T G Allen

Summary

Affiliation: University College London
Country: UK

Publications

  1. pmc The role of N-, Q- and R-type Ca2+ channels in feedback inhibition of ACh release from rat basal forebrain neurones
    T G Allen
    Department of Pharmacology, University College London, Gower Street, London WC1E 6BT, UK
    J Physiol 515:93-107. 1999
  2. pmc Detection and modulation of acetylcholine release from neurites of rat basal forebrain cells in culture
    T G Allen
    Department of Pharmacology, University College London, London, UK
    J Physiol 492:453-66. 1996
  3. pmc Ca2+-permeable non-NMDA glutamate receptors in rat magnocellular basal forebrain neurones
    D J Waters
    Department of Pharmacology, University College London, Gower Street, London WC1E 6BT, UK
    J Physiol 508:453-69. 1998

Collaborators

  • D J Waters

Detail Information

Publications3

  1. pmc The role of N-, Q- and R-type Ca2+ channels in feedback inhibition of ACh release from rat basal forebrain neurones
    T G Allen
    Department of Pharmacology, University College London, Gower Street, London WC1E 6BT, UK
    J Physiol 515:93-107. 1999
    ....
  2. pmc Detection and modulation of acetylcholine release from neurites of rat basal forebrain cells in culture
    T G Allen
    Department of Pharmacology, University College London, London, UK
    J Physiol 492:453-66. 1996
    ..It is suggested that the latter results from inhibition of presynaptic Ca(2+) channels and that it might be responsible for feedback autoinhibition of ACh release from cortical afferents of nucleus basalis neurones in vivo...
  3. pmc Ca2+-permeable non-NMDA glutamate receptors in rat magnocellular basal forebrain neurones
    D J Waters
    Department of Pharmacology, University College London, Gower Street, London WC1E 6BT, UK
    J Physiol 508:453-69. 1998
    ..7. Ca2+ permeability of many of the non-NMDA receptors expressed by magnocellular basal forebrain neurones may underlie the unusual sensitivity of cholinergic basal forebrain neurones to non-NMDA receptor-mediated excitotoxicity...