Kim Midwood

Summary

Affiliation: Imperial College
Country: UK

Publications

  1. doi request reprint Tenascin-C is an endogenous activator of Toll-like receptor 4 that is essential for maintaining inflammation in arthritic joint disease
    Kim Midwood
    Kennedy Institute of Rheumatology Division, Faculty of Medicine, Imperial College of Science, Technology and Medicine, London, UK
    Nat Med 15:774-80. 2009
  2. doi request reprint Intrinsic danger: activation of Toll-like receptors in rheumatoid arthritis
    Fui G Goh
    Kennedy Institute of Rheumatology Division, Matrix Biology Department, Faculty of Medicine, Imperial College of Science, Technology and Medicine, 65 Aspenlea Road, Hammersmith, London W6 8LH, UK
    Rheumatology (Oxford) 51:7-23. 2012
  3. doi request reprint Myofibroblast distribution in Dupuytren's cords: correlation with digital contracture
    Liaquat Suleman Verjee
    Kennedy Institute of Rheumatology, Imperial College London, London, UK
    J Hand Surg Am 34:1785-94. 2009
  4. doi request reprint Post-transcriptional regulation of alpha-smooth muscle actin determines the contractile phenotype of Dupuytren's nodular cells
    Liaquat Suleman Verjee
    Kennedy Institute of Rheumatology, Imperial College London, London, UK
    J Cell Physiol 224:681-90. 2010
  5. doi request reprint MicroRNA epigenetics: a new avenue for wound healing research
    Luckshman Bavan
    School of Medicine, Imperial College London, London, UK
    BioDrugs 25:27-41. 2011

Collaborators

Detail Information

Publications6

  1. doi request reprint Tenascin-C is an endogenous activator of Toll-like receptor 4 that is essential for maintaining inflammation in arthritic joint disease
    Kim Midwood
    Kennedy Institute of Rheumatology Division, Faculty of Medicine, Imperial College of Science, Technology and Medicine, London, UK
    Nat Med 15:774-80. 2009
    ..Thus, we have identified tenascin-C as a novel endogenous activator of TLR4-mediated immunity that mediates persistent synovial inflammation and tissue destruction in arthritic joint disease...
  2. doi request reprint Intrinsic danger: activation of Toll-like receptors in rheumatoid arthritis
    Fui G Goh
    Kennedy Institute of Rheumatology Division, Matrix Biology Department, Faculty of Medicine, Imperial College of Science, Technology and Medicine, 65 Aspenlea Road, Hammersmith, London W6 8LH, UK
    Rheumatology (Oxford) 51:7-23. 2012
    ....
  3. doi request reprint Myofibroblast distribution in Dupuytren's cords: correlation with digital contracture
    Liaquat Suleman Verjee
    Kennedy Institute of Rheumatology, Imperial College London, London, UK
    J Hand Surg Am 34:1785-94. 2009
    ....
  4. doi request reprint Post-transcriptional regulation of alpha-smooth muscle actin determines the contractile phenotype of Dupuytren's nodular cells
    Liaquat Suleman Verjee
    Kennedy Institute of Rheumatology, Imperial College London, London, UK
    J Cell Physiol 224:681-90. 2010
    ..The contractile phenotype of Dupuytren's myofibroblasts is determined by increased alpha-SMA protein distributed in stress fibres, not by cellular mRNA levels. Dupuytren's cell contractility is not influenced by dermal fibroblasts...
  5. doi request reprint MicroRNA epigenetics: a new avenue for wound healing research
    Luckshman Bavan
    School of Medicine, Imperial College London, London, UK
    BioDrugs 25:27-41. 2011
    ..The future potential for improving wound healing outcomes using miRNA-based therapies is also discussed...