Nicholas G Jones

Summary

Affiliation: King's College London
Country: UK

Publications

  1. ncbi Acid-induced pain and its modulation in humans
    Nicholas G Jones
    Sensory Function Group, Center for Neuroscience, King s College London, London, SE1 1UL, United Kingdom
    J Neurosci 24:10974-9. 2004
  2. ncbi Plasticity of pain signaling: role of neurotrophic factors exemplified by acid-induced pain
    Stephen B McMahon
    London Pain Consortium, Kings College London, Center for Neuroscience Research, Hodgkin Building, London Bridge, London SE1 1UL, UK
    J Neurobiol 61:72-87. 2004
  3. ncbi Modulation of acid-sensing ion channel activity by nitric oxide
    Herve Cadiou
    Department of Pharmacology, University of Cambridge, Cambridge CB2 1PD, United Kingdom
    J Neurosci 27:13251-60. 2007

Collaborators

Detail Information

Publications3

  1. ncbi Acid-induced pain and its modulation in humans
    Nicholas G Jones
    Sensory Function Group, Center for Neuroscience, King s College London, London, SE1 1UL, United Kingdom
    J Neurosci 24:10974-9. 2004
    ..Together these data suggest a prominent role for ASIC channels and only a minor role for transient receptor potential vanilloid receptor-1 as mediators of cutaneous acid-induced pain...
  2. ncbi Plasticity of pain signaling: role of neurotrophic factors exemplified by acid-induced pain
    Stephen B McMahon
    London Pain Consortium, Kings College London, Center for Neuroscience Research, Hodgkin Building, London Bridge, London SE1 1UL, UK
    J Neurobiol 61:72-87. 2004
    ..We review the neurobiology of such pain states, and discuss how acid stimuli both initiate sensitization and how the neuronal processing of acid stimuli is subject to sensitization...
  3. ncbi Modulation of acid-sensing ion channel activity by nitric oxide
    Herve Cadiou
    Department of Pharmacology, University of Cambridge, Cambridge CB2 1PD, United Kingdom
    J Neurosci 27:13251-60. 2007
    ..ASICs may therefore play an important role in the pain associated with metabolic stress and inflammation, where both tissue acidosis and a high level of NO are present...