K Broberg

Summary

Affiliation: University Hospital
Country: Sweden

Publications

  1. ncbi request reprint Polyclonal expansion of cells with trisomy 7 in synovia from patients with osteoarthritis
    K Broberg
    Clinical Genetics and Orthopedics, University Hospital, Lund Sweden
    Cytogenet Cell Genet 83:30-4. 1998
  2. ncbi request reprint Rearrangement of the neoplasia-associated gene HMGIC in synovia from patients with osteoarthritis
    K Broberg
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    Genes Chromosomes Cancer 24:278-82. 1999
  3. ncbi request reprint The tumor-associated gene HMGIC is expressed in normal and osteoarthritis-affected synovia
    K Broberg
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    Mod Pathol 14:311-7. 2001
  4. ncbi request reprint Analysis of the distribution and frequency of trisomy 7 in vivo in synovia from patients with osteoarthritis and pigmented villonodular synovitis
    A Dahlen
    Department of Clinical Genetics, University Hospital, SE 221 85, Lund, Sweden
    Cancer Genet Cytogenet 131:19-24. 2001
  5. ncbi request reprint Trisomy 7 accumulates with age in solid tumors and non-neoplastic synovia
    K Broberg
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    Genes Chromosomes Cancer 30:310-5. 2001
  6. doi request reprint Telomeric associations correlate with telomere length reduction and clonal chromosome aberrations in giant cell tumor of bone
    S Gebre-Medhin
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    Cytogenet Genome Res 124:121-7. 2009
  7. ncbi request reprint Cytogenetic polyclonality in hematologic malignancies
    B Johansson
    Department of Clinical Genetics, Lund University Hospital, Sweden
    Genes Chromosomes Cancer 24:222-9. 1999

Detail Information

Publications7

  1. ncbi request reprint Polyclonal expansion of cells with trisomy 7 in synovia from patients with osteoarthritis
    K Broberg
    Clinical Genetics and Orthopedics, University Hospital, Lund Sweden
    Cytogenet Cell Genet 83:30-4. 1998
    ..The finding of a polyclonal X-inactivation pattern in two of the cases provides strong evidence that gain of an extra copy of chromosome 7 occurs independently in multiple cells...
  2. ncbi request reprint Rearrangement of the neoplasia-associated gene HMGIC in synovia from patients with osteoarthritis
    K Broberg
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    Genes Chromosomes Cancer 24:278-82. 1999
    ....
  3. ncbi request reprint The tumor-associated gene HMGIC is expressed in normal and osteoarthritis-affected synovia
    K Broberg
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    Mod Pathol 14:311-7. 2001
    ..From the results of the present study, it is evident that expression of HMGIC cannot simply be considered a sign of neoplasia or an effect of proliferation...
  4. ncbi request reprint Analysis of the distribution and frequency of trisomy 7 in vivo in synovia from patients with osteoarthritis and pigmented villonodular synovitis
    A Dahlen
    Department of Clinical Genetics, University Hospital, SE 221 85, Lund, Sweden
    Cancer Genet Cytogenet 131:19-24. 2001
    ..By hematoxylin-eosin staining of the slides used for FISH analysis it could be shown that the clustered trisomic cells were proliferating synoviocytes within villous extensions of the synovial membrane...
  5. ncbi request reprint Trisomy 7 accumulates with age in solid tumors and non-neoplastic synovia
    K Broberg
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    Genes Chromosomes Cancer 30:310-5. 2001
    ....
  6. doi request reprint Telomeric associations correlate with telomere length reduction and clonal chromosome aberrations in giant cell tumor of bone
    S Gebre-Medhin
    Department of Clinical Genetics, University Hospital, Lund, Sweden
    Cytogenet Genome Res 124:121-7. 2009
    ..Thus, the present findings provide strong support for the notion that decreased telomere length is a prerequisite for tas in GCTBs and that the clonal changes occurring in GCTBs are derived from tas...
  7. ncbi request reprint Cytogenetic polyclonality in hematologic malignancies
    B Johansson
    Department of Clinical Genetics, Lund University Hospital, Sweden
    Genes Chromosomes Cancer 24:222-9. 1999
    ..001), and between B- and T-cell CLD (P < 0.001). Furthermore, the incidence was higher in therapy-related AML and MDS than in de novo AML and MDS (P < 0.001 and P < 0.01, respectively)...