Affiliation: King's College London
- Subdural hematoma (SDH): assessment of macrophage reactivity within the dura mater and underlying hematomaS Al-Sarraj
Department of Clinical Neuropathology, King s College, London, UK
Clin Neuropathol 23:62-75. 2004..This study investigated the progressive and orderly pattern of reactivity of resident and infiltrating dural macrophages that occurs in response to injury associated with SDH...
- Ubiquitin-only intraneuronal inclusion in the substantia nigra is a characteristic feature of motor neurone disease with dementiaS Al-Sarraj
Department of Neuropathology and Neurology, King s College Hospital Institute of Psychiatry, King s College London, London, UK
Neuropathol Appl Neurobiol 28:120-8. 2002..MND disease is a spectrum and multisystem disorder with DMND located at the extreme end of a spectrum affecting the CNS more widely than just the motor system...
- Cortical selective vulnerability in motor neuron disease: a morphometric studyS Maekawa
Department of Neurology, Institute of Psychiatry, De Crespigny Park, London SE5 8AF, UK
Brain 127:1237-51. 2004..These findings support the notion that MND should be considered a multisystem disorder...
- Ultrastructural examination is essential for diagnosis of papillary meningiomaS Al-Sarraj
Department of Neuropathology, Neuroscience Centre, King s College Hospital, Institute of Psychiatry, London, UK
Histopathology 38:318-24. 2001..Papillary meningioma is a rare meningeal tumour. To date only a few cases have been reported and their immunohistochemical features have not been fully documented...
- Spinal meningioma after treatment for Hodgkin disease. Case reportA J Martin
Department of Neurosurgery, King s College Hospital, London, United Kingdom
J Neurosurg 95:232-5. 2001..Whereas radiation-induced intracranial meningiomas are well described in the literature, treatment-induced meningiomas of the spine have not been widely recognized...
- Neuropathology of the hippocampus in FTLD-Tau with Pick bodies: a study of the BrainNet Europe ConsortiumG G Kovacs
Institute of Neurology, Medical University of Vienna, Austria, Semmelweis University Neuropathology and Prion Disease Reference Center Former Department of Neuropathology, National Institute of Psychiatry and Neurology, Budapest, Hungary, Netherlands Brain Bank and VU University Medical Center, Amsterdam, Erasmus MC, Rotterdam, The Netherlands, Clinical Neuropathology Department, King s College Hospital, King s College London, MRC Centre for Neurodegeneration Research, Department of Clinical Neuroscience, Institute of Psychiatry, London, Department of Neuropathology, John Radcliffe Hospital, Oxford, Department of Pathology, University of Edinburgh, Western General Hospital, Edinburgh, UK, Fondazione IRCCS Istituto Neurologico Carlo Besta, Milan, Italy, Institut de Neuropatologia, Hospital Universitari de Bellvitge, Universitat de Bacelona, Hospitalet de Llobregat, Barcelona, Spain, Centre for Neuropathology and Prion Research, Ludwig Maximilians University, and Neurobiobank Munich BrainNet Germany, Munich, Germany, Department of Geriatrics, Karolinska Institutet, HuddingeRudbeck Laboratory, Department of Genetics and Pathology, Uppsala University, Uppsala, SwedenDepartment of Neuroscience and Neurology, Kuopio University, Kuopio, Germany
Neuropathol Appl Neurobiol 39:166-78. 2013..In view of confusion about the molecular pathology of Pick's disease, we aimed to evaluate the spectrum of tau pathology and concomitant neurodegeneration-associated protein depositions in the characteristically affected hippocampus...
- Transportin 1 colocalization with Fused in Sarcoma (FUS) inclusions is not characteristic for amyotrophic lateral sclerosis-FUS confirming disrupted nuclear import of mutant FUS and distinguishing it from frontotemporal lobar degeneration with FUS inclusiC Troakes
KHP Centre for Neurodegeneration Research, Institute of Psychiatry Department of Clinical Neuropathology, King s College London, London, UK
Neuropathol Appl Neurobiol 39:553-61. 2013..If this held true for human ALS then we predicted that FUS inclusions in patients with C-terminal FUS mutations would not colocalize with TNPO 1...
- Frontotemporal lobar degeneration and ubiquitin immunohistochemistryK A Josephs
Department of Neurology, Mayo Clinic, Rochester, USA
Neuropathol Appl Neurobiol 30:369-73. 2004..Therefore in our brain bank series of frontotemporal degeneration, most cases were non-tauopathies with FTLD-U accounting for 62% of all the diagnoses...
- Specificity and sensitivity of betaAPP in head injuryM Lambri
Department of Neuropathology, Institute of Psychiatry, King's College Hospital, Denmark Hill, London, UK
Clin Neuropathol 20:263-71. 2001..These could be due to pathophysiological differences. The results may be helpful in differentiating head injury from hypoxia in medicolegal cases...
- A clinical and pathological study of motor neurone disease on GuamH R Morris
Neurogenetics Section, University Department of Clinical Neurology, Reta Lila Weston Institute of Neurological Sciences, University College London, London, UK
Brain 124:2215-22. 2001....
- Oculopharyngeal myopathy with inflammation and calcinosis: an unusual phenotypeT Jenkins
Department of Neurology, Kings College Hospital, Denmark Hill, London SE5 9RS, UK
J Neurol Neurosurg Psychiatry 79:731-3. 2008..The clinical phenotype fits best with hereditary inclusion body myopathy or distal-oculopharyngeal muscular dystrophy, but the degree of inflammation seen is unusual. None of these are associated with calcinosis...