Markus Heidenblad

Summary

Affiliation: University Hospital
Country: Sweden

Publications

  1. ncbi request reprint Microarray analyses reveal strong influence of DNA copy number alterations on the transcriptional patterns in pancreatic cancer: implications for the interpretation of genomic amplifications
    Markus Heidenblad
    Department of Clinical Genetics, Lund University Hospital, SE 221 85 Lund, Sweden
    Oncogene 24:1794-801. 2005
  2. ncbi request reprint Detailed genomic mapping and expression analyses of 12p amplifications in pancreatic carcinomas reveal a 3.5-Mb target region for amplification
    Markus Heidenblad
    Department of Clinical Genetics, Lund University Hospital, Sweden
    Genes Chromosomes Cancer 34:211-23. 2002
  3. ncbi request reprint Genome-wide array-based comparative genomic hybridization reveals multiple amplification targets and novel homozygous deletions in pancreatic carcinoma cell lines
    Markus Heidenblad
    Department of Clinical Genetics, Lund University Hospital, Lund, Sweden
    Cancer Res 64:3052-9. 2004
  4. ncbi request reprint Pancreatic carcinoma cell lines with SMAD4 inactivation show distinct expression responses to TGFB1
    Tord Jonson
    Department of Clinical Genetics, Lund University Hospital, SE 221 85 Lund, Sweden
    Genes Chromosomes Cancer 36:340-52. 2003
  5. doi request reprint Two genetic pathways, t(1;10) and amplification of 3p11-12, in myxoinflammatory fibroblastic sarcoma, haemosiderotic fibrolipomatous tumour, and morphologically similar lesions
    Karolin H Hallor
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    J Pathol 217:716-27. 2009
  6. ncbi request reprint High-resolution genomic profiles of breast cancer cell lines assessed by tiling BAC array comparative genomic hybridization
    Goran Jonsson
    Department of Oncology, University Hospital, Lund, Sweden
    Genes Chromosomes Cancer 46:543-58. 2007
  7. ncbi request reprint Tiling resolution array comparative genomic hybridization, expression and methylation analyses of dup(1q) in Burkitt lymphomas and pediatric high hyperdiploid acute lymphoblastic leukemias reveal clustered near-centromeric breakpoints and overexpression o
    Josef Davidsson
    Department of Clinical Genetics, Lund University Hospital, Sweden
    Hum Mol Genet 16:2215-25. 2007
  8. ncbi request reprint Tiling resolution array comparative genomic hybridization analysis of a fibrosarcoma of bone
    Karolin H Hallor
    Department of Clinical Genetics, Lund University Hospital, SE 221 85 Lund, Sweden
    Cancer Genet Cytogenet 172:80-3. 2007
  9. ncbi request reprint Array-based comparative genomic hybridization characterization of cytogenetically polyclonal myeloid malignancies
    Josef Davidsson
    Cancer Genet Cytogenet 169:179-80. 2006

Detail Information

Publications9

  1. ncbi request reprint Microarray analyses reveal strong influence of DNA copy number alterations on the transcriptional patterns in pancreatic cancer: implications for the interpretation of genomic amplifications
    Markus Heidenblad
    Department of Clinical Genetics, Lund University Hospital, SE 221 85 Lund, Sweden
    Oncogene 24:1794-801. 2005
    ..The presented findings indicate that more than one putative target gene may be of importance in most pancreatic cancer amplicons...
  2. ncbi request reprint Detailed genomic mapping and expression analyses of 12p amplifications in pancreatic carcinomas reveal a 3.5-Mb target region for amplification
    Markus Heidenblad
    Department of Clinical Genetics, Lund University Hospital, Sweden
    Genes Chromosomes Cancer 34:211-23. 2002
    ..These findings indicate two target regions within the 3.5-Mb segment in 12p11-12, one proximal including PPFIBP1, and one distal including KRAS2...
  3. ncbi request reprint Genome-wide array-based comparative genomic hybridization reveals multiple amplification targets and novel homozygous deletions in pancreatic carcinoma cell lines
    Markus Heidenblad
    Department of Clinical Genetics, Lund University Hospital, Lund, Sweden
    Cancer Res 64:3052-9. 2004
    ..The described amplification and deletion targets are likely to contain genes important in pancreatic tumorigenesis...
  4. ncbi request reprint Pancreatic carcinoma cell lines with SMAD4 inactivation show distinct expression responses to TGFB1
    Tord Jonson
    Department of Clinical Genetics, Lund University Hospital, SE 221 85 Lund, Sweden
    Genes Chromosomes Cancer 36:340-52. 2003
    ....
  5. doi request reprint Two genetic pathways, t(1;10) and amplification of 3p11-12, in myxoinflammatory fibroblastic sarcoma, haemosiderotic fibrolipomatous tumour, and morphologically similar lesions
    Karolin H Hallor
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    J Pathol 217:716-27. 2009
    ..The identified genetic aberrations were not confined to MIFS; an identical t(1;10) was also found in a case of HFT and the amplicon in 3p was seen in an IMFH...
  6. ncbi request reprint High-resolution genomic profiles of breast cancer cell lines assessed by tiling BAC array comparative genomic hybridization
    Goran Jonsson
    Department of Oncology, University Hospital, Lund, Sweden
    Genes Chromosomes Cancer 46:543-58. 2007
    ..The tiling BAC-arrays constitute a powerful tool for high-resolution genomic profiling suitable for cancer research and clinical diagnostics...
  7. ncbi request reprint Tiling resolution array comparative genomic hybridization, expression and methylation analyses of dup(1q) in Burkitt lymphomas and pediatric high hyperdiploid acute lymphoblastic leukemias reveal clustered near-centromeric breakpoints and overexpression o
    Josef Davidsson
    Department of Clinical Genetics, Lund University Hospital, Sweden
    Hum Mol Genet 16:2215-25. 2007
    ..However, involvement of these genes in dup(1q)-positive ALLs and BLs has previously not been reported...
  8. ncbi request reprint Tiling resolution array comparative genomic hybridization analysis of a fibrosarcoma of bone
    Karolin H Hallor
    Department of Clinical Genetics, Lund University Hospital, SE 221 85 Lund, Sweden
    Cancer Genet Cytogenet 172:80-3. 2007
    ..Thus, a reasonable interpretation of the pathogenetic significance of the ring formation would be that it resulted in the loss of one or more putative tumor suppressor gene loci distal to the two breakpoints...
  9. ncbi request reprint Array-based comparative genomic hybridization characterization of cytogenetically polyclonal myeloid malignancies
    Josef Davidsson
    Cancer Genet Cytogenet 169:179-80. 2006