S F Cooke

Summary

Affiliation: National Institute for Medical Research
Country: UK

Publications

  1. ncbi request reprint Long-term potentiation and cognitive drug discovery
    Sam F Cooke
    National Institute for Medical Research, London, UK
    Curr Opin Investig Drugs 6:25-34. 2005
  2. ncbi request reprint Plasticity in the human central nervous system
    S F Cooke
    Division of Neurophysiology, National Institute for Medical Research London, UK
    Brain 129:1659-73. 2006
  3. pmc Autophosphorylation of alphaCaMKII is not a general requirement for NMDA receptor-dependent LTP in the adult mouse
    Sam F Cooke
    MRC National Institute for Medical Research, Mill Hill, London NW7 1AA, UK
    J Physiol 574:805-18. 2006
  4. pmc Preservation of long-term memory and synaptic plasticity despite short-term impairments in the Tc1 mouse model of Down syndrome
    Elise Morice
    Division of Neurophysiology, National Institute for Medical Research, London NW7 1AA, United Kingdom
    Learn Mem 15:492-500. 2008
  5. ncbi request reprint The genetic enhancement of memory
    S F Cooke
    Division of Neurophysiology, National Institute for Medical Research, Mill Hill, London NW7 1AA, United Kingdom
    Cell Mol Life Sci 60:1-5. 2003
  6. ncbi request reprint Synaptic plasticity, memory and the hippocampus: a neural network approach to causality
    Guilherme Neves
    Division of Neurophysiology, Medical Research Council National Institute for Medical Research, Mill Hill, London, NW7 1AA, UK
    Nat Rev Neurosci 9:65-75. 2008
  7. ncbi request reprint Arc/Arg3.1 is essential for the consolidation of synaptic plasticity and memories
    Niels Plath
    Molecular Neurobiology, Department of Biology Chemistry Pharmacy, Freie Universitat Berlin, 14195 Berlin, Germany
    Neuron 52:437-44. 2006
  8. ncbi request reprint Glycogen synthase kinase-3 inhibition is integral to long-term potentiation
    Claudie Hooper
    MRC Centre for Neurodegeneration Research, Centre for the Cellular Basis of Behaviour, Institute of Psychiatry, King s College London, De Crespigny Park, London SE5 8AF, UK
    Eur J Neurosci 25:81-6. 2007

Collaborators

  • Tim Bliss
  • K P Giese
  • Robert Waltereit
  • Kobi Rosenblum
  • S G N Grant
  • J Avila
  • Florian Plattner
  • Elise Morice
  • Guilherme Neves
  • Claudie Hooper
  • Niels Plath
  • Felix Hernandez
  • John Stephenson
  • Simon Lovestone
  • Richard Killick
  • Tobias Engel
  • Emma Schofield
  • Jose J Lucas
  • Brian Anderton
  • Vladimir Markevich
  • Anastasia Stawrakakis
  • Esperanza Fernandez
  • Anika Bick-Sander
  • Dietmar Schmitz
  • Hans Peter Lipp
  • Björn Dammermann
  • Veronique Blanquet
  • Benedikt Salmen
  • Mick L Errington
  • Arne Engelsberg
  • Xiaosong Mao
  • Hans Welzl
  • Claudia Mahlke
  • Ora Ohana
  • Dietmar Kuhl
  • David P Wolfer
  • Ursula Kobalz
  • Christina Gross
  • Eric Therstappen
  • Wolfgang Wurst
  • Michael R Bösl

Detail Information

Publications8

  1. ncbi request reprint Long-term potentiation and cognitive drug discovery
    Sam F Cooke
    National Institute for Medical Research, London, UK
    Curr Opin Investig Drugs 6:25-34. 2005
    ..In this review we describe the contributions of pharmacology to the field of synaptic plasticity, and also discuss the role of LTP in developing potential nootropic drugs to enhance learning and memory...
  2. ncbi request reprint Plasticity in the human central nervous system
    S F Cooke
    Division of Neurophysiology, National Institute for Medical Research London, UK
    Brain 129:1659-73. 2006
    ..These techniques have potential therapeutic application in manipulating neural plasticity to treat a variety of conditions, including depression, Parkinson's disease, epilepsy and neuropathic pain...
  3. pmc Autophosphorylation of alphaCaMKII is not a general requirement for NMDA receptor-dependent LTP in the adult mouse
    Sam F Cooke
    MRC National Institute for Medical Research, Mill Hill, London NW7 1AA, UK
    J Physiol 574:805-18. 2006
    ..Thus, the autophosphorylation of alphaCaMKII is not a general requirement for NMDA receptor-dependent LTP in the adult mouse...
  4. pmc Preservation of long-term memory and synaptic plasticity despite short-term impairments in the Tc1 mouse model of Down syndrome
    Elise Morice
    Division of Neurophysiology, National Institute for Medical Research, London NW7 1AA, United Kingdom
    Learn Mem 15:492-500. 2008
    ..Our observations also provide further evidence that STM and LTM for hippocampus-dependent tasks are subserved by parallel processing streams...
  5. ncbi request reprint The genetic enhancement of memory
    S F Cooke
    Division of Neurophysiology, National Institute for Medical Research, Mill Hill, London NW7 1AA, United Kingdom
    Cell Mol Life Sci 60:1-5. 2003
  6. ncbi request reprint Synaptic plasticity, memory and the hippocampus: a neural network approach to causality
    Guilherme Neves
    Division of Neurophysiology, Medical Research Council National Institute for Medical Research, Mill Hill, London, NW7 1AA, UK
    Nat Rev Neurosci 9:65-75. 2008
    ..In the process, the hypothesis that synaptic plasticity is necessary and sufficient for information storage in the brain may finally be validated...
  7. ncbi request reprint Arc/Arg3.1 is essential for the consolidation of synaptic plasticity and memories
    Niels Plath
    Molecular Neurobiology, Department of Biology Chemistry Pharmacy, Freie Universitat Berlin, 14195 Berlin, Germany
    Neuron 52:437-44. 2006
    ..In addition, long-term depression is significantly impaired. Together, these results demonstrate a critical role for Arc/Arg3.1 in the consolidation of enduring synaptic plasticity and memory storage...
  8. ncbi request reprint Glycogen synthase kinase-3 inhibition is integral to long-term potentiation
    Claudie Hooper
    MRC Centre for Neurodegeneration Research, Centre for the Cellular Basis of Behaviour, Institute of Psychiatry, King s College London, De Crespigny Park, London SE5 8AF, UK
    Eur J Neurosci 25:81-6. 2007
    ..It follows that this role of GSK-3beta in LTP might underlie some of the cognitive dysfunction in diseases where GSK-3 dysfunction has been implicated, including Alzheimer's and other dementias...