Per Lindstrom

Summary

Country: Sweden

Publications

  1. ncbi The physiology of obese-hyperglycemic mice [ob/ob mice]
    Per Lindstrom
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, Umea, Sweden
    ScientificWorldJournal 7:666-85. 2007
  2. ncbi beta-cell function in obese-hyperglycemic mice [ob/ob Mice]
    Per Lindstrom
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, S 901 87 Umea, Sweden
    Adv Exp Med Biol 654:463-77. 2010
  3. ncbi Vasoactive intestinal polypeptide and pituitary adenylate cyclase activating polypeptide: effects on insulin release in isolated mouse islets in relation to metabolic status and age
    Solveig Persson-Sjögren
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, SE 901 87 Umea, Sweden
    Neuropeptides 40:283-90. 2006
  4. ncbi Cell specificity of the cytoplasmic Ca2+ response to tolbutamide is impaired in beta-cells from hyperglycemic mice
    Natalia Gustavsson
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, Umeå S 901 87, Sweden
    J Endocrinol 190:461-70. 2006
  5. ncbi Effects of cholinergic m-receptor agonists on insulin release in islets from obese and lean mice of different ages: the importance of bicarbonate
    Solveig Persson-Sjögren
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, Umea, Sweden
    Pancreas 29:e90-9. 2004
  6. ncbi Pancreatic beta cells from db/db mice show cell-specific [Ca2+]i and NADH responses to glucose but not to alpha-ketoisocaproic acid
    Natalia Gustavsson
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, Umea, Sweden
    Pancreas 31:242-50. 2005

Detail Information

Publications6

  1. ncbi The physiology of obese-hyperglycemic mice [ob/ob mice]
    Per Lindstrom
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, Umea, Sweden
    ScientificWorldJournal 7:666-85. 2007
    ..ob/ob Mice are well suited for studies on the interaction between leptin and insulin, and for studies on initial aspects of metabolic disturbances leading to type-2 diabetes...
  2. ncbi beta-cell function in obese-hyperglycemic mice [ob/ob Mice]
    Per Lindstrom
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, S 901 87 Umea, Sweden
    Adv Exp Med Biol 654:463-77. 2010
    ..ob/ob Mice can perhaps be described as a model for the prediabetic state. The large capacity for islet growth and insulin release makes ob/ob mice a good model for studies on how beta-cells can cope with prolonged functional stress...
  3. ncbi Vasoactive intestinal polypeptide and pituitary adenylate cyclase activating polypeptide: effects on insulin release in isolated mouse islets in relation to metabolic status and age
    Solveig Persson-Sjögren
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, SE 901 87 Umea, Sweden
    Neuropeptides 40:283-90. 2006
    ..We suggest that VIP and PACAP contribute to the developing metabolic syndrome in ob/ob mice by aggravating hyperinsulinemia...
  4. ncbi Cell specificity of the cytoplasmic Ca2+ response to tolbutamide is impaired in beta-cells from hyperglycemic mice
    Natalia Gustavsson
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, Umeå S 901 87, Sweden
    J Endocrinol 190:461-70. 2006
    ..The loss of temporal cell specificity for the response to tolbutamide in single beta-cells from hyperglycemic mice may be a sign of K(ATP)- or voltage-dependent calcium channel dysfunction...
  5. ncbi Effects of cholinergic m-receptor agonists on insulin release in islets from obese and lean mice of different ages: the importance of bicarbonate
    Solveig Persson-Sjögren
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, Umea, Sweden
    Pancreas 29:e90-9. 2004
    ..We have studied the age-related effects of M-receptor agonism on insulin release in islets isolated from female ob/ ob and lean mice...
  6. ncbi Pancreatic beta cells from db/db mice show cell-specific [Ca2+]i and NADH responses to glucose but not to alpha-ketoisocaproic acid
    Natalia Gustavsson
    Department of Integrative Medical Biology, Section for Histology and Cell Biology, Umea University, Umea, Sweden
    Pancreas 31:242-50. 2005
    ..Besides glucose, we studied the effects of glyceraldehyde, a glycolytic intermediate, and alpha-ketoisocaproic acid (KIC), a mitochondrial substrate...