Research Topics
| Raymond David PridmoreSummaryCountry: Switzerland Publications
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Detail Information
Publications
The genome sequence of the probiotic intestinal bacterium Lactobacillus johnsonii NCC 533R David Pridmore
Department of Nutrition and Health, Nestle Research Center, PO Box 44, Vers chez les Blanc, 1000 Lausanne 26, Switzerland
Proc Natl Acad Sci U S A 101:2512-7. 2004..Many of these regions of difference appear to encode metabolic or structural components that could affect the organisms competitiveness or interactions with the GIT ecosystem...
Hydrogen peroxide production by Lactobacillus johnsonii NCC 533 and its role in anti-Salmonella activityRaymond David Pridmore
Department of Nutrition and Health, Nestle Research Center, Vers chez les Blancs, Lausanne, Switzerland
FEMS Microbiol Lett 283:210-5. 2008....
The role of prophage for genome diversification within a clonal lineage of Lactobacillus johnsonii: characterization of the defective prophage LJ771Emmanuel Denou
Nestle Research Center, Nestec Ltd, P O Box 44, CH 1000 Lausanne 26, Switzerland
J Bacteriol 190:5806-13. 2008..The two L. johnsonii strains had identical in vitro growth and in vivo gut persistence phenotypes. Also, in an isogenic background, the presence of the prophage resulted in no growth disadvantage...
Cell surface-associated elongation factor Tu mediates the attachment of Lactobacillus johnsonii NCC533 (La1) to human intestinal cells and mucinsDominique Granato
Nestle Research Center, CH 1000 Lausanne 26, Switzerland
Infect Immun 72:2160-9. 2004..Our in vitro results indicate that EF-Tu, through its binding to the intestinal mucosa, might participate in gut homeostasis...
Identification of genes associated with the long-gut-persistence phenotype of the probiotic Lactobacillus johnsonii strain NCC533 using a combination of genomics and transcriptome analysisEmmanuel Denou
Nestle Research Centre, CH 1000 Lausanne 26 Vers chez les Blanc, Switzerland
J Bacteriol 190:3161-8. 2008..The experiments revealed that deletion of LJ1654 to LJ1656 and LJ1680 decreased the gut residence time, while a mutant with a deleted exopolysaccharide biosynthesis cluster had a slightly increased residence time...
