Research Topics | John AnderssonSummaryAffiliation: Microbiology and Tumor Biology Center Country: Sweden Publications
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Publications
Chronic murine Chagas' disease: the impact of host and parasite genotypesJohn Andersson
Microbiology and Tumor Biology Center, Karolinska Institutet, P O Box 280, S 171 77, Stockholm, Sweden
Immunol Lett 86:207-12. 2003..cruzi infection. We also present a murine model suitable for investigating which host factors may be necessary to induce a chronic inflammatory disease after T. cruzi infection...
CBA/J mice infected with Trypanosoma cruzi: an experimental model for inflammatory myopathiesJohn Andersson
Microbiology and Tumorbiology Center, Karolinska Institutet, Stockholm, Sweden
Muscle Nerve 27:442-8. 2003..The murine model described in this article may be useful in studying certain aspects of idiopathic inflammatory myopathies such as the role of T-cells in chronic muscle inflammation and chronic myocytotoxicity...
Molecular diagnosis of experimental Chagas diseaseJohn Andersson
Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Building 10, Room 11N315, Bethesda, MD 20892, USA
Trends Parasitol 20:52-3. 2004..cruzi isolate. The authors of this study also demonstrated higher sensitivity for this PCR setup compared with a commercially available enzyme-linked immunosorbent assay kit...
CD4+CD25+ regulatory T cells are activated in vivo by recognition of selfJohn Andersson
Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institute of Health, 10 Center Drive, MSC 1892, Bethesda, MD 20892 1892, USA
Int Immunol 19:557-66. 2007..Thus, a sub-population of nT(R) is activated by recognition of self-peptide-MHC class II ligands in vivo, resulting in their capacity to be induced to mediate suppressor function in vitro in the absence of TCR stimulation...
Distinct subsets of FoxP3+ regulatory T cells participate in the control of immune responsesGeoffrey L Stephens
Cellular Immunology Section, Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
J Immunol 178:6901-11. 2007..Effector FoxP3(+) T cells may act as a sentinel of tolerance, providing a first line of defense against potentially harmful responses by rapidly suppressing immunity to peripheral self-Ags...
