Research Topics

Fabio Martinon

Summary

Affiliation: University of Lausanne
Country: Switzerland

Publications

  1. doi request reprint Did cholera toxin finally get caught?
    Fabio Martinon
    Department of Biochemistry, University of Lausanne, 155 Chemin des Boveresses, 1066 Epalinges, Switzerland
    Cell Host Microbe 13:501-3. 2013
  2. doi request reprint The endoplasmic reticulum: a sensor of cellular stress that modulates immune responses
    Fabio Martinon
    Dept of Biochemistry, University of Lausanne, 155 ch des Boveresses, Epalinges 1066, Switzerland
    Microbes Infect 14:1293-300. 2012
  3. ncbi request reprint Dangerous liaisons: mitochondrial DNA meets the NLRP3 inflammasome
    Fabio Martinon
    Department of Biochemistry, University of Lausanne, 155 ch des Boveresses, Epalinges 1066, Switzerland
    Immunity 36:313-5. 2012

Detail Information

Publications3

  1. doi request reprint Did cholera toxin finally get caught?
    Fabio Martinon
    Department of Biochemistry, University of Lausanne, 155 Chemin des Boveresses, 1066 Epalinges, Switzerland
    Cell Host Microbe 13:501-3. 2013
    ..In this issue of Cell Host & Microbe, a study by Cho et al. reveals a mechanism of immune recognition that relies on the sensing of cholera toxin within the endoplasmic reticulum...
  2. doi request reprint The endoplasmic reticulum: a sensor of cellular stress that modulates immune responses
    Fabio Martinon
    Dept of Biochemistry, University of Lausanne, 155 ch des Boveresses, Epalinges 1066, Switzerland
    Microbes Infect 14:1293-300. 2012
    ..These pathways, primarily associated with loss of ER homeostasis, are emerging as key regulators of inflammation and infection. Recent advances shed light on the mechanisms linking ER-stress and immune responses...
  3. ncbi request reprint Dangerous liaisons: mitochondrial DNA meets the NLRP3 inflammasome
    Fabio Martinon
    Department of Biochemistry, University of Lausanne, 155 ch des Boveresses, Epalinges 1066, Switzerland
    Immunity 36:313-5. 2012
    ..Danger signals released by damaged organelles can promote inflammation. In this issue of Immunity, Shimada et al. (2012) report that oxidized DNA, released by mitochondria, directly binds and activates the NLRP3 inflammasome...