Gerald Cooray

Summary

Affiliation: Karolinska Institutet
Country: Sweden

Publications

  1. ncbi Effects of intensified metabolic control on CNS function in type 2 diabetes
    Gerald Cooray
    Department of Clinical Neurophysiology, Karolinska University Hospital Solna, SE 17176 Stockholm, Sweden
    Psychoneuroendocrinology 36:77-86. 2011
  2. ncbi Decreased cortical connectivity and information flow in type 1 diabetes
    Gerald K Cooray
    Department of Clinical Neuroscience, Karolinska Institutet, SE 17176 Stockholm, Sweden
    Clin Neurophysiol 122:1943-50. 2011
  3. ncbi Predictors of cognitive impairment in type 1 diabetes
    Tom Brismar
    Department of Clinical Neurophysiology, Karolinska University Hospital Solna, SE 17176 Stockholm, Sweden
    Psychoneuroendocrinology 32:1041-51. 2007

Detail Information

Publications3

  1. ncbi Effects of intensified metabolic control on CNS function in type 2 diabetes
    Gerald Cooray
    Department of Clinical Neurophysiology, Karolinska University Hospital Solna, SE 17176 Stockholm, Sweden
    Psychoneuroendocrinology 36:77-86. 2011
    ..Furthermore, there was an improvement in electro-physiological measures, suggesting increased availability of compensatory mechanisms in subjects with intensified treatment...
  2. ncbi Decreased cortical connectivity and information flow in type 1 diabetes
    Gerald K Cooray
    Department of Clinical Neuroscience, Karolinska Institutet, SE 17176 Stockholm, Sweden
    Clin Neurophysiol 122:1943-50. 2011
    ..To investigate the effect of type 1 diabetes on EEG connectivity and information flow and study the relationship between these parameters and electrophysiological, neuropsychological and clinical variables...
  3. ncbi Predictors of cognitive impairment in type 1 diabetes
    Tom Brismar
    Department of Clinical Neurophysiology, Karolinska University Hospital Solna, SE 17176 Stockholm, Sweden
    Psychoneuroendocrinology 32:1041-51. 2007
    ..The influence of diabetes duration, BMI, height, age and CMAP may suggest that loss of the neuroprotective effects of insulin or insulin-like growth factors plays a role...