Bente C Appelhof

Summary

Affiliation: Academic Medical Center
Country: The Netherlands

Publications

  1. ncbi Triiodothyronine addition to paroxetine in the treatment of major depressive disorder
    Bente C Appelhof
    Department of Endocrinology and Metabolism, Academic Medical Center, University of Amsterdam, 1100 DE Amsterdam, The Netherlands
    J Clin Endocrinol Metab 89:6271-6. 2004
  2. ncbi Combined therapy with levothyroxine and liothyronine in two ratios, compared with levothyroxine monotherapy in primary hypothyroidism: a double-blind, randomized, controlled clinical trial
    Bente C Appelhof
    Department of Endocrinology and Metabolism, Academic Medical Center, University of Amsterdam, F5 161, P O Box 22700, 1100 DE Amsterdam, The Netherlands
    J Clin Endocrinol Metab 90:2666-74. 2005
  3. ncbi Thyrotropin, but not a polymorphism in type II deiodinase, predicts response to paroxetine in major depression
    Jantien P Brouwer
    Department of Endocrinology, Academic Medical Center, The Netherlands
    Eur J Endocrinol 154:819-25. 2006
  4. ncbi Glucocorticoids and relapse of major depression (dexamethasone/corticotropin-releasing hormone test in relation to relapse of major depression)
    Bente C Appelhof
    Department of Endocrinology and Metabolism, Academic Medical Center of the University of Amsterdam, The Netherlands
    Biol Psychiatry 59:696-701. 2006
  5. ncbi Polymorphisms in type 2 deiodinase are not associated with well-being, neurocognitive functioning, and preference for combined thyroxine/3,5,3'-triiodothyronine therapy
    Bente C Appelhof
    Department of Endocrinology and Metabolism, Academic Medical Center, University of Amsterdam, F5 171, P O Box 22700, 1100 DE Amsterdam, The Netherlands
    J Clin Endocrinol Metab 90:6296-9. 2005
  6. ncbi Cognitive functioning and well-being in euthyroid patients on thyroxine replacement therapy for primary hypothyroidism
    Ellie M Wekking
    Department of Psychiatry, Academic Medical Centre, University of Amsterdam, Amsterdam, The Netherlands
    Eur J Endocrinol 153:747-53. 2005
  7. ncbi Thyroid and adrenal axis in major depression: a controlled study in outpatients
    Jantien P Brouwer
    Department of Endocrinology, University of Amsterdam, Amsterdam, The Netherlands
    Eur J Endocrinol 152:185-91. 2005
  8. ncbi Prediction of treatment response by HPA-axis and glucocorticoid receptor polymorphisms in major depression
    Jantien P Brouwer
    Department of Endocrinology, Academic Medical Center, Meibergdreef 9, 1105 AZ Amsterdam, Netherlands
    Psychoneuroendocrinology 31:1154-63. 2006

Detail Information

Publications8

  1. ncbi Triiodothyronine addition to paroxetine in the treatment of major depressive disorder
    Bente C Appelhof
    Department of Endocrinology and Metabolism, Academic Medical Center, University of Amsterdam, 1100 DE Amsterdam, The Netherlands
    J Clin Endocrinol Metab 89:6271-6. 2004
    ..On the contrary, more adverse reactions occurred in T3-treated patients...
  2. ncbi Combined therapy with levothyroxine and liothyronine in two ratios, compared with levothyroxine monotherapy in primary hypothyroidism: a double-blind, randomized, controlled clinical trial
    Bente C Appelhof
    Department of Endocrinology and Metabolism, Academic Medical Center, University of Amsterdam, F5 161, P O Box 22700, 1100 DE Amsterdam, The Netherlands
    J Clin Endocrinol Metab 90:2666-74. 2005
    ..Decrease in body weight was associated with satisfaction with study medication...
  3. ncbi Thyrotropin, but not a polymorphism in type II deiodinase, predicts response to paroxetine in major depression
    Jantien P Brouwer
    Department of Endocrinology, Academic Medical Center, The Netherlands
    Eur J Endocrinol 154:819-25. 2006
    ....
  4. ncbi Glucocorticoids and relapse of major depression (dexamethasone/corticotropin-releasing hormone test in relation to relapse of major depression)
    Bente C Appelhof
    Department of Endocrinology and Metabolism, Academic Medical Center of the University of Amsterdam, The Netherlands
    Biol Psychiatry 59:696-701. 2006
    ..Knowledge of pathogenic mechanisms and predictors of relapse in major depressive disorder is still limited. Hypothalamic-pituitary-adrenocortical (HPA) axis dysregulation is thought to be related to the development and course of depression...
  5. ncbi Polymorphisms in type 2 deiodinase are not associated with well-being, neurocognitive functioning, and preference for combined thyroxine/3,5,3'-triiodothyronine therapy
    Bente C Appelhof
    Department of Endocrinology and Metabolism, Academic Medical Center, University of Amsterdam, F5 171, P O Box 22700, 1100 DE Amsterdam, The Netherlands
    J Clin Endocrinol Metab 90:6296-9. 2005
    ..Levothyroxine-treated patients can be assumed to derive T3 exclusively from deiodination of T4, which, in the central nervous system, is regulated by type II deiodinase (DII)...
  6. ncbi Cognitive functioning and well-being in euthyroid patients on thyroxine replacement therapy for primary hypothyroidism
    Ellie M Wekking
    Department of Psychiatry, Academic Medical Centre, University of Amsterdam, Amsterdam, The Netherlands
    Eur J Endocrinol 153:747-53. 2005
    ..We also investigated whether serum TSH and thyroid antibodies are determinants of neurocognitive functioning and well-being...
  7. ncbi Thyroid and adrenal axis in major depression: a controlled study in outpatients
    Jantien P Brouwer
    Department of Endocrinology, University of Amsterdam, Amsterdam, The Netherlands
    Eur J Endocrinol 152:185-91. 2005
    ..We therefore conducted a controlled study in unipolar depressed outpatients who had been free of antidepressants for at least 3 months...
  8. ncbi Prediction of treatment response by HPA-axis and glucocorticoid receptor polymorphisms in major depression
    Jantien P Brouwer
    Department of Endocrinology, Academic Medical Center, Meibergdreef 9, 1105 AZ Amsterdam, Netherlands
    Psychoneuroendocrinology 31:1154-63. 2006
    ..We investigated whether treatment response is predicted by hypothalamus-pituitary-adrenal (HPA) axis parameters, or by genetic polymorphisms in the glucocorticoid receptor (GR), that regulates its feedback...