Nienke Folkeringa

Summary

Affiliation: University Medical Center Groningen
Country: The Netherlands

Publications

  1. ncbi request reprint High risk of pregnancy-related venous thromboembolism in women with multiple thrombophilic defects
    Nienke Folkeringa
    Division of Haemostasis, Thrombosis and Rheology, Department of Haematology, University Medical Centre Groningen, Groningen, The Netherlands
    Br J Haematol 138:110-6. 2007
  2. ncbi request reprint Reduction of high fetal loss rate by anticoagulant treatment during pregnancy in antithrombin, protein C or protein S deficient women
    Nienke Folkeringa
    Division of Haemostasis, Thrombosis and Rheology, Department of Haematology, University Medical Centre Groningen, Groningen, The Netherlands
    Br J Haematol 136:656-61. 2007
  3. doi request reprint Absolute risk of venous and arterial thromboembolism in thrombophilic families is not increased by high thrombin-activatable fibrinolysis inhibitor (TAFI) levels
    Nienke Folkeringa
    Division of Haemostasis, Thrombosis and Rheology, Department of Haematology, University Medical Center Groningen, Hanzeplein 1, 9713 GZ Groningen, The Netherlands
    Thromb Haemost 100:38-44. 2008
  4. doi request reprint Prevalence of parental thrombophilic defects after fetal death and relation to cause
    Fleurisca J Korteweg
    Department of Obstetrics, Division of Haemostasis, Trial Coordination Center, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands
    Obstet Gynecol 116:355-64. 2010

Detail Information

Publications4

  1. ncbi request reprint High risk of pregnancy-related venous thromboembolism in women with multiple thrombophilic defects
    Nienke Folkeringa
    Division of Haemostasis, Thrombosis and Rheology, Department of Haematology, University Medical Centre Groningen, Groningen, The Netherlands
    Br J Haematol 138:110-6. 2007
    ..In conclusion, women with hereditary deficiencies of antithrombin, protein C or protein S are at high risk of pregnancy-related venous thromboembolism. This risk is increased by multiple additional thrombophilic defects...
  2. ncbi request reprint Reduction of high fetal loss rate by anticoagulant treatment during pregnancy in antithrombin, protein C or protein S deficient women
    Nienke Folkeringa
    Division of Haemostasis, Thrombosis and Rheology, Department of Haematology, University Medical Centre Groningen, Groningen, The Netherlands
    Br J Haematol 136:656-61. 2007
    ..07 (95% confidence interval 0.001-0.7; P = 0.02). Our data suggest that anticoagulant treatment during pregnancy reduces the high fetal loss rate in women with hereditary deficiencies of antithrombin, protein C or protein S...
  3. doi request reprint Absolute risk of venous and arterial thromboembolism in thrombophilic families is not increased by high thrombin-activatable fibrinolysis inhibitor (TAFI) levels
    Nienke Folkeringa
    Division of Haemostasis, Thrombosis and Rheology, Department of Haematology, University Medical Center Groningen, Hanzeplein 1, 9713 GZ Groningen, The Netherlands
    Thromb Haemost 100:38-44. 2008
    ..None of these concomitant defects showed interaction with high TAFI levels. High TAFI levels were not associated with an increased risk of venous and arterial thromboembolism in thrombophilic families...
  4. doi request reprint Prevalence of parental thrombophilic defects after fetal death and relation to cause
    Fleurisca J Korteweg
    Department of Obstetrics, Division of Haemostasis, Trial Coordination Center, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands
    Obstet Gynecol 116:355-64. 2010
    ....