E Balada

Summary

Affiliation: Hospital Universitari Vall d'Hebron
Country: Spain

Publications

  1. ncbi DNA methylation and systemic lupus erythematosus
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Ann N Y Acad Sci 1108:127-36. 2007
  2. ncbi Molecular mechanisms mediated by human endogenous retroviruses (HERVs) in autoimmunity
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Rev Med Virol 19:273-86. 2009
  3. ncbi Lack of association of the PTPN22 gene polymorphism R620W with systemic sclerosis
    E Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Clin Exp Rheumatol 24:321-4. 2006
  4. ncbi Implication of human endogenous retroviruses in the development of autoimmune diseases
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Barcelona, Spain
    Int Rev Immunol 29:351-70. 2010
  5. ncbi Enhanced transcript levels of CD48 in CD4⁺ T cells from systemic lupus erythematosus patients
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Passeig Vall d Hebron 119 129, 08035 Barcelona, Spain
    Immunobiology 216:1034-7. 2011
  6. ncbi Xenotropic murine leukemia virus-related virus (XMRV) in patients with systemic lupus erythematosus
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Vall d Hebron Hospital, Autonomous University of Barcelona, Barcelona, Spain
    J Clin Immunol 31:584-7. 2011
  7. ncbi Multiplex family-based study in systemic lupus erythematosus: association between the R620W polymorphism of PTPN22 and the FcgammaRIIa (CD32A) R131 allele
    E Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Tissue Antigens 68:432-8. 2006
  8. ncbi Anti-PDGFR-alpha antibodies measured by non-bioactivity assays are not specific for systemic sclerosis
    E Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Ann Rheum Dis 67:1027-9. 2008
  9. ncbi Beta2-glycoprotein I gene polymorphisms Val247Leu and Trp316Ser in Spanish patients with primary antiphospholipid syndrome
    J Pardos-Gea
    Autoimmune Diseases Research Laboratory, Vall d Hebron University Hospital Research Institute, Universitat Autonoma de Barcelona, Passeig de Vall d Hebron 119 129, 08035 Barcelona, Spain
    Rheumatol Int 32:927-32. 2012
  10. ncbi The PTPN22*R620W polymorphism does not confer genetic susceptibility to antiphospholipid syndrome in the Spanish population
    J Castro-Marrero
    Vall d Hebron University Hospital Research Institute, Universitat Autonoma de Barcelona, Barcelona, Spain
    Int J Immunogenet 38:529-31. 2011

Collaborators

  • Albert Selva-O'Callaghan
  • J Ordi-Ros
  • J Pardos-Gea
  • Maria Urquizu-Padilla
  • M Labrador-Horrillo
  • A Segarra
  • J Ordi
  • M Vilardell-Tarres
  • Miquel Vilardell-Tarres
  • J Castro-Marrero
  • F Martinez-Valle
  • Jesús Castro
  • Fernando Martinez Valle
  • Ana Bodaño
  • L Martinez Lostao
  • Josefina Cortes-Hernandez
  • J Cortes-Hernandez
  • J Villarreal
  • S Bujan-Rivas
  • A Sellas-Fernandez
  • J Ordi Ros
  • M Labrador Horrillo
  • Antonio Gonzalez
  • A Segarra Medrano
  • Patricia Carreira
  • Juan J Gomez-Reino
  • L Martinez-Lostao
  • J Majó Masferrer
  • A Segarra-Medrano
  • Carmen Conde
  • M Vilardell Tarres
  • Segundo Buján
  • Moisés Labrador
  • M Labrador
  • J L Tovar
  • W Thomson
  • K Poulton
  • D Crosdale
  • M Villardell
  • A Hajeer
  • W Ollier
  • L S Teh

Detail Information

Publications26

  1. ncbi DNA methylation and systemic lupus erythematosus
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Ann N Y Acad Sci 1108:127-36. 2007
    ..We suggest some alternative hypothesis that could help to understand the causes of the global DNA hypomethylation observed in the CD4+ T cells of these patients...
  2. ncbi Molecular mechanisms mediated by human endogenous retroviruses (HERVs) in autoimmunity
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Rev Med Virol 19:273-86. 2009
    ....
  3. ncbi Lack of association of the PTPN22 gene polymorphism R620W with systemic sclerosis
    E Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Clin Exp Rheumatol 24:321-4. 2006
    ..The aim of our study was to check for the prevalence of the PTPN22 R620W polymorphism in patients with systemic sclerosis...
  4. ncbi Implication of human endogenous retroviruses in the development of autoimmune diseases
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Barcelona, Spain
    Int Rev Immunol 29:351-70. 2010
    ..This review is aimed at discussing evidence for a possible role of HERVs in the etiopathogenesis of different autoimmune diseases...
  5. ncbi Enhanced transcript levels of CD48 in CD4⁺ T cells from systemic lupus erythematosus patients
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Passeig Vall d Hebron 119 129, 08035 Barcelona, Spain
    Immunobiology 216:1034-7. 2011
    ....
  6. ncbi Xenotropic murine leukemia virus-related virus (XMRV) in patients with systemic lupus erythematosus
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Vall d Hebron Hospital, Autonomous University of Barcelona, Barcelona, Spain
    J Clin Immunol 31:584-7. 2011
    ..Since chronic fatigue is commonly reported in patients with systemic lupus erythematosus (SLE) we aimed at testing the presence of this virus in these patients...
  7. ncbi Multiplex family-based study in systemic lupus erythematosus: association between the R620W polymorphism of PTPN22 and the FcgammaRIIa (CD32A) R131 allele
    E Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Tissue Antigens 68:432-8. 2006
    ....
  8. ncbi Anti-PDGFR-alpha antibodies measured by non-bioactivity assays are not specific for systemic sclerosis
    E Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Ann Rheum Dis 67:1027-9. 2008
    ..To evaluate the presence of anti-PDGFR-alpha antibodies by immunological methods in patients with systemic sclerosis (SSc)...
  9. ncbi Beta2-glycoprotein I gene polymorphisms Val247Leu and Trp316Ser in Spanish patients with primary antiphospholipid syndrome
    J Pardos-Gea
    Autoimmune Diseases Research Laboratory, Vall d Hebron University Hospital Research Institute, Universitat Autonoma de Barcelona, Passeig de Vall d Hebron 119 129, 08035 Barcelona, Spain
    Rheumatol Int 32:927-32. 2012
    ....
  10. ncbi The PTPN22*R620W polymorphism does not confer genetic susceptibility to antiphospholipid syndrome in the Spanish population
    J Castro-Marrero
    Vall d Hebron University Hospital Research Institute, Universitat Autonoma de Barcelona, Barcelona, Spain
    Int J Immunogenet 38:529-31. 2011
    ..The effect of the PTPN22 SNP on clinical manifestations and presence of antiphospholipid antibodies in APS warrants further investigations...
  11. ncbi Activation of the signal transducer and activator of transcription-1 in diffuse proliferative lupus nephritis
    L Martinez Lostao
    Autoimmune Diseases Research Lab, Vall d Hebron Research Institute, Barcelona, Spain
    Lupus 16:483-8. 2007
    ..041). Our results show that STAT-1 is activated in DPLN suggesting that biological effects of IFN-gamma in renal tissues depend, at least in part, on the activation of STAT-1...
  12. ncbi DNase 1 activity in patients with systemic lupus erythematosus: relationship with epidemiological, clinical, immunological and therapeutical features
    F Martinez-Valle
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Lupus 18:418-23. 2009
    ..DNase1 activity was significantly lower in patients with SLE. Nevertheless, we did not find further relationships with any other of the epidemiological, clinical, immunological or therapeutical variables considered...
  13. ncbi Echocardiography at diagnosis of antiphospholipid syndrome provides prognostic information on valvular disease evolution and identifies two subtypes of patients
    J Pardos-Gea
    Department of Internal Medicine, Autoimmune Diseases Unit, Hospital Universitari Vall d Hebron, Universitat Autonoma de Barcelona, Barcelona, Spain
    Lupus 19:575-82. 2010
    ....
  14. ncbi Mutations of activin-receptor-like kinase 1 (ALK-1) are not found in patients with pulmonary hypertension and underlying connective tissue disease
    Albert Selva-O'Callaghan
    Internal Medicine Department, Vall d Hebron General Hospital, Barcelona, Spain
    Clin Rheumatol 26:947-9. 2007
    ..The presence of mutations in the ALK-1 gene in ten patients with underlying connective tissue diseases was investigated...
  15. ncbi Chronic intestinal pseudo-obstruction associated with biliary tract dilatation in a patient with systemic lupus erythematosus
    J Pardos-Gea
    , Research Unit in Systemic Autoimmune Diseases, Universitat Autonoma de Barcelona, Barcelona, Spain
    Lupus 14:328-30. 2005
    ..The combination of bile duct associated with CIPO in a patient with lupus has not been previously reported in the literature and it probably suggests a smooth muscle dysmotility...
  16. ncbi Predictors of poor renal outcome in patients with lupus nephritis treated with combined pulses of cyclophosphamide and methylprednisolone
    J Cortes-Hernandez
    Department of Internal Medicine, Vall d Hebron Hospitals, Barcelona, Spain
    Lupus 12:287-96. 2003
    ..Patients with persistent proteinuria alone or a high chronicity index are less likely to respond to immunosuppression, and strict control of the hypertension may be the best approach...
  17. ncbi Mannose binding lectin and FcgammaRIIa (CD32) polymorphism in Spanish systemic lupus erythematosus patients
    J Villarreal
    Hospital General Vall d Hebron, Passieg Vall d Hebron 119 129, Barcelona, Spain
    Rheumatology (Oxford) 40:1009-12. 2001
    ..The aim of this study was to evaluate the relationship of these polymorphisms with SLE...
  18. ncbi Transcript levels of DNA methyltransferases DNMT1, DNMT3A and DNMT3B in CD4+ T cells from patients with systemic lupus erythematosus
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    Immunology 124:339-47. 2008
    ..It is possible that patients were reacting indirectly to an underlying DNA hypomethylation status by increasing the mRNA levels of DNA methyltransferases when the disease was being definitely active...
  19. ncbi Transcript overexpression of the MBD2 and MBD4 genes in CD4+ T cells from systemic lupus erythematosus patients
    Eva Balada
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    J Leukoc Biol 81:1609-16. 2007
    ....
  20. ncbi Serum levels of soluble CD40 ligand at flare and at remission in patients with systemic lupus erythematosus
    Maria Urquizu-Padilla
    Systemic Autoimmune Diseases Research Laboratory, Vall d Hebron Research Institute, Hospital Vall d Hebron, Barcelona, Spain
    J Rheumatol 36:953-60. 2009
    ..To perform a prospective evaluation of soluble CD40 ligand (sCD40L) levels according to the activity of systemic lupus erythematosus (SLE)...
  21. ncbi DNase 1 and systemic lupus erythematosus
    Fernando Martinez Valle
    Research Unit in Systemic Autoimmune Diseases, Vall d Hebron Research Institute, Hospital Vall d Hebron, Passeig Vall d Hebron 119 129, Barcelona, Spain
    Autoimmun Rev 7:359-63. 2008
    ..In this review we highlight the current findings in the pathophysiology, genetics, and therapeutical role of DNase1 in SLE...
  22. ncbi The complex immunogenetic basis of systemic lupus erythematosus
    Jesús Castro
    Autoimmune Diseases Research Laboratory, Vall d Hebron Research Institute, Passeig Vall d Hebron 119 129, Barcelona, Spain
    Autoimmun Rev 7:345-51. 2008
    ..In this review we highlight the current findings in the genetics of human SLE after using these approaches...
  23. ncbi Antihistone and anti-double-stranded deoxyribonucleic acid antibodies are associated with renal disease in systemic lupus erythematosus
    Josefina Cortes-Hernandez
    Internal Medicine, Vall d Hebron Hospitals, Barcelona, Spain
    Am J Med 116:165-73. 2004
    ..We sought to assess the nephritogenic antibody profile of patients with systemic lupus erythematosus (SLE), and to determine which antibodies were most useful in identifying patients at risk of nephritis...
  24. ncbi Changes in lipid profile between flare and remission of patients with systemic lupus erythematosus: a prospective study
    Maria Urquizu-Padilla
    Systemic Autoimmune Diseases Research Laboratory, Valld Hebron Research Institute, Vall d Hebron Hospital, Barcelona, Spain
    J Rheumatol 36:1639-45. 2009
    ..To determine the lipid profile of patients with systemic lupus erythematosus (SLE) according to the disease activity, and to calculate the percentage of patients that diverged from optimal values...
  25. ncbi Study of DNASE I gene polymorphisms in systemic lupus erythematosus susceptibility
    Ana Bodaño
    Ann Rheum Dis 66:560-1. 2007
  26. ncbi DNASE I mutation and systemic lupus erythematosus in a Spanish population: comment on the article by Tew et al
    Eva Balada
    Arthritis Rheum 46:1974-6; author reply 1976-7. 2002