Research Topics
Species | Jozef BurdaSummaryAffiliation: Slovak Academy of Sciences Country: Slovakia Publications
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Detail Information
Publications
Postconditioning and anticonditioning: possibilities to interfere to evoked apoptosisJozef Burda
Institute of Neurobiology, Slovak Academy of Sciences, 04001 Kosice, Slovakia
Cell Mol Neurobiol 29:821-5. 2009..This observation could be very valuable in situation when the aim of treatment is elimination of unwanted cell population from the organism...
Role of protein synthesis in the ischemic tolerance acquisition induced by transient forebrain ischemia in the ratJozef Burda
Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, 040 01 Kosice, Slovakia
Neurochem Res 28:1213-9. 2003....- Effect of L-carnitine on postischemic inhibition of protein synthesis in the rat brainJozef Burda
Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, 040 01 Kosice, Slovakia
Gen Physiol Biophys 28:242-8. 2009..We expect that the ability of L-carnitine to keep translational machinery on facilitates efficacy of postischemic remodulation of gene expression... - Evidence for a role of second pathophysiological stress in prevention of delayed neuronal death in the hippocampal CA1 regionJozef Burda
Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, Kosice, 040 01, Slovakia
Neurochem Res 30:1397-405. 2005.... - Delayed postconditionig initiates additive mechanism necessary for survival of selectively vulnerable neurons after transient ischemia in rat brainJozef Burda
Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovakia
Cell Mol Neurobiol 26:1141-51. 2006..This statement also means that up to 15 min of ischemia, postconditioning does not induce cumulation of injuries produced by the first and the second stress... - Bradykinin postconditioning protects pyramidal CA1 neurons against delayed neuronal death in rat hippocampusViera Danielisova
Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovak Republic
Cell Mol Neurobiol 29:871-8. 2009..The present study was undertaken to evaluate possible neuroprotective effect of bradykinin against delayed neuronal death in hippocampal CA1 neurons if applied two days after transient forebrain ischemia in the rat... - Effects of bradykinin postconditioning on endogenous antioxidant enzyme activity after transient forebrain ischemia in ratViera Danielisova
Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 6, 040 01, Kosice, Slovak Republic
Neurochem Res 33:1057-64. 2008..This study demonstrated that bradykinin postconditioning induces protection against ischemic brain injury and promotes neuronal survival... - Effects of one-day reperfusion after transient forebrain ischemia on circulatory system in the ratPetra Kravcukova
Institute of Neurobiology SAS, Soltesovej 4 6, 040 01 Kosice, Slovakia
Gen Physiol Biophys 29:113-21. 2010..Reduced antioxidant capacity in the blood and an elevated level of excitotoxic amino acid glutamate may cause lymphocyte DNA damage, and probably contribute to insufficient postischemic recovery of brain tissue... - The changes in endogenous antioxidant enzyme activity after postconditioningViera Danielisova
Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej, 040 01 Kosice, Soltesovej, Slovak Republic
Cell Mol Neurobiol 26:1181-91. 2006..5. Our results suggest that endogenous antioxidants SOD and CAT could play considerable neuroprotective role after postconditioning... - Activities of endogenous antioxidant enzymes in the cerebrospinal fluid and the hippocampus after transient forebrain ischemia in ratViera Danielisova
Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 6, 040 01 Kosice, Slovak Republic
J Neurol Sci 253:61-5. 2007..Ischemic attack causes a rapid response in hippocampal tissue as well as in the CSF, represented by an increase in the activity of endogenous antioxidant enzymes SOD and CAT... - Transient forebrain ischemia impact on lymphocyte DNA damage, glutamic acid level, and SOD activity in bloodPetra Kravcukova
Institute of Neurobiology, Slovak Academy of Sciences, Kosice, 040 01, Slovak Republic
Cell Mol Neurobiol 29:887-94. 2009..In the present work we studied the impact of postischemic condition in the early phase of reperfusion on plasma and blood cells... - Post-conditioning exacerbates the MnSOD immune-reactivity after experimental cerebral global ischemia and reperfusion in the rat brain hippocampusMiroslava Nemethova
Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4 6, 040 01 Kosice, Slovakia
Cell Biol Int 32:128-35. 2008.... - Changes of endogenous antioxidant enzymes during ischemic tolerance acquisitionViera Danielisova
Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, 040 01, Kosice, Slovak Republic
Neurochem Res 30:559-65. 2005..In spite of significant upregulation of these enzymes a large number of neurons in selectively vulnerable CA1 region of hippocampus undergoes to neurodegeneration within 7 days after ischemia... - Iron deposition in the brain following the ischemia in a rat model of ischemic toleranceViera Danielisova
Department of Neurochemistry, Slovak Academy of Sciences, Institute of Neurobiology, Kosice, Slovak Republic
Acta Medica (Hradec Kralove) 47:285-8. 2004..Preconditioning 5 min ischemia followed by 2 days of reperfusion before 20 min ischemia also prevented degeneration of the pyramidal neurons in layers III and V of the frontal cortex... - Aminoguanidine administration ameliorates hippocampal damage after middle cerebral artery occlusion in ratViera Danielisova
Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4 6, 040 01, Kosice, Slovak Republic
Neurochem Res 36:476-86. 2011..Our findings demonstrate that aminoguanidine decreases ischemic brain damage and improves neurological recovery after transient focal ischemia induced by MCAO... - Iron deposition after transient forebrain ischemia in rat brainViera Danielisova
Department of Neurochemistry, Institute of Neurobiology, Slovak Academy of Sciences, Kosice
Neurochem Res 27:237-42. 2002.... - The effect of normovolemic hemodilution on c-Fos protein immunoreactivity in the postischemic rat brain cortexNémethová Miroslava
Institute of Neurobiology, Slovak Academy of Sciences, Koice, Slovak Republic
Int J Neurosci 115:523-36. 2005..The number of c-Fos positive neuronal nuclei was significantly decreased in the hemodilution group after 4 h of reperfusion in the parietal and basal neocortex compared to the reperfusion without hemodilution... - An effective combination of two different methods of postconditioningViera Danielisova
Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4, 040 01 Kosice, Slovakia
Neurochem Res 37:2085-91. 2012..Our findings further confirm that in cases of delayed neuronal death, which is practically pathologically-induced apoptosis, there exists a 2-day-wide therapeutic window that can be effectively exploited... - Development of a pattern in biochemical parameters in the core and penumbra during infarct evolution after transient MCAO in ratsPetra Bonova
Institute of Neurobiology, Slovak Academy of Sciences, Soltesovej 4 6, Kosice, SK 040 01, Slovakia Electronic address
Neurochem Int 62:8-14. 2013..Moreover, the first day of post-ischaemic reperfusion in the present model of stroke seems to be the breakpoint, i.e. the time at which expanding cell death from the infarct core to the penumbra can be at least partially eliminated... - Fluoro-Jade B staining following zymosan microinjection into the spinal cord white matterKamila Saganová
Center of Excellence, Institute of Neurobiology, Slovak Academy of Sciences, Kosice, Slovak Republic
Cell Mol Neurobiol 26:1463-73. 2006....