Beth Stevens

Summary

Publications

  1. ncbi Enhanced synaptic connectivity and epilepsy in C1q knockout mice
    Yunxiang Chu
    Department of Neurology and Neurological Sciences and Neurobiology, Stanford University School of Medicine, Stanford University, Stanford, CA 94305, USA
    Proc Natl Acad Sci U S A 107:7975-80. 2010
  2. ncbi The classical complement cascade mediates CNS synapse elimination
    Beth Stevens
    Department of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Cell 131:1164-78. 2007
  3. ncbi The Down syndrome critical region regulates retinogeniculate refinement
    Martina Blank
    Department of Psychiatry, Stanford University School of Medicine, Stanford, California, 94305, USA
    J Neurosci 31:5764-76. 2011
  4. ncbi The complement system: an unexpected role in synaptic pruning during development and disease
    Alexander H Stephan
    Department of Neurobiology, Stanford University School of Medicine, Stanford, California 94305 5125, USA
    Annu Rev Neurosci 35:369-89. 2012
  5. ncbi Neuron-astrocyte signaling in the development and plasticity of neural circuits
    Beth Stevens
    Department of Neurobiology, Stanford University School of Medicine, Stanford, CA, USA
    Neurosignals 16:278-88. 2008
  6. ncbi NS21: re-defined and modified supplement B27 for neuronal cultures
    Yucui Chen
    Department of Pharmacology, University of Iowa, Iowa City, IA 52242 1109, United States
    J Neurosci Methods 171:239-47. 2008
  7. ncbi Adenosine: a neuron-glial transmitter promoting myelination in the CNS in response to action potentials
    Beth Stevens
    Laboratory of Cellular and Synaptic Neurophysiology, National Institutes of Health, NICHD, Bethesda, MD 20892, USA
    Neuron 36:855-68. 2002
  8. ncbi Glia: much more than the neuron's side-kick
    Beth Stevens
    Section on Nervous System Development and Plasticity, NICHD, National Institutes of Health, Bldg. 49, Room 5A78, MSC 4495, 49 Convent Drive, Bethesda, Maryland 20892, USA
    Curr Biol 13:R469-72. 2003
  9. ncbi Astrocytes promote myelination in response to electrical impulses
    Tomoko Ishibashi
    Nervous System Development and Plasticity Section, National Institute of Child Health and Human Development, Bethesda, Maryland 20892, USA
    Neuron 49:823-32. 2006

Collaborators

Detail Information

Publications9

  1. ncbi Enhanced synaptic connectivity and epilepsy in C1q knockout mice
    Yunxiang Chu
    Department of Neurology and Neurological Sciences and Neurobiology, Stanford University School of Medicine, Stanford University, Stanford, CA 94305, USA
    Proc Natl Acad Sci U S A 107:7975-80. 2010
    ..Results indicate that epileptogenesis in C1q KO mice is related to a genetically determined failure to prune excessive excitatory synapses during development...
  2. ncbi The classical complement cascade mediates CNS synapse elimination
    Beth Stevens
    Department of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Cell 131:1164-78. 2007
    ..These findings support a model in which unwanted synapses are tagged by complement for elimination and suggest that complement-mediated synapse elimination may become aberrantly reactivated in neurodegenerative disease...
  3. ncbi The Down syndrome critical region regulates retinogeniculate refinement
    Martina Blank
    Department of Psychiatry, Stanford University School of Medicine, Stanford, California, 94305, USA
    J Neurosci 31:5764-76. 2011
    ..Thus, altered developmental refinement of visual circuits that occurs before sensory experience is likely to contribute to visual impairment in individuals with Down syndrome...
  4. ncbi The complement system: an unexpected role in synaptic pruning during development and disease
    Alexander H Stephan
    Department of Neurobiology, Stanford University School of Medicine, Stanford, California 94305 5125, USA
    Annu Rev Neurosci 35:369-89. 2012
    ....
  5. ncbi Neuron-astrocyte signaling in the development and plasticity of neural circuits
    Beth Stevens
    Department of Neurobiology, Stanford University School of Medicine, Stanford, CA, USA
    Neurosignals 16:278-88. 2008
    ....
  6. ncbi NS21: re-defined and modified supplement B27 for neuronal cultures
    Yucui Chen
    Department of Pharmacology, University of Iowa, Iowa City, IA 52242 1109, United States
    J Neurosci Methods 171:239-47. 2008
    ..Furthermore, we found that holo-transferrin used in NS21 is preferable over apo-transferrin used in B27 for the quality of neuronal cultures...
  7. ncbi Adenosine: a neuron-glial transmitter promoting myelination in the CNS in response to action potentials
    Beth Stevens
    Laboratory of Cellular and Synaptic Neurophysiology, National Institutes of Health, NICHD, Bethesda, MD 20892, USA
    Neuron 36:855-68. 2002
    ....
  8. ncbi Glia: much more than the neuron's side-kick
    Beth Stevens
    Section on Nervous System Development and Plasticity, NICHD, National Institutes of Health, Bldg. 49, Room 5A78, MSC 4495, 49 Convent Drive, Bethesda, Maryland 20892, USA
    Curr Biol 13:R469-72. 2003
  9. ncbi Astrocytes promote myelination in response to electrical impulses
    Tomoko Ishibashi
    Nervous System Development and Plasticity Section, National Institute of Child Health and Human Development, Bethesda, Maryland 20892, USA
    Neuron 49:823-32. 2006
    ..This activity-dependent mechanism promoting myelination could regulate myelination according to functional activity or environmental experience and may offer new approaches to treating demyelinating diseases...