Francisco J Ortega

Summary

Affiliation: Universitat de Barcelona
Country: Spain

Publications

  1. pmc Glibenclamide enhances neurogenesis and improves long-term functional recovery after transient focal cerebral ischemia
    Francisco J Ortega
    Unitat de Bioquímica i Biologia Molecular, Facultat de Medicina, Institut d Investigacions Biomediques August Pi i Sunyer, Universitat de Barcelona and Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas, Barcelona, Spain
    J Cereb Blood Flow Metab 33:356-64. 2013
  2. pmc Targeting microglial K(ATP) channels to treat neurodegenerative diseases: a mitochondrial issue
    Manuel J Rodriguez
    Unitat de Bioquímica i Biologia Molecular, Facultat de Medicina, Institut d Investigacions Biomediques August Pi i Sunyer, Universitat de Barcelona and Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas, UB, E 08036 Barcelona, Spain
    Oxid Med Cell Longev 2013:194546. 2013

Collaborators

Detail Information

Publications2

  1. pmc Glibenclamide enhances neurogenesis and improves long-term functional recovery after transient focal cerebral ischemia
    Francisco J Ortega
    Unitat de Bioquímica i Biologia Molecular, Facultat de Medicina, Institut d Investigacions Biomediques August Pi i Sunyer, Universitat de Barcelona and Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas, Barcelona, Spain
    J Cereb Blood Flow Metab 33:356-64. 2013
    ..Therefore, acute blockade of SUR1 by glibenclamide enhanced long-term brain repair in MCAO rats, which was associated with improved behavioral outcome...
  2. pmc Targeting microglial K(ATP) channels to treat neurodegenerative diseases: a mitochondrial issue
    Manuel J Rodriguez
    Unitat de Bioquímica i Biologia Molecular, Facultat de Medicina, Institut d Investigacions Biomediques August Pi i Sunyer, Universitat de Barcelona and Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas, UB, E 08036 Barcelona, Spain
    Oxid Med Cell Longev 2013:194546. 2013
    ..In this review, we discuss the mitochondrial KATP channel as a new target to control microglia activity, avoid its toxic phenotype, and facilitate a positive disease outcome. ..