathologic study reveals three types of neuroblastoma tumors
    M Lastowska
    Human Genetics Unit, School of Biochemistry and Genetics, University of Newcastle upon Tyne, United Kingdom
    J Clin Oncol 19:3080-90. 2001
  2. ncbi Breakpoint position on 17q identifies the most aggressive neuroblastoma tumors
    Maria Łastowska
    Institute of Human Genetics, International Centre for Life, University of Newcastle upon Tyne, Central Parkway, Newcastle upon Tyne NE1 3BZ, UK
    Genes Chromosomes Cancer 34:428-36. 2002
  3. ncbi Regions syntenic to human 17q are gained in mouse and rat neuroblastoma
    Maria Łastowska
    Institute of Human Genetics, International Centre for Life, University of Newcastle upon Tyne, Newcastle upon Tyne, United Kingdom
    Genes Chromosomes Cancer 40:158-63. 2004
  4. ncbi 17q gain in neuroblastoma predicts adverse clinical outcome. U.K. Cancer Cytogenetics Group and the U.K. Children's Cancer Study Group
    N Bown
    Department of Human Genetics, University of Newcastle upon Tyne, United Kingdom
    Med Pediatr Oncol 36:14-9. 2001
  5. ncbi Gain of chromosome arm 17q and adverse outcome in patients with neuroblastoma
    N Bown
    Department of Human Genetics, University of Newcastle upon Tyne, United Kingdom
    N Engl J Med 340:1954-61. 1999
  6. ncbi Molecular cytogenetic definition of 17q translocation breakpoints in neuroblastoma
    M Lastowska
    Department of Human Genetics, University of Newcastle upon Tyne, United Kingdom
    Med Pediatr Oncol 36:20-3. 2001

Collaborators

Detail Information

Publications6

  1. ncbi Comprehensive genetic and histopathologic study reveals three types of neuroblastoma tumors
    M Lastowska
    Human Genetics Unit, School of Biochemistry and Genetics, University of Newcastle upon Tyne, United Kingdom
    J Clin Oncol 19:3080-90. 2001
    ..To determine the relationship between multiple genetic features, tumor morphology, and prognosis in neuroblastoma...
  2. ncbi Breakpoint position on 17q identifies the most aggressive neuroblastoma tumors
    Maria Łastowska
    Institute of Human Genetics, International Centre for Life, University of Newcastle upon Tyne, Central Parkway, Newcastle upon Tyne NE1 3BZ, UK
    Genes Chromosomes Cancer 34:428-36. 2002
    ..1 and 17qter acting to promote tumor progression...
  3. ncbi Regions syntenic to human 17q are gained in mouse and rat neuroblastoma
    Maria Łastowska
    Institute of Human Genetics, International Centre for Life, University of Newcastle upon Tyne, Newcastle upon Tyne, United Kingdom
    Genes Chromosomes Cancer 40:158-63. 2004
    ..2 Mb from the previously defined region of 17q gain in humans as a likely location of the candidate gene or genes, and strongly suggests that the molecular etiology of neuroblastoma is similar in all three species...
  4. ncbi 17q gain in neuroblastoma predicts adverse clinical outcome. U.K. Cancer Cytogenetics Group and the U.K. Children's Cancer Study Group
    N Bown
    Department of Human Genetics, University of Newcastle upon Tyne, United Kingdom
    Med Pediatr Oncol 36:14-9. 2001
    ..We sought to further investigate the clinical and prognostic associations of chromosome 17 status in relation to other well-established predictive factors...
  5. ncbi Gain of chromosome arm 17q and adverse outcome in patients with neuroblastoma
    N Bown
    Department of Human Genetics, University of Newcastle upon Tyne, United Kingdom
    N Engl J Med 340:1954-61. 1999
    ..We investigated these associations and evaluated the prognostic importance of the status of chromosome 17...
  6. ncbi Molecular cytogenetic definition of 17q translocation breakpoints in neuroblastoma
    M Lastowska
    Department of Human Genetics, University of Newcastle upon Tyne, United Kingdom
    Med Pediatr Oncol 36:20-3. 2001
    ..CONCLUSION: These results suggest that the dosage of a gene, or genes, in 17q22-qter is responsible for the clinical effects of 17q gain, rather than the disruption of a specific gene...