Louise Adermark

Summary

Country: Sweden

Publications

  1. ncbi Region-specific depression of striatal activity in Wistar rat by modest ethanol consumption over a ten-month period
    L Adermark
    Addiction Biology Unit, Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, PO Box 410, 405 30 Gothenburg, Sweden Electronic address
    Alcohol 47:289-98. 2013
  2. ncbi Modulation of endocannabinoid-mediated long-lasting disinhibition of striatal output by cholinergic interneurons
    Louise Adermark
    Addiction Biology Unit, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, University of Gothenburg, Box 410, 405 30 Gothenburg, Sweden
    Neuropharmacology 61:1314-20. 2011
  3. ncbi Ethanol-induced modulation of synaptic output from the dorsolateral striatum in rat is regulated by cholinergic interneurons
    Louise Adermark
    Addiction Biology Unit, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, University of Gothenburg, Gothenburg, Sweden
    Neurochem Int 58:693-9. 2011
  4. ncbi Intermittent ethanol consumption depresses endocannabinoid-signaling in the dorsolateral striatum of rat
    Louise Adermark
    Addiction Biology Unit, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, University of Gothenburg, Box 410, 405 30 Gothenburg, Sweden
    Neuropharmacology 61:1160-5. 2011
  5. ncbi Ethanol acutely decreases astroglial gap junction permeability in primary cultures from defined brain regions
    Louise Adermark
    Institute of Clinical Neuroscience, Goteborg University, Box 420, SE 405 30 Goteborg, Sweden
    Neurochem Int 45:971-8. 2004
  6. ncbi Implications for glycine receptors and astrocytes in ethanol-induced elevation of dopamine levels in the nucleus accumbens
    Louise Adermark
    Department of Psychiatry and Neurochemistry, Institute for Neuroscience and Physiology, University of Gothenburg, Sweden
    Addict Biol 16:43-54. 2011
  7. ncbi Rising taurine and ethanol concentrations in nucleus accumbens interact to produce dopamine release after ethanol administration
    Mia Ericson
    Addiction Biology Unit, Section of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, Sahlgrenska Academy at University of Gothenburg, Sweden
    Addict Biol 16:377-85. 2011
  8. ncbi beta-Alanine elevates dopamine levels in the rat nucleus accumbens: antagonism by strychnine
    Mia Ericson
    Addiction Biology Unit, Section of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, the Sahlgrenska Academy at University of Gothenburg, PO Box 410, 405 30, Goteborg, Sweden
    Amino Acids 38:1051-5. 2010
  9. ncbi Acute ethanol treatment prevents endocannabinoid-mediated long-lasting disinhibition of striatal output
    Rhona B C Clarke
    Addiction Biology Unit, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, University of Gothenburg, Box 410, 405 30 Gothenburg, Sweden
    Neuropharmacology 58:799-805. 2010
  10. ncbi Rising taurine and ethanol concentrations in nucleus accumbens interact to produce the dopamine-activating effects of alcohol
    Mia Ericson
    Institute of Neuroscience and Physiology, the Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden
    Adv Exp Med Biol 775:215-23. 2013

Detail Information

Publications16

  1. ncbi Region-specific depression of striatal activity in Wistar rat by modest ethanol consumption over a ten-month period
    L Adermark
    Addiction Biology Unit, Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, PO Box 410, 405 30 Gothenburg, Sweden Electronic address
    Alcohol 47:289-98. 2013
    ....
  2. ncbi Modulation of endocannabinoid-mediated long-lasting disinhibition of striatal output by cholinergic interneurons
    Louise Adermark
    Addiction Biology Unit, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, University of Gothenburg, Box 410, 405 30 Gothenburg, Sweden
    Neuropharmacology 61:1314-20. 2011
    ....
  3. ncbi Ethanol-induced modulation of synaptic output from the dorsolateral striatum in rat is regulated by cholinergic interneurons
    Louise Adermark
    Addiction Biology Unit, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, University of Gothenburg, Gothenburg, Sweden
    Neurochem Int 58:693-9. 2011
    ..In conclusion, EtOH modulates striatal microcircuitry and neurotransmission in a way that could be of importance for understanding the intoxicating properties as well as the acute reward sensation of EtOH...
  4. ncbi Intermittent ethanol consumption depresses endocannabinoid-signaling in the dorsolateral striatum of rat
    Louise Adermark
    Addiction Biology Unit, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, University of Gothenburg, Box 410, 405 30 Gothenburg, Sweden
    Neuropharmacology 61:1160-5. 2011
    ..In conclusion, alcohol consumption inhibits striatal eCB signaling in a way that could be of importance for understanding the neurological underpinnings of addictive behavior...
  5. ncbi Ethanol acutely decreases astroglial gap junction permeability in primary cultures from defined brain regions
    Louise Adermark
    Institute of Clinical Neuroscience, Goteborg University, Box 420, SE 405 30 Goteborg, Sweden
    Neurochem Int 45:971-8. 2004
    ..The results presented here indicate that ethanol-mediated decrease in dye spreading is directly or indirectly dependent on sodium...
  6. ncbi Implications for glycine receptors and astrocytes in ethanol-induced elevation of dopamine levels in the nucleus accumbens
    Louise Adermark
    Department of Psychiatry and Neurochemistry, Institute for Neuroscience and Physiology, University of Gothenburg, Sweden
    Addict Biol 16:43-54. 2011
    ..In conclusion, our data suggest that extracellular concentrations of dopamine and taurine are interconnected and that swelling of astrocytes contributes to the acute rewarding sensation of EtOH...
  7. ncbi Rising taurine and ethanol concentrations in nucleus accumbens interact to produce dopamine release after ethanol administration
    Mia Ericson
    Addiction Biology Unit, Section of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, Sahlgrenska Academy at University of Gothenburg, Sweden
    Addict Biol 16:377-85. 2011
    ..These data also provide support for the notion that the nAc is the primary target for ethanol in its dopamine-activating effect after systemic administration...
  8. ncbi beta-Alanine elevates dopamine levels in the rat nucleus accumbens: antagonism by strychnine
    Mia Ericson
    Addiction Biology Unit, Section of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, the Sahlgrenska Academy at University of Gothenburg, PO Box 410, 405 30, Goteborg, Sweden
    Amino Acids 38:1051-5. 2010
    ....
  9. ncbi Acute ethanol treatment prevents endocannabinoid-mediated long-lasting disinhibition of striatal output
    Rhona B C Clarke
    Addiction Biology Unit, Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, University of Gothenburg, Box 410, 405 30 Gothenburg, Sweden
    Neuropharmacology 58:799-805. 2010
    ..Our data suggest that changes in eCB-signaling could be involved in the physiological response to acute alcohol intoxication...
  10. ncbi Rising taurine and ethanol concentrations in nucleus accumbens interact to produce the dopamine-activating effects of alcohol
    Mia Ericson
    Institute of Neuroscience and Physiology, the Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden
    Adv Exp Med Biol 775:215-23. 2013
    ....
  11. ncbi Changes in glycine receptor subunit expression in forebrain regions of the Wistar rat over development
    Susanne Jonsson
    Addiction Biology Unit, Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, the Sahlgrenska Academy at University of Gothenburg, Sweden
    Brain Res 1446:12-21. 2012
    ..This indicates that alterations in GlyR subunit expression are not simply a maturation effect common for the entire CNS, but rather a unique pattern of transition depending on the region at hand...
  12. ncbi Lactate contributes to ammonia-mediated astroglial dysfunction during hyperammonemia
    Anna K Andersson
    Department of Clinical Neuroscience and Rehabilitation, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Guldhedsgatan 19, 1 tr, Gothenburg 41345, Sweden
    Neurochem Res 34:556-65. 2009
    ..Our results suggest that ammonium underlies brain edema, but that lactate can contribute to some of the cellular dysfunctions associated with elevated cerebral levels of ammonia...
  13. ncbi Subregion-Specific Modulation of Excitatory Input and Dopaminergic Output in the Striatum by Tonically Activated Glycine and GABA(A) Receptors
    Louise Adermark
    Addiction Biology Unit, Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, University of Gothenburg Gothenburg, Sweden
    Front Syst Neurosci 5:85. 2011
    ..Our data suggest that GABA(A) and glycine receptors are tonically activated and modulate striatal transmission in a partially subregion-specific manner...
  14. ncbi Ethanol effects on electrophysiological properties of astrocytes in striatal brain slices
    Louise Adermark
    Section on Synaptic Pharmacology, Laboratory for Integrative Neuroscience, NIAAA/NIH, 5625 Fishers Lane, TS-13, Bethesda, MD 20892, USA
    Neuropharmacology 51:1099-108. 2006
    ..Astrocytes play a critical role in brain potassium homeostasis, and therefore EtOH effects on astrocytic function could influence neuronal activity...
  15. ncbi Electrophysiological properties and gap junction coupling of striatal astrocytes
    Louise Adermark
    Section on Synaptic Pharmacology, Laboratory for Integrative Neuroscience, National Institute on Alcohol Abuse and Alcoholism, National Institutes of Health, 5625 Fishers Lane, Bethesda, MD 20892, USA
    Neurochem Int 52:1365-72. 2008
    ..Our findings indicate that the prevalent gap junction coupling is vital for astrocyte function in the striatum, and that whole-cell recordings will be distorted by currents activated in neighboring cells...
  16. ncbi Ethanol reverses the direction of long-term synaptic plasticity in the dorsomedial striatum
    Henry H Yin
    Section on Synaptic Pharmacology, Laboratory for Integrative Neuroscience, NIAAA NIH, 5625 Fishers Lane, TS 13, Bethesda, Maryland 20892, USA
    Eur J Neurosci 25:3226-32. 2007
    ....