Martin Berglund

Summary

Country: Sweden

Publications

  1. doi Toll-like receptor cross-hyporesponsiveness is functional in interleukin-1-receptor-associated kinase-1 (IRAK-1)-deficient macrophages: differential role played by IRAK-1 in regulation of tumour necrosis factor and interleukin-10 production
    M Berglund
    Department of Microbiology and Immunology, Institute of Biomedicine, The Sahlgrenska Academy at Goteborg University, Goteborg, Sweden
    Scand J Immunol 67:473-9. 2008
  2. doi Gender dependent importance of IRAK-1 in dextran sulfate sodium induced colitis
    Martin Berglund
    Dept of Microbiology and Immunology, Institute of Biomedicine, The Sahlgrenska Academy at Goteborg University, Goteborg, Sweden
    Cell Immunol 259:27-32. 2009
  3. doi IL-1 receptor-associated kinase M downregulates DSS-induced colitis
    Martin Berglund
    Department of Microbiology and Immunology, Institute of Biomedicine, The Sahlgrenska Academy at Goteborg University, Sweden
    Inflamm Bowel Dis 16:1778-86. 2010
  4. ncbi Serum interleukin-1 receptor antagonist is an early indicator of colitis onset in Galphai2-deficient mice
    O H Hultgren
    Department of Clinical Immunology, Goteborg University, Guldhedsgatan 10 A, S 413 46 Goteborg, Sweden
    World J Gastroenterol 12:621-4. 2006
  5. ncbi The inflammatory response and hyaluronan synthases in the rabbit flexor tendon and tendon sheath following injury
    M Berglund
    Department of Hand Surgery, Uppsala University Hospital, Uppsala, Sweden
    J Hand Surg Eur Vol 32:581-7. 2007

Detail Information

Publications5

  1. doi Toll-like receptor cross-hyporesponsiveness is functional in interleukin-1-receptor-associated kinase-1 (IRAK-1)-deficient macrophages: differential role played by IRAK-1 in regulation of tumour necrosis factor and interleukin-10 production
    M Berglund
    Department of Microbiology and Immunology, Institute of Biomedicine, The Sahlgrenska Academy at Goteborg University, Goteborg, Sweden
    Scand J Immunol 67:473-9. 2008
    ..Our results demonstrate that IRAK-1 is not necessary for induction of TLR cross-tolerance as judged by TNF production...
  2. doi Gender dependent importance of IRAK-1 in dextran sulfate sodium induced colitis
    Martin Berglund
    Dept of Microbiology and Immunology, Institute of Biomedicine, The Sahlgrenska Academy at Goteborg University, Goteborg, Sweden
    Cell Immunol 259:27-32. 2009
    ..Our results indicate a gender specific effect of IRAK-1 in the DSS-induced colitis, an interesting finding since the Irak-1 gene is located on the X-chromosome and several inflammatory diseases have a gender dependent incidence...
  3. doi IL-1 receptor-associated kinase M downregulates DSS-induced colitis
    Martin Berglund
    Department of Microbiology and Immunology, Institute of Biomedicine, The Sahlgrenska Academy at Goteborg University, Sweden
    Inflamm Bowel Dis 16:1778-86. 2010
    ..We investigate the importance of the Toll-like receptor (TLR) regulating protein IL-1 receptor-associated kinase M (IRAK-M) using the erosive dextran sulfate sodium (DSS)-induced model of colitis...
  4. ncbi Serum interleukin-1 receptor antagonist is an early indicator of colitis onset in Galphai2-deficient mice
    O H Hultgren
    Department of Clinical Immunology, Goteborg University, Guldhedsgatan 10 A, S 413 46 Goteborg, Sweden
    World J Gastroenterol 12:621-4. 2006
    ..To study the serum concentration of IL-1beta, IL-1 receptor antagonist (IL-1Ra) and IL-18 in Galphai2-deficient mice at the age of 6 (healthy), 12 (pre-colitic) and 24 wk (colitic) and in healthy control mice...
  5. ncbi The inflammatory response and hyaluronan synthases in the rabbit flexor tendon and tendon sheath following injury
    M Berglund
    Department of Hand Surgery, Uppsala University Hospital, Uppsala, Sweden
    J Hand Surg Eur Vol 32:581-7. 2007
    ..These findings support the concept that healing in the flexor tendon and the sheath involve different molecular events and that each tissue may require unique modifications if healing is to be enhanced and adhesions reduced...