M G Robson

Summary

Affiliation: King's College London
Country: UK

Publications

  1. pmc Toll-like receptor 2 or toll-like receptor 4 deficiency does not modify lupus in MRLlpr mice
    Simon J Freeley
    MRC Centre for Transplantation King s College London, London, United Kingdom
    PLoS ONE 8:e74112. 2013
  2. pmc Antibody-mediated glomerulonephritis in mice: the role of endotoxin, complement and genetic background
    M G Robson
    Division of Medicine and Department Histopathology, Faculty of Medicine, Imperial College, Hammersmith Hospital, London, UK
    Clin Exp Immunol 133:326-33. 2003
  3. doi request reprint Toll-like receptors and renal disease
    Michael G Robson
    Medical Research Council Centre for Transplantation, Kings College London School of Medicine, Guy s Hospital, London SE1 9RT, UK
    Nephron Exp Nephrol 113:e1-7. 2009
  4. ncbi request reprint Accelerated nephrotoxic nephritis is exacerbated in C1q-deficient mice
    M G Robson
    Division of Medicine, Imperial College School of Science, Technology, and Medicine, Hammersmith Hospital, Du Cane Road, London W12 0NN, United Kingdom
    J Immunol 166:6820-8. 2001
  5. pmc Animal models of anti-neutrophil cytoplasmic antibody-associated vasculitis
    A M Coughlan
    King s College London, King s Health Partners, Medical Research Council MRC Centre for Transplantation, Guy s Hospital, London SE1 9RT, UK
    Clin Exp Immunol 169:229-37. 2012

Collaborators

Detail Information

Publications5

  1. pmc Toll-like receptor 2 or toll-like receptor 4 deficiency does not modify lupus in MRLlpr mice
    Simon J Freeley
    MRC Centre for Transplantation King s College London, London, United Kingdom
    PLoS ONE 8:e74112. 2013
    ..These results show that Toll-like receptor 2 and Toll-like receptor 4 do not play a significant role in MRLlpr mice, and therefore they may not be important in human lupus. ..
  2. pmc Antibody-mediated glomerulonephritis in mice: the role of endotoxin, complement and genetic background
    M G Robson
    Division of Medicine and Department Histopathology, Faculty of Medicine, Imperial College, Hammersmith Hospital, London, UK
    Clin Exp Immunol 133:326-33. 2003
    ..C1q may play a protective role in mixed-strain 129/Sv x C57BL/6 mice, but the data may also be explained by systematic bias in background genes, as there is a large difference in disease susceptibility between C57BL/6 and 129/Sv mice...
  3. doi request reprint Toll-like receptors and renal disease
    Michael G Robson
    Medical Research Council Centre for Transplantation, Kings College London School of Medicine, Guy s Hospital, London SE1 9RT, UK
    Nephron Exp Nephrol 113:e1-7. 2009
    ..I also discuss evidence that they are important in other forms of glomerulonephritis, with evidence derived mainly from experimental models in which exogenous ligands have been administered...
  4. ncbi request reprint Accelerated nephrotoxic nephritis is exacerbated in C1q-deficient mice
    M G Robson
    Division of Medicine, Imperial College School of Science, Technology, and Medicine, Hammersmith Hospital, Du Cane Road, London W12 0NN, United Kingdom
    J Immunol 166:6820-8. 2001
    ..The increased IgG deposits and apoptotic cells in the glomeruli of C1q-deficient mice suggest that the exacerbation of disease may be due to a defect in the clearance of immune complexes and/or apoptotic cells from their kidneys...
  5. pmc Animal models of anti-neutrophil cytoplasmic antibody-associated vasculitis
    A M Coughlan
    King s College London, King s Health Partners, Medical Research Council MRC Centre for Transplantation, Guy s Hospital, London SE1 9RT, UK
    Clin Exp Immunol 169:229-37. 2012
    ..Other potential therapies studied in these models include the use of bortezemib and strategies to modify antibody glycosylation...