Research Topics
| K L Woodford RichensSummaryAffiliation: Imperial Cancer Research Fund Country: UK Publications
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Detail Information
Publications
Allelic loss at SMAD4 in polyps from juvenile polyposis patients and use of fluorescence in situ hybridization to demonstrate clonal origin of the epitheliumK Woodford-Richens
Molecular and Population Genetics, Imperial Cancer Research Fund, London, United Kingdom
Cancer Res 60:2477-82. 2000..SMAD4/DPC4 probably acts as a "gatekeeper" tumor suppressor in juvenile polyps, and there is no need to invoke a "landscaper hypothesis."..
SMAD4 mutations in colorectal cancer probably occur before chromosomal instability, but after divergence of the microsatellite instability pathwayK L Woodford-Richens
Molecular and Population Genetics Laboratory, Imperial Cancer Research Fund, London, United Kingdom
Proc Natl Acad Sci U S A 98:9719-23. 2001..Thus, MSI(+) cancers may diverge first, followed by CIN(+) (chromosomal instability) cancers, leaving other cancers to follow a CIN(-)MSI(-) pathway...
Comprehensive analysis of SMAD4 mutations and protein expression in juvenile polyposis: evidence for a distinct genetic pathway and polyp morphology in SMAD4 mutation carriersK L Woodford-Richens
Molecular and Population Genetics Laboratory, Imperial Cancer Research Fund, London, United Kingdom
Am J Pathol 159:1293-300. 2001..The morphology of polyps from SMAD4 mutation carriers is subtly different from other JPS polyps, notably including a more prominent epithelial component in the former...
