Eric M Snyder

Summary

Publications

  1. ncbi Novel protein kinase A-dependent long-term depression of excitatory synapses
    Johanna L Gutlerner
    Department of Molecular Pharmacology, Physiology, and Biotechnology, Brown University, Providence, RI 02912, USA
    Neuron 36:921-31. 2002
  2. ncbi Ubiquitination regulates PSD-95 degradation and AMPA receptor surface expression
    Marcie Colledge
    Howard Hughes Medical Institute, Vollum Institute, Oregon Health and Science University, Portland, OR 97239, USA
    Neuron 40:595-607. 2003
  3. ncbi Regulation of NMDA receptor trafficking by amyloid-beta
    Eric M Snyder
    Laboratory for Molecular and Cellular Neuroscience, Rockefeller University, 1230 York Avenue, New York, New York 10021, USA
    Nat Neurosci 8:1051-8. 2005
  4. ncbi Role for A kinase-anchoring proteins (AKAPS) in glutamate receptor trafficking and long term synaptic depression
    Eric M Snyder
    Howard Hughes Medical Institute, The Picower Center for Learning and Memory and Department of Brain and Cognitive Sciences, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139, USA
    J Biol Chem 280:16962-8. 2005
  5. ncbi Beta-amyloid accumulation in APP mutant neurons reduces PSD-95 and GluR1 in synapses
    Claudia G Almeida
    Department of Neurology and Neuroscience, Laboratory of Alzheimer s Disease Neurobiology, Weill Medical College of Cornell University, 525 E 68th Street, NY 10021, USA
    Neurobiol Dis 20:187-98. 2005

Collaborators

Detail Information

Publications5

  1. ncbi Novel protein kinase A-dependent long-term depression of excitatory synapses
    Johanna L Gutlerner
    Department of Molecular Pharmacology, Physiology, and Biotechnology, Brown University, Providence, RI 02912, USA
    Neuron 36:921-31. 2002
    ..However, unlike the case for any previously described form of LTD, activation of cyclic AMP-dependent protein kinase (PKA) is necessary and sufficient to trigger LTD at synapses on VTA dopamine neurons...
  2. ncbi Ubiquitination regulates PSD-95 degradation and AMPA receptor surface expression
    Marcie Colledge
    Howard Hughes Medical Institute, Vollum Institute, Oregon Health and Science University, Portland, OR 97239, USA
    Neuron 40:595-607. 2003
    ..These data suggest that ubiquitination of PSD-95 through an Mdm2-mediated pathway is critical in regulating AMPA receptor surface expression during synaptic plasticity...
  3. ncbi Regulation of NMDA receptor trafficking by amyloid-beta
    Eric M Snyder
    Laboratory for Molecular and Cellular Neuroscience, Rockefeller University, 1230 York Avenue, New York, New York 10021, USA
    Nat Neurosci 8:1051-8. 2005
    ..Dephosphorylation of the NMDA receptor subunit NR2B at Tyr1472 correlated with receptor endocytosis. These data indicate a new mechanism by which amyloid-beta can cause synaptic dysfunction and contribute to Alzheimer disease pathology...
  4. ncbi Role for A kinase-anchoring proteins (AKAPS) in glutamate receptor trafficking and long term synaptic depression
    Eric M Snyder
    Howard Hughes Medical Institute, The Picower Center for Learning and Memory and Department of Brain and Cognitive Sciences, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139, USA
    J Biol Chem 280:16962-8. 2005
    ..These data indicate that synaptic anchoring of PKA through association with AKAPs plays an important role in the regulation of AMPA receptor surface expression and synaptic plasticity...
  5. ncbi Beta-amyloid accumulation in APP mutant neurons reduces PSD-95 and GluR1 in synapses
    Claudia G Almeida
    Department of Neurology and Neuroscience, Laboratory of Alzheimer s Disease Neurobiology, Weill Medical College of Cornell University, 525 E 68th Street, NY 10021, USA
    Neurobiol Dis 20:187-98. 2005
    ....