Research Topics



Affiliation: University of Cambridge
Country: UK


  1. Frost S, Volz E. Viral phylodynamics and the search for an 'effective number of infections'. Philos Trans R Soc Lond B Biol Sci. 2010;365:1879-90 pubmed publisher
    ..This has important implications when integrating phylogenetic data in the context of other epidemiological data. ..
  2. Frost S, Volz E. Modelling tree shape and structure in viral phylodynamics. Philos Trans R Soc Lond B Biol Sci. 2013;368:20120208 pubmed publisher
  3. Dennis A, Volz E, FROST S, Hossain A, Poon A, Rebeiro P, et al. HIV-1 transmission clustering and phylodynamics highlight the important role of young men who have sex with men. AIDS Res Hum Retroviruses. 2018;: pubmed publisher
    ..Detecting active clusters could help direct interventions to disrupt ongoing transmission chains. ..
  4. Frost S, Pybus O, Gog J, Viboud C, Bonhoeffer S, Bedford T. Eight challenges in phylodynamic inference. Epidemics. 2015;10:88-92 pubmed publisher
    ..An additional challenge exists in making efficient inferences from an ever increasing corpus of sequence data. ..
  5. Drumright L, Frost S, Elliot A, Catchpole M, Pebody R, Atkins M, et al. Assessing the use of hospital staff influenza-like absence (ILA) for enhancing hospital preparedness and national surveillance. BMC Infect Dis. 2015;15:110 pubmed publisher
    ..Hospital-ILA appears to have the potential to be a robust, yet simple syndromic surveillance method that could be used to enhance estimates of disease burden and early warning, and assist with local hospital preparedness. ..
  6. Eames K, Bansal S, Frost S, Riley S. Six challenges in measuring contact networks for use in modelling. Epidemics. 2015;10:72-7 pubmed publisher
    ..Finally, we consider the privacy and ethical issues surrounding the collection of contact network data. ..
  7. Brayne A, Dearlove B, Lester J, Kosakovsky Pond S, Frost S. Genotype-Specific Evolution of Hepatitis E Virus. J Virol. 2017;91: pubmed publisher
    ..Genotype 1, which infects humans only, appears to be evolving differently from genotypes 3 and 4, which infect multiple species, possibly because genotypes 3 and 4 are unable to achieve the same fitness due to repeated host jumps. ..