Jozef Burda

Summary

Affiliation: Slovak Academy of Sciences
Country: Slovakia

Publications

  1. ncbi request reprint Evidence for a role of second pathophysiological stress in prevention of delayed neuronal death in the hippocampal CA1 region
    Jozef Burda
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, Kosice, 040 01, Slovakia
    Neurochem Res 30:1397-405. 2005
  2. ncbi request reprint Role of protein synthesis in the ischemic tolerance acquisition induced by transient forebrain ischemia in the rat
    Jozef Burda
    Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, 040 01 Kosice, Slovakia
    Neurochem Res 28:1213-9. 2003
  3. ncbi request reprint Effect of L-carnitine on postischemic inhibition of protein synthesis in the rat brain
    Jozef Burda
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, 040 01 Kosice, Slovakia
    Gen Physiol Biophys 28:242-8. 2009
  4. doi request reprint Postconditioning and anticonditioning: possibilities to interfere to evoked apoptosis
    Jozef Burda
    Institute of Neurobiology, Slovak Academy of Sciences, 04001 Kosice, Slovakia
    Cell Mol Neurobiol 29:821-5. 2009
  5. ncbi request reprint Delayed postconditionig initiates additive mechanism necessary for survival of selectively vulnerable neurons after transient ischemia in rat brain
    Jozef Burda
    Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovakia
    Cell Mol Neurobiol 26:1141-51. 2006
  6. doi request reprint Bradykinin postconditioning protects pyramidal CA1 neurons against delayed neuronal death in rat hippocampus
    Viera Danielisova
    Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovak Republic
    Cell Mol Neurobiol 29:871-8. 2009
  7. ncbi request reprint Effects of bradykinin postconditioning on endogenous antioxidant enzyme activity after transient forebrain ischemia in rat
    Viera Danielisova
    Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 6, 040 01, Kosice, Slovak Republic
    Neurochem Res 33:1057-64. 2008
  8. ncbi request reprint Effects of one-day reperfusion after transient forebrain ischemia on circulatory system in the rat
    Petra Kravcukova
    Institute of Neurobiology SAS, Soltesovej 4 6, 040 01 Kosice, Slovakia
    Gen Physiol Biophys 29:113-21. 2010
  9. ncbi request reprint The changes in endogenous antioxidant enzyme activity after postconditioning
    Viera Danielisova
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej, 040 01 Kosice, Soltesovej, Slovak Republic
    Cell Mol Neurobiol 26:1181-91. 2006
  10. doi request reprint Brain-derived neurotrophic factor blood levels in two models of transient brain ischemia in rats
    Miroslav Gottlieb
    Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovak Republic
    Gen Physiol Biophys 32:139-42. 2013

Detail Information

Publications22

  1. ncbi request reprint Evidence for a role of second pathophysiological stress in prevention of delayed neuronal death in the hippocampal CA1 region
    Jozef Burda
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, Kosice, 040 01, Slovakia
    Neurochem Res 30:1397-405. 2005
    ....
  2. ncbi request reprint Role of protein synthesis in the ischemic tolerance acquisition induced by transient forebrain ischemia in the rat
    Jozef Burda
    Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, 040 01 Kosice, Slovakia
    Neurochem Res 28:1213-9. 2003
    ....
  3. ncbi request reprint Effect of L-carnitine on postischemic inhibition of protein synthesis in the rat brain
    Jozef Burda
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, 040 01 Kosice, Slovakia
    Gen Physiol Biophys 28:242-8. 2009
    ..We expect that the ability of L-carnitine to keep translational machinery on facilitates efficacy of postischemic remodulation of gene expression...
  4. doi request reprint Postconditioning and anticonditioning: possibilities to interfere to evoked apoptosis
    Jozef Burda
    Institute of Neurobiology, Slovak Academy of Sciences, 04001 Kosice, Slovakia
    Cell Mol Neurobiol 29:821-5. 2009
    ..This observation could be very valuable in situation when the aim of treatment is elimination of unwanted cell population from the organism...
  5. ncbi request reprint Delayed postconditionig initiates additive mechanism necessary for survival of selectively vulnerable neurons after transient ischemia in rat brain
    Jozef Burda
    Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovakia
    Cell Mol Neurobiol 26:1141-51. 2006
    ..This statement also means that up to 15 min of ischemia, postconditioning does not induce cumulation of injuries produced by the first and the second stress...
  6. doi request reprint Bradykinin postconditioning protects pyramidal CA1 neurons against delayed neuronal death in rat hippocampus
    Viera Danielisova
    Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovak Republic
    Cell Mol Neurobiol 29:871-8. 2009
    ..The present study was undertaken to evaluate possible neuroprotective effect of bradykinin against delayed neuronal death in hippocampal CA1 neurons if applied two days after transient forebrain ischemia in the rat...
  7. ncbi request reprint Effects of bradykinin postconditioning on endogenous antioxidant enzyme activity after transient forebrain ischemia in rat
    Viera Danielisova
    Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 6, 040 01, Kosice, Slovak Republic
    Neurochem Res 33:1057-64. 2008
    ..This study demonstrated that bradykinin postconditioning induces protection against ischemic brain injury and promotes neuronal survival...
  8. ncbi request reprint Effects of one-day reperfusion after transient forebrain ischemia on circulatory system in the rat
    Petra Kravcukova
    Institute of Neurobiology SAS, Soltesovej 4 6, 040 01 Kosice, Slovakia
    Gen Physiol Biophys 29:113-21. 2010
    ..Reduced antioxidant capacity in the blood and an elevated level of excitotoxic amino acid glutamate may cause lymphocyte DNA damage, and probably contribute to insufficient postischemic recovery of brain tissue...
  9. ncbi request reprint The changes in endogenous antioxidant enzyme activity after postconditioning
    Viera Danielisova
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej, 040 01 Kosice, Soltesovej, Slovak Republic
    Cell Mol Neurobiol 26:1181-91. 2006
    ..5. Our results suggest that endogenous antioxidants SOD and CAT could play considerable neuroprotective role after postconditioning...
  10. doi request reprint Brain-derived neurotrophic factor blood levels in two models of transient brain ischemia in rats
    Miroslav Gottlieb
    Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovak Republic
    Gen Physiol Biophys 32:139-42. 2013
    ....
  11. ncbi request reprint Post-conditioning exacerbates the MnSOD immune-reactivity after experimental cerebral global ischemia and reperfusion in the rat brain hippocampus
    Miroslava Nemethova
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4 6, 040 01 Kosice, Slovakia
    Cell Biol Int 32:128-35. 2008
    ....
  12. ncbi request reprint Activities of endogenous antioxidant enzymes in the cerebrospinal fluid and the hippocampus after transient forebrain ischemia in rat
    Viera Danielisova
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 6, 040 01 Kosice, Slovak Republic
    J Neurol Sci 253:61-5. 2007
    ..Ischemic attack causes a rapid response in hippocampal tissue as well as in the CSF, represented by an increase in the activity of endogenous antioxidant enzymes SOD and CAT...
  13. doi request reprint Transient forebrain ischemia impact on lymphocyte DNA damage, glutamic acid level, and SOD activity in blood
    Petra Kravcukova
    Institute of Neurobiology, Slovak Academy of Sciences, Kosice, 040 01, Slovak Republic
    Cell Mol Neurobiol 29:887-94. 2009
    ..In the present work we studied the impact of postischemic condition in the early phase of reperfusion on plasma and blood cells...
  14. doi request reprint Development of a pattern in biochemical parameters in the core and penumbra during infarct evolution after transient MCAO in rats
    Petra Bonova
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4 6, Kosice, SK 040 01, Slovakia
    Neurochem Int 62:8-14. 2013
    ..Moreover, the first day of post-ischaemic reperfusion in the present model of stroke seems to be the breakpoint, i.e. the time at which expanding cell death from the infarct core to the penumbra can be at least partially eliminated...
  15. doi request reprint An effective combination of two different methods of postconditioning
    Viera Danielisova
    Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, 040 01 Kosice, Slovakia
    Neurochem Res 37:2085-91. 2012
    ..Our findings further confirm that in cases of delayed neuronal death, which is practically pathologically-induced apoptosis, there exists a 2-day-wide therapeutic window that can be effectively exploited...
  16. ncbi request reprint Changes of endogenous antioxidant enzymes during ischemic tolerance acquisition
    Viera Danielisova
    Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, 040 01, Kosice, Slovak Republic
    Neurochem Res 30:559-65. 2005
    ..In spite of significant upregulation of these enzymes a large number of neurons in selectively vulnerable CA1 region of hippocampus undergoes to neurodegeneration within 7 days after ischemia...
  17. ncbi request reprint Iron deposition in the brain following the ischemia in a rat model of ischemic tolerance
    Viera Danielisova
    Department of Neurochemistry, Slovak Academy of Sciences, Institute of Neurobiology, Kosice, Slovak Republic
    Acta Medica (Hradec Kralove) 47:285-8. 2004
    ..Preconditioning 5 min ischemia followed by 2 days of reperfusion before 20 min ischemia also prevented degeneration of the pyramidal neurons in layers III and V of the frontal cortex...
  18. doi request reprint Aminoguanidine administration ameliorates hippocampal damage after middle cerebral artery occlusion in rat
    Viera Danielisova
    Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4 6, 040 01, Kosice, Slovak Republic
    Neurochem Res 36:476-86. 2011
    ..Our findings demonstrate that aminoguanidine decreases ischemic brain damage and improves neurological recovery after transient focal ischemia induced by MCAO...
  19. ncbi request reprint Iron deposition after transient forebrain ischemia in rat brain
    Viera Danielisova
    Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Kosice
    Neurochem Res 27:237-42. 2002
    ....
  20. ncbi request reprint The effect of normovolemic hemodilution on c-Fos protein immunoreactivity in the postischemic rat brain cortex
    Némethová Miroslava
    Institute of Neurobiology, Slovak Academy of Sciences, Koice, Slovak Republic
    Int J Neurosci 115:523-36. 2005
    ..The number of c-Fos positive neuronal nuclei was significantly decreased in the hemodilution group after 4 h of reperfusion in the parietal and basal neocortex compared to the reperfusion without hemodilution...
  21. doi request reprint Delayed post-conditioning reduces post-ischemic glutamate level and improves protein synthesis in brain
    Petra Bonova
    Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovak Republic Electronic address
    Neurochem Int 62:854-60. 2013
    ..In addition, the potential of glutamate for monitoring the progress of ischemia and efficacy of therapy was shown...
  22. ncbi request reprint Fluoro-Jade B staining following zymosan microinjection into the spinal cord white matter
    Kamila Saganová
    Center of Excellence, Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovak Republic
    Cell Mol Neurobiol 26:1463-73. 2006
    ....