Magdalena Nutu

Summary

Country: Sweden

Publications

  1. doi request reprint Evaluation of the cerebrospinal fluid amyloid-β1-42/amyloid-β1-40 ratio measured by alpha-LISA to distinguish Alzheimer's disease from other dementia disorders
    Magdalena Nutu
    Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, Sahlgrenska Academy, University of Gothenburg, Molndal, Sweden
    Dement Geriatr Cogn Disord 36:99-110. 2013
  2. doi request reprint Aβ1-15/16 as a potential diagnostic marker in neurodegenerative diseases
    Magdalena Nutu
    Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Sahlgrenska University Hospital Molndal, S 431 80, Molndal, Sweden
    Neuromolecular Med 15:169-79. 2013
  3. pmc Rapid effects of progesterone on ciliary beat frequency in the mouse fallopian tube
    Anna Bylander
    Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, SE 405 30 Goteborg, Sweden
    Reprod Biol Endocrinol 8:48. 2010
  4. pmc Amyloid-β(1-15/16) as a marker for γ-secretase inhibition in Alzheimer's disease
    Erik Portelius
    Clinical Neurochemistry Laboratory, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, The Sahlgrenska Academy, University of Gothenburg, Molndal, Sweden
    J Alzheimers Dis 31:335-41. 2012
  5. doi request reprint Soluble amyloid precursor protein α and β in CSF in Alzheimer's disease
    Gunnar Brinkmalm
    Department of Psychiatry and Neurochemistry, Clinical Neurochemistry Laboratory, Institute of Neuroscience and Physiology, Sahlgrenska Academy at University of Gothenburg, Molndal, Sweden
    Brain Res 1513:117-26. 2013

Collaborators

Detail Information

Publications5

  1. doi request reprint Evaluation of the cerebrospinal fluid amyloid-β1-42/amyloid-β1-40 ratio measured by alpha-LISA to distinguish Alzheimer's disease from other dementia disorders
    Magdalena Nutu
    Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, Sahlgrenska Academy, University of Gothenburg, Molndal, Sweden
    Dement Geriatr Cogn Disord 36:99-110. 2013
    ..This study aimed to evaluate whether the cerebrospinal fluid (CSF) amyloid-β (Aβ)1-42/Aβ1-40 ratio, measured by a novel method, could improve the differential diagnosis of AD, DLB and PDD...
  2. doi request reprint Aβ1-15/16 as a potential diagnostic marker in neurodegenerative diseases
    Magdalena Nutu
    Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Sahlgrenska University Hospital Molndal, S 431 80, Molndal, Sweden
    Neuromolecular Med 15:169-79. 2013
    ..We found reduced levels of Aβ1-15 in parkinsonian disease groups. The molecular mechanism behind this reduction is at present unknown...
  3. pmc Rapid effects of progesterone on ciliary beat frequency in the mouse fallopian tube
    Anna Bylander
    Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, SE 405 30 Goteborg, Sweden
    Reprod Biol Endocrinol 8:48. 2010
    ..The aims of this study were to establish methodology for studying fallopian tube CBF in the mouse, as it is an important model species, and to investigate if progesterone rapidly affects the CBF of mice at nM concentrations...
  4. pmc Amyloid-β(1-15/16) as a marker for γ-secretase inhibition in Alzheimer's disease
    Erik Portelius
    Clinical Neurochemistry Laboratory, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, The Sahlgrenska Academy, University of Gothenburg, Molndal, Sweden
    J Alzheimers Dis 31:335-41. 2012
    ..Our data shows that CSF levels of Aβ(1-15/16) increase during treatment with semagacestat supporting its feasibility as a pharmacodynamic biomarker for drug candidates aimed at inhibiting γ-secretase-mediated AβPP-processing...
  5. doi request reprint Soluble amyloid precursor protein α and β in CSF in Alzheimer's disease
    Gunnar Brinkmalm
    Department of Psychiatry and Neurochemistry, Clinical Neurochemistry Laboratory, Institute of Neuroscience and Physiology, Sahlgrenska Academy at University of Gothenburg, Molndal, Sweden
    Brain Res 1513:117-26. 2013
    ..Conclusion: Using high resolution MS, we show here for the first time that sAPPα in CSF ends at Q686 and K687. The findings also support the conclusion from several previous studies that sAPPα and sAPPβ levels are unaltered in AD...