Petter Vikman

Summary

Affiliation: Lund University
Country: Sweden

Publications

  1. ncbi request reprint Transcriptional regulation of inflammatory and extracellular matrix-regulating genes in cerebral arteries following experimental subarachnoid hemorrhage in rats. Laboratory investigation
    Petter Vikman
    Department of Clinical Sciences, Experimental Vascular Research, Lund, Sweden
    J Neurosurg 107:1015-22. 2007
  2. ncbi request reprint Gene expression and molecular changes in cerebral arteries following subarachnoid hemorrhage in the rat
    Petter Vikman
    Department of Clinical Sciences, Experimental Vascular Research, Lunds universitet, Lund, Sweden
    J Neurosurg 105:438-44. 2006
  3. ncbi request reprint Gene expression profiling in the human middle cerebral artery after cerebral ischemia
    P Vikman
    Department of Experimental Vascular Research, Institution of Medicine, Lund University, Lund, Sweden
    Eur J Neurol 13:1324-32. 2006
  4. ncbi request reprint Cerebrovascular ETB, 5-HT1B, and AT1 receptor upregulation correlates with reduction in regional CBF after subarachnoid hemorrhage
    Saema Ansar
    Department of Clinical Sciences, Division of Experimental Vascular Research, Lund University, 221 84 Lund, Sweden
    Am J Physiol Heart Circ Physiol 293:H3750-8. 2007
  5. ncbi request reprint Cerebral ischemia induces transcription of inflammatory and extracellular-matrix-related genes in rat cerebral arteries
    Petter Vikman
    Department of Clinical Sciences, Experimental Vascular Research, BMC A13, 221 84, Lund, Sweden
    Exp Brain Res 183:499-510. 2007
  6. pmc Protein kinase C inhibition attenuates vascular ETB receptor upregulation and decreases brain damage after cerebral ischemia in rat
    Marie Henriksson
    Division of Experimental Vascular Research, Department of Clinical Sciences in Lund, Lund University, Sweden
    BMC Neurosci 8:7. 2007
  7. pmc Inhibition of PKC activity blocks the increase of ETB receptor expression in cerebral arteries
    Marie Henriksson
    Division of Experimental Vascular Research, Clinical Sciences, Lund University, Lund, Sweden
    BMC Pharmacol 6:13. 2006
  8. pmc Lipid-soluble cigarette smoking particles induce expression of inflammatory and extracellular-matrix-related genes in rat cerebral arteries
    Petter Vikman
    Department of Clinical Sciences, Experimental Vascular Research, Lund, Sweden
    Vasc Health Risk Manag 5:333-41. 2009
  9. ncbi request reprint MEK1/2 inhibition attenuates vascular ETA and ETB receptor alterations after cerebral ischaemia
    Marie Henriksson
    Division of Experimental Vascular Research, Department of Clinical Sciences in Lund, BMC A13, Lund University, Lund, 221 84 Sweden
    Exp Brain Res 178:470-6. 2007
  10. pmc Smoking particles enhance endothelin A and endothelin B receptor-mediated contractions by enhancing translation in rat bronchi
    Bengt W Granström
    Department of Medicine, Clinical Sciences, Lund University, Lund, Sweden
    BMC Pulm Med 6:6. 2006

Collaborators

Detail Information

Publications12

  1. ncbi request reprint Transcriptional regulation of inflammatory and extracellular matrix-regulating genes in cerebral arteries following experimental subarachnoid hemorrhage in rats. Laboratory investigation
    Petter Vikman
    Department of Clinical Sciences, Experimental Vascular Research, Lund, Sweden
    J Neurosurg 107:1015-22. 2007
    ....
  2. ncbi request reprint Gene expression and molecular changes in cerebral arteries following subarachnoid hemorrhage in the rat
    Petter Vikman
    Department of Clinical Sciences, Experimental Vascular Research, Lunds universitet, Lund, Sweden
    J Neurosurg 105:438-44. 2006
    ..The authors investigated early changes in the cerebral arteries of rats that occur after subarachnoid hemorrhage (SAH)...
  3. ncbi request reprint Gene expression profiling in the human middle cerebral artery after cerebral ischemia
    P Vikman
    Department of Experimental Vascular Research, Institution of Medicine, Lund University, Lund, Sweden
    Eur J Neurol 13:1324-32. 2006
    ..Gene expression studies in the human MCA leading to the ischemic region is similar to that seen after MCA occlusion in rats. We found new genes that support the dynamic changes that occur in the MCA distributing to the ischemic region...
  4. ncbi request reprint Cerebrovascular ETB, 5-HT1B, and AT1 receptor upregulation correlates with reduction in regional CBF after subarachnoid hemorrhage
    Saema Ansar
    Department of Clinical Sciences, Division of Experimental Vascular Research, Lund University, 221 84 Lund, Sweden
    Am J Physiol Heart Circ Physiol 293:H3750-8. 2007
    ..These changes occur in parallel with a successive decrease in CBF. Thus there is a temporal correlation between the changes in receptor expression and CBF reduction, suggesting a linkage...
  5. ncbi request reprint Cerebral ischemia induces transcription of inflammatory and extracellular-matrix-related genes in rat cerebral arteries
    Petter Vikman
    Department of Clinical Sciences, Experimental Vascular Research, BMC A13, 221 84, Lund, Sweden
    Exp Brain Res 183:499-510. 2007
    ....
  6. pmc Protein kinase C inhibition attenuates vascular ETB receptor upregulation and decreases brain damage after cerebral ischemia in rat
    Marie Henriksson
    Division of Experimental Vascular Research, Department of Clinical Sciences in Lund, Lund University, Sweden
    BMC Neurosci 8:7. 2007
    ..The present study aimed to examine the effect of the PKC inhibitor Ro-32-0432 on endothelin receptor upregulation, infarct volume and neurology outcome after middle cerebral artery occlusion in rat...
  7. pmc Inhibition of PKC activity blocks the increase of ETB receptor expression in cerebral arteries
    Marie Henriksson
    Division of Experimental Vascular Research, Clinical Sciences, Lund University, Lund, Sweden
    BMC Pharmacol 6:13. 2006
    ..Furthermore, immunohistochemistry was used to demonstrate the ETB receptor protein distribution in the MCA and Western blot to measure which of the PKC subtypes that were affected by the inhibitors...
  8. pmc Lipid-soluble cigarette smoking particles induce expression of inflammatory and extracellular-matrix-related genes in rat cerebral arteries
    Petter Vikman
    Department of Clinical Sciences, Experimental Vascular Research, Lund, Sweden
    Vasc Health Risk Manag 5:333-41. 2009
    ..Cigarette smoking is one of the strongest risk factors for stroke. However, the underlying molecular mechanisms that smoke leads to the pathogenesis of stroke are incompletely understood...
  9. ncbi request reprint MEK1/2 inhibition attenuates vascular ETA and ETB receptor alterations after cerebral ischaemia
    Marie Henriksson
    Division of Experimental Vascular Research, Department of Clinical Sciences in Lund, BMC A13, Lund University, Lund, 221 84 Sweden
    Exp Brain Res 178:470-6. 2007
    ..The vascular effects of U0126 provide new perspective on possible mechanisms of actions of MAPK inhibition in cerebral ischaemia...
  10. pmc Smoking particles enhance endothelin A and endothelin B receptor-mediated contractions by enhancing translation in rat bronchi
    Bengt W Granström
    Department of Medicine, Clinical Sciences, Lund University, Lund, Sweden
    BMC Pulm Med 6:6. 2006
    ..To study the effect of smoking on the endothelin system in rat airways, bronchial segments were exposed to DMSO-soluble smoking particles (DSP) from cigarette smoke, to nicotine and to DMSO, respectively...
  11. ncbi request reprint Angiotensin II-induced vasodilatation in cerebral arteries is mediated by endothelium-derived hyperpolarising factor
    Angelica Wackenfors
    Division of Experimental Vascular Research, Department of Clinical Science, Lund University Hospital, Lund, Sweden
    Eur J Pharmacol 531:259-63. 2006
    ..The angiotensin II dilatation was blocked by the angiotensin AT1 and AT2 receptor blockers candesartan and PD 123319. Both angiotensin AT1 and AT2 receptors were detected on the endothelium by immunohistochemistry...
  12. doi request reprint A systems genetics approach identifies genes and pathways for type 2 diabetes in human islets
    Jalal Taneera
    Lund University Diabetes Center, Department of Clinical Sciences, Diabetes and Endocrinology, Skåne University Hospital Malmö, Lund University, Malmo 20502, Sweden
    Cell Metab 16:122-34. 2012
    ..The data present a global map of genes associated with islet dysfunction and demonstrate the value of systems genetics for the identification of genes potentially involved in T2D...