M Carmen Montesinos

Summary

Affiliation: University of Valencia
Country: Spain

Publications

  1. ncbi request reprint The antiinflammatory mechanism of methotrexate depends on extracellular conversion of adenine nucleotides to adenosine by ecto-5'-nucleotidase: findings in a study of ecto-5'-nucleotidase gene-deficient mice
    M Carmen Montesinos
    University of Valencia, Valencia, Spain
    Arthritis Rheum 56:1440-5. 2007
  2. pmc Suppression of inflammation by low-dose methotrexate is mediated by adenosine A2A receptor but not A3 receptor activation in thioglycollate-induced peritonitis
    M Carmen Montesinos
    Department of Pharmacology, Universidad de Valencia, Burjassot, Valencia, Spain
    Arthritis Res Ther 8:R53. 2006
  3. pmc Adenosine receptor agonists for promotion of dermal wound healing
    María D Valls
    Department de Farmacologia, Universitat de Valencia, Burjassot, Spain
    Biochem Pharmacol 77:1117-24. 2009
  4. ncbi request reprint An interaction between genetic factors and gender determines the magnitude of the inflammatory response in the mouse air pouch model of acute inflammation
    David L Delano
    New York University Medical Center, New York, New York 10016, USA
    Inflammation 29:1-7. 2005
  5. pmc Genetically based resistance to the antiinflammatory effects of methotrexate in the air-pouch model of acute inflammation
    David L Delano
    New York University Medical Center, NY 10016, USA
    Arthritis Rheum 52:2567-75. 2005
  6. ncbi request reprint Adenosine A2A receptor occupancy stimulates expression of proteins involved in reverse cholesterol transport and inhibits foam cell formation in macrophages
    Allison B Reiss
    Department of Medicine, New York University School of Medicine, New York, NY, USA
    J Leukoc Biol 76:727-34. 2004
  7. pmc Adenosine A(2A) receptor activation promotes wound neovascularization by stimulating angiogenesis and vasculogenesis
    M Carmen Montesinos
    Departments of Medicine and Surgery, The Veterans Administration New York Harbor Healthcare System, New York University Cancer Institute, New York University School of Medicine, New York, New York, USA
    Am J Pathol 164:1887-92. 2004
  8. ncbi request reprint Th1 cytokines regulate adenosine receptors and their downstream signaling elements in human microvascular endothelial cells
    D Khoa Nguyen
    Division of Clinical Pharmacology, Department of Medicine, New York University School of Medicine, New York, NY 10016, USA
    J Immunol 171:3991-8. 2003
  9. ncbi request reprint Adenosine A2A or A3 receptors are required for inhibition of inflammation by methotrexate and its analog MX-68
    M Carmen Montesinos
    New York University School of Medicine, New York, New York 10016, USA
    Arthritis Rheum 48:240-7. 2003
  10. pmc Adenosine promotes wound healing and mediates angiogenesis in response to tissue injury via occupancy of A(2A) receptors
    M Carmen Montesinos
    Department of Medicine, New York University School of Medicine, New York, New York, USA
    Am J Pathol 160:2009-18. 2002

Collaborators

Detail Information

Publications13

  1. ncbi request reprint The antiinflammatory mechanism of methotrexate depends on extracellular conversion of adenine nucleotides to adenosine by ecto-5'-nucleotidase: findings in a study of ecto-5'-nucleotidase gene-deficient mice
    M Carmen Montesinos
    University of Valencia, Valencia, Spain
    Arthritis Rheum 56:1440-5. 2007
    ....
  2. pmc Suppression of inflammation by low-dose methotrexate is mediated by adenosine A2A receptor but not A3 receptor activation in thioglycollate-induced peritonitis
    M Carmen Montesinos
    Department of Pharmacology, Universidad de Valencia, Burjassot, Valencia, Spain
    Arthritis Res Ther 8:R53. 2006
    ....
  3. pmc Adenosine receptor agonists for promotion of dermal wound healing
    María D Valls
    Department de Farmacologia, Universitat de Valencia, Burjassot, Spain
    Biochem Pharmacol 77:1117-24. 2009
    ....
  4. ncbi request reprint An interaction between genetic factors and gender determines the magnitude of the inflammatory response in the mouse air pouch model of acute inflammation
    David L Delano
    New York University Medical Center, New York, New York 10016, USA
    Inflammation 29:1-7. 2005
    ..The loci identified by in silico mapping likely contain genes with differential roles in determining this phenotype between genders...
  5. pmc Genetically based resistance to the antiinflammatory effects of methotrexate in the air-pouch model of acute inflammation
    David L Delano
    New York University Medical Center, NY 10016, USA
    Arthritis Rheum 52:2567-75. 2005
    ..To determine whether genetic factors or the model itself is responsible for the poor response to MTX, we directly compared the responses of 4 inbred mouse strains to MTX in the air-pouch model of acute inflammation...
  6. ncbi request reprint Adenosine A2A receptor occupancy stimulates expression of proteins involved in reverse cholesterol transport and inhibits foam cell formation in macrophages
    Allison B Reiss
    Department of Medicine, New York University School of Medicine, New York, NY, USA
    J Leukoc Biol 76:727-34. 2004
    ..These findings suggest a novel approach to the development of agents that prevent progression of atherosclerosis...
  7. pmc Adenosine A(2A) receptor activation promotes wound neovascularization by stimulating angiogenesis and vasculogenesis
    M Carmen Montesinos
    Departments of Medicine and Surgery, The Veterans Administration New York Harbor Healthcare System, New York University Cancer Institute, New York University School of Medicine, New York, New York, USA
    Am J Pathol 164:1887-92. 2004
    ....
  8. ncbi request reprint Th1 cytokines regulate adenosine receptors and their downstream signaling elements in human microvascular endothelial cells
    D Khoa Nguyen
    Division of Clinical Pharmacology, Department of Medicine, New York University School of Medicine, New York, NY 10016, USA
    J Immunol 171:3991-8. 2003
    ....
  9. ncbi request reprint Adenosine A2A or A3 receptors are required for inhibition of inflammation by methotrexate and its analog MX-68
    M Carmen Montesinos
    New York University School of Medicine, New York, New York 10016, USA
    Arthritis Rheum 48:240-7. 2003
    ....
  10. pmc Adenosine promotes wound healing and mediates angiogenesis in response to tissue injury via occupancy of A(2A) receptors
    M Carmen Montesinos
    Department of Medicine, New York University School of Medicine, New York, New York, USA
    Am J Pathol 160:2009-18. 2002
    ..Thus, targeting the adenosine A(2A) receptor is a novel approach to promoting wound healing and angiogenesis in normal individuals and those suffering from chronic wounds...
  11. ncbi request reprint Adenosine A2A receptor stimulation increases angiogenesis by down-regulating production of the antiangiogenic matrix protein thrombospondin 1
    Avani Desai
    Department of Medicine, New York University School of Medicine, New York, New York 10016, USA
    Mol Pharmacol 67:1406-13. 2005
    ..These results indicate that the angiogenic effects of adenosine A2A receptor activation are, at least in part, caused by the suppression of TSP1 secretion...
  12. ncbi request reprint Non-prostaglandin effects of aspirin III and salicylate: inhibition of integrin-dependent human neutrophil aggregation and inflammation in COX 2- and NF kappa B (P105)-knockout mice
    Gerald Weissmann
    Division of Rheumatology, Department of Medicine, NYU School of Medicine, New York, NY 10016, USA
    Adv Exp Med Biol 507:571-7. 2002
    ..In contrast, glucocorticoid action depended on the p105...
  13. ncbi request reprint Adenosine A2A receptor agonists promote more rapid wound healing than recombinant human platelet-derived growth factor (Becaplermin gel)
    Cassandre Victor-Vega
    Department of Medicine, New York University School of Medicine, NY 10016, USA
    Inflammation 26:19-24. 2002
    ....