- Neuronal Ca(2+) dyshomeostasis in Huntington diseaseMarta Giacomello
University of Geneva, Geneva, Switzerland
Prion 7:76-84. 2013....
- OPA1 promotes pH flashes that spread between contiguous mitochondria without matrix protein exchangeJaime Santo-Domingo
Department of Cell Physiology and Metabolism, University of Geneva, Geneva, Switzerland
EMBO J 32:1927-40. 2013....
- Mitochondrial fission and cristae disruption increase the response of cell models of Huntington's disease to apoptotic stimuliVeronica Costa
Department of Cell Physiology and Medicine, University of Geneva, Geneva, Switzerland
EMBO Mol Med 2:490-503. 2010..Conversely, the increased apoptosis can be corrected by manoeuvres that prevent fission and cristae remodelling. In conclusion, the cristae remodelling of the fragmented HD mitochondria contributes to their hypersensitivity to apoptosis...