John Andersson

Summary

Affiliation: Microbiology and Tumor Biology Center
Country: Sweden

Publications

  1. ncbi request reprint Chronic murine Chagas' disease: the impact of host and parasite genotypes
    John Andersson
    Microbiology and Tumor Biology Center, Karolinska Institutet, P O Box 280, S 171 77, Stockholm, Sweden
    Immunol Lett 86:207-12. 2003
  2. ncbi request reprint CBA/J mice infected with Trypanosoma cruzi: an experimental model for inflammatory myopathies
    John Andersson
    Microbiology and Tumorbiology Center, Karolinska Institutet, Stockholm, Sweden
    Muscle Nerve 27:442-8. 2003
  3. ncbi request reprint Molecular diagnosis of experimental Chagas disease
    John Andersson
    Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Building 10, Room 11N315, Bethesda, MD 20892, USA
    Trends Parasitol 20:52-3. 2004
  4. ncbi request reprint CD4+CD25+ regulatory T cells are activated in vivo by recognition of self
    John Andersson
    Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institute of Health, 10 Center Drive, MSC 1892, Bethesda, MD 20892 1892, USA
    Int Immunol 19:557-66. 2007
  5. ncbi request reprint Distinct subsets of FoxP3+ regulatory T cells participate in the control of immune responses
    Geoffrey L Stephens
    Cellular Immunology Section, Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    J Immunol 178:6901-11. 2007

Collaborators

Detail Information

Publications5

  1. ncbi request reprint Chronic murine Chagas' disease: the impact of host and parasite genotypes
    John Andersson
    Microbiology and Tumor Biology Center, Karolinska Institutet, P O Box 280, S 171 77, Stockholm, Sweden
    Immunol Lett 86:207-12. 2003
    ..cruzi infection. We also present a murine model suitable for investigating which host factors may be necessary to induce a chronic inflammatory disease after T. cruzi infection...
  2. ncbi request reprint CBA/J mice infected with Trypanosoma cruzi: an experimental model for inflammatory myopathies
    John Andersson
    Microbiology and Tumorbiology Center, Karolinska Institutet, Stockholm, Sweden
    Muscle Nerve 27:442-8. 2003
    ..The murine model described in this article may be useful in studying certain aspects of idiopathic inflammatory myopathies such as the role of T-cells in chronic muscle inflammation and chronic myocytotoxicity...
  3. ncbi request reprint Molecular diagnosis of experimental Chagas disease
    John Andersson
    Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Building 10, Room 11N315, Bethesda, MD 20892, USA
    Trends Parasitol 20:52-3. 2004
    ..cruzi isolate. The authors of this study also demonstrated higher sensitivity for this PCR setup compared with a commercially available enzyme-linked immunosorbent assay kit...
  4. ncbi request reprint CD4+CD25+ regulatory T cells are activated in vivo by recognition of self
    John Andersson
    Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institute of Health, 10 Center Drive, MSC 1892, Bethesda, MD 20892 1892, USA
    Int Immunol 19:557-66. 2007
    ..Thus, a sub-population of nT(R) is activated by recognition of self-peptide-MHC class II ligands in vivo, resulting in their capacity to be induced to mediate suppressor function in vitro in the absence of TCR stimulation...
  5. ncbi request reprint Distinct subsets of FoxP3+ regulatory T cells participate in the control of immune responses
    Geoffrey L Stephens
    Cellular Immunology Section, Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    J Immunol 178:6901-11. 2007
    ..Effector FoxP3(+) T cells may act as a sentinel of tolerance, providing a first line of defense against potentially harmful responses by rapidly suppressing immunity to peripheral self-Ags...