Peter Saetre

Summary

Affiliation: Uppsala University
Country: Sweden

Publications

  1. ncbi Speciation, introgressive hybridization and nonlinear rate of molecular evolution in flycatchers
    G P Saetre
    Department of Evolutionary Biology, Evolutionary Biology Centre, Uppsala University, Norbyvagen 18D, SE 752 36 Uppsala, Sweden
    Mol Ecol 10:737-49. 2001
  2. ncbi Inflammation-related genes up-regulated in schizophrenia brains
    Peter Saetre
    Department of Development and Genetics, Uppsala University, Sweden
    BMC Psychiatry 7:46. 2007
  3. ncbi Support for schizophrenia susceptibility locus on chromosome 2q detected in a Swedish isolate using a dense map of microsatellites and SNPs
    Karolina Aberg
    Department of Evolution, Genomics and Systematics, Uppsala University, Uppsala, Sweden
    Am J Med Genet B Neuropsychiatr Genet 147:1238-44. 2008
  4. ncbi Human QKI, a new candidate gene for schizophrenia involved in myelination
    Karolina Aberg
    Department of Evolution, Genomics and Systematics, Uppsala University, Norbyvagen 18D, 752 36 Uppsala, Sweden
    Am J Med Genet B Neuropsychiatr Genet 141:84-90. 2006
  5. ncbi Human QKI, a potential regulator of mRNA expression of human oligodendrocyte-related genes involved in schizophrenia
    Karolina Aberg
    Department of Evolution, Genomics and Systematics, Uppsala University, SE 75236 Uppsala, Sweden
    Proc Natl Acad Sci U S A 103:7482-7. 2006
  6. ncbi An evolutionarily conserved sexual signature in the primate brain
    Björn Reinius
    Department of Development and Genetics, Uppsala University, Uppsala, Sweden
    PLoS Genet 4:e1000100. 2008
  7. ncbi Haloperidol changes mRNA expression of a QKI splice variant in human astrocytoma cells
    Lin Jiang
    Department of Development and Genetics, Uppsala University, Sweden
    BMC Pharmacol 9:6. 2009
  8. ncbi Serotonin receptor 2C (HTR2C) and schizophrenia: examination of possible medication and genetic influences on expression levels
    Anja Castensson
    Department of Evolution, Genomics and Systematics, Evolutionary Biology Centre EBC, Uppsala University, Uppsala, Sweden
    Am J Med Genet B Neuropsychiatr Genet 134:84-9. 2005
  9. ncbi Flycatcher song in allopatry and sympatry--convergence, divergence and reinforcement
    J Haavie
    Department of Evolutionary Biology, Evolutionary Biology Centre, Uppsala University, Uppsala, Sweden
    J Evol Biol 17:227-37. 2004
  10. ncbi Reduced expression of TAC1, PENK and SOCS2 in Hcrtr-2 mutated narcoleptic dog brain
    Julia Lindberg
    Evolutionary Biology Centre, Department of Evolution, Genomics and Systematics, Uppsala University, Norbyvagen 18D, Uppsala, Sweden
    BMC Neurosci 8:34. 2007

Detail Information

Publications15

  1. ncbi Speciation, introgressive hybridization and nonlinear rate of molecular evolution in flycatchers
    G P Saetre
    Department of Evolutionary Biology, Evolutionary Biology Centre, Uppsala University, Norbyvagen 18D, SE 752 36 Uppsala, Sweden
    Mol Ecol 10:737-49. 2001
    ..After the last glaciation period, pied and collared flycatchers expanded their breeding ranges and eventually came into secondary contact in Central and Eastern Europe and on the Baltic Isles...
  2. ncbi Inflammation-related genes up-regulated in schizophrenia brains
    Peter Saetre
    Department of Development and Genetics, Uppsala University, Sweden
    BMC Psychiatry 7:46. 2007
    ..However, disentangling disease effects from alterations caused by medication is a challenging task. The main goal of this study is to find transcriptional alterations in schizophrenia that are independent of neuroleptic treatment...
  3. ncbi Support for schizophrenia susceptibility locus on chromosome 2q detected in a Swedish isolate using a dense map of microsatellites and SNPs
    Karolina Aberg
    Department of Evolution, Genomics and Systematics, Uppsala University, Uppsala, Sweden
    Am J Med Genet B Neuropsychiatr Genet 147:1238-44. 2008
    ..Haplotype sharing in families helped to delimit the detected regions that potentially are susceptibility loci for schizophrenia...
  4. ncbi Human QKI, a new candidate gene for schizophrenia involved in myelination
    Karolina Aberg
    Department of Evolution, Genomics and Systematics, Uppsala University, Norbyvagen 18D, 752 36 Uppsala, Sweden
    Am J Med Genet B Neuropsychiatr Genet 141:84-90. 2006
    ..Taken together, we propose QKI as a possible target for functional studies related to the role of myelination in schizophrenia...
  5. ncbi Human QKI, a potential regulator of mRNA expression of human oligodendrocyte-related genes involved in schizophrenia
    Karolina Aberg
    Department of Evolution, Genomics and Systematics, Uppsala University, SE 75236 Uppsala, Sweden
    Proc Natl Acad Sci U S A 103:7482-7. 2006
    ..Moreover, we hypothesize that previously observed decreased activity of myelin-related genes in schizophrenia might be caused by disturbed QKI splicing...
  6. ncbi An evolutionarily conserved sexual signature in the primate brain
    Björn Reinius
    Department of Development and Genetics, Uppsala University, Uppsala, Sweden
    PLoS Genet 4:e1000100. 2008
    ..Genes within sexual expression profiles may underlie important functional differences between the sexes, with possible importance during primate evolution...
  7. ncbi Haloperidol changes mRNA expression of a QKI splice variant in human astrocytoma cells
    Lin Jiang
    Department of Development and Genetics, Uppsala University, Sweden
    BMC Pharmacol 9:6. 2009
    ..Messenger RNA expression levels of splice variants QKI-5, QKI-6 and QKI-7 were measured by Real-Time PCR...
  8. ncbi Serotonin receptor 2C (HTR2C) and schizophrenia: examination of possible medication and genetic influences on expression levels
    Anja Castensson
    Department of Evolution, Genomics and Systematics, Evolutionary Biology Centre EBC, Uppsala University, Uppsala, Sweden
    Am J Med Genet B Neuropsychiatr Genet 134:84-9. 2005
    ..The disease related changes in HTR2C expression are not related to the promoter variants typed in our sample, but could be due to other regulatory variants or trans-acting factors...
  9. ncbi Flycatcher song in allopatry and sympatry--convergence, divergence and reinforcement
    J Haavie
    Department of Evolutionary Biology, Evolutionary Biology Centre, Uppsala University, Uppsala, Sweden
    J Evol Biol 17:227-37. 2004
    ..Whether a culturally inherited trait promotes or opposes speciation in sympatry may depend on its plasticity. The degree of plasticity may be genetically determined and accordingly under selection by reinforcement...
  10. ncbi Reduced expression of TAC1, PENK and SOCS2 in Hcrtr-2 mutated narcoleptic dog brain
    Julia Lindberg
    Evolutionary Biology Centre, Department of Evolution, Genomics and Systematics, Uppsala University, Norbyvagen 18D, Uppsala, Sweden
    BMC Neurosci 8:34. 2007
    ..However, little is known about molecular alterations downstream of the hypocretin signals...
  11. ncbi Genome-wide prediction of human VNTRs
    Karl Näslund
    Department of Genetics and Pathology, Rudbeck Laboratory, Uppsala University, S 751 85, Uppsala, Sweden
    Genomics 85:24-35. 2005
    ..Contrary to previous results, VNTRs appear to be widespread and abundant throughout the human genome, with an estimated density of 9.1 VNTRs/Mb...
  12. ncbi From wild wolf to domestic dog: gene expression changes in the brain
    Peter Saetre
    Department of Evolution, Genomics and Systematics, Uppsala University, Norbyvagen 18D, S-752 36 Uppsala, Sweden
    Brain Res Mol Brain Res 126:198-206. 2004
    ..Instead, they may provide a common mechanism for rapid adaptive changes during speciation, particularly in cases that present strong selective pressures on behavioral characters...
  13. ncbi Hybridization and adaptive mate choice in flycatchers
    T Veen
    Department of Animal Ecology, Evolutionary Biology Centre, Uppsala University, SE-752 36 Uppsala, Sweden
    Nature 411:45-50. 2001
    ..Apparent hybridization may thus represent adaptive mate choice under some circumstances...
  14. ncbi Increased monoamine oxidase messenger RNA expression levels in frontal cortex of Alzheimer's disease patients
    Lina Emilsson
    Department of Genetics and Pathology, Section of Medical Genetics, Rudbeck Laboratory, SE-751 85 Uppsala, Sweden
    Neurosci Lett 326:56-60. 2002
    ..We found a significant increase in mRNA levels for both MAOA (P=0.001) and MAOB (P=0.002) in disease brain tissue. This indicates that both MAO enzymes might be important in the progression of AD...
  15. ncbi Selection for tameness has changed brain gene expression in silver foxes
    Julia Lindberg
    Curr Biol 15:R915-6. 2005