D A Knorre

Summary

Affiliation: Moscow State University
Country: Russia

Publications

  1. ncbi Sir2-dependent daughter-to-mother transport of the damaged proteins in yeast is required to prevent high stress sensitivity of the daughters
    Dmitry A Knorre
    Belozersky Institute of Physico Chemical Biology and Institute of Mitoegineering, Moscow State University, Russia
    Cell Cycle 9:4501-5. 2010
  2. doi Dodecyltriphenylphosphonium inhibits multiple drug resistance in the yeast Saccharomyces cerevisiae
    Dmitry A Knorre
    Belozersky Institute of Physico Chemical Biology, Moscow State University, Vorobyevy Gory 1, Moscow, Russia Institute of Mitoengineering, Moscow State University, Vorobyevy Gory 1, Moscow, Russia Electronic address
    Biochem Biophys Res Commun 450:1481-4. 2014
  3. doi Mitochondrial signaling in Saccharomyces cerevisiae pseudohyphae formation induced by butanol
    Anna N Starovoytova
    Faculty of Bioengineering and Bioinformatics, Moscow State University, Moscow, Russia
    FEMS Yeast Res 13:367-74. 2013
  4. ncbi Cyclosporin A-sensitive cytochrome c release and activation of external pathway of NADH oxidation in liver mitochondria due to pore opening by acidification of phosphate-containing incubation medium
    D A Knorre
    Department of Bioenergetics, A N Belozersky Institute of Physico Chemical Biology, Moscow State University, Moscow, Russia
    Biosci Rep 23:67-75. 2003
  5. doi Prooxidants prevent yeast cell death induced by genotoxic stress
    Dmitry A Knorre
    Belozersky Institute of Physico Chemical Biology and Institute of Mitoengineering, Moscow State University, Vorobyevy Gory 1, Moscow, Russia
    Cell Biol Int 35:431-5. 2011
  6. doi Amiodarone inhibits multiple drug resistance in yeast Saccharomyces cerevisiae
    Dmitry A Knorre
    A N Belozersky Institute of Physico Chemical Biology, Moscow State University, GSP 2, Leninskie Gory, 119992, Moscow, Russia
    Arch Microbiol 191:675-9. 2009
  7. ncbi Natural conditions inducing programmed cell death in the yeast Saccharomyces cerevisiae
    D A Knorre
    Belozersky Institute of Physico Chemical Biology, Lomonosov Moscow State University, Moscow 119992, Russia
    Biochemistry (Mosc) 70:264-6. 2005
  8. ncbi Physiological scenarios of programmed loss of mitochondrial DNA function and death of yeast
    S A Kochmak
    Faculty of Bioengineering and Bioinformatics, Lomonosov Moscow State University, Russia
    Biochemistry (Mosc) 76:167-71. 2011
  9. doi Longevity and mitochondrial membrane potential
    D A Knorre
    Belozersky Institute of Physico Chemical Biology, Lomonosov Moscow State University, Moscow, 119991, Russia
    Biochemistry (Mosc) 77:793-4. 2012
  10. ncbi Mitochondrial matrix fragmentation as a protection mechanism of yeast Saccharomyces cerevisiae
    D A Knorre
    Belozersky Institute of Physico Chemical Biology, Lomonosov Moscow State University, Moscow, 119992, Russia
    Biochemistry (Mosc) 73:1254-9. 2008

Collaborators

  • F F Severin
  • E N Mokhova
  • I E Karavaeva
  • Anna N Starovoytova
  • S A Kochmak
  • K V Shekhireva
  • Maxim I Sorokin
  • Svyatoslav S Sokolov
  • S S Sokolov

Detail Information

Publications11

  1. ncbi Sir2-dependent daughter-to-mother transport of the damaged proteins in yeast is required to prevent high stress sensitivity of the daughters
    Dmitry A Knorre
    Belozersky Institute of Physico Chemical Biology and Institute of Mitoegineering, Moscow State University, Russia
    Cell Cycle 9:4501-5. 2010
    ..Thus, daughter cells are more vulnerable to a variety of stresses than the young mothers, and Sir2-dependent transport of the adverse factors acts to equalize the resistance...
  2. doi Dodecyltriphenylphosphonium inhibits multiple drug resistance in the yeast Saccharomyces cerevisiae
    Dmitry A Knorre
    Belozersky Institute of Physico Chemical Biology, Moscow State University, Vorobyevy Gory 1, Moscow, Russia Institute of Mitoengineering, Moscow State University, Vorobyevy Gory 1, Moscow, Russia Electronic address
    Biochem Biophys Res Commun 450:1481-4. 2014
    ..The chemical nature of C12TPP suggests that after Pdr5p-driven extrusion the molecules return to the plasma membrane and then into the cytosol, thus effectively competing with other substrates of the pump...
  3. doi Mitochondrial signaling in Saccharomyces cerevisiae pseudohyphae formation induced by butanol
    Anna N Starovoytova
    Faculty of Bioengineering and Bioinformatics, Moscow State University, Moscow, Russia
    FEMS Yeast Res 13:367-74. 2013
    ..These mitochondria-activated signaling pathways appear to converge at Mih1p level...
  4. ncbi Cyclosporin A-sensitive cytochrome c release and activation of external pathway of NADH oxidation in liver mitochondria due to pore opening by acidification of phosphate-containing incubation medium
    D A Knorre
    Department of Bioenergetics, A N Belozersky Institute of Physico Chemical Biology, Moscow State University, Moscow, Russia
    Biosci Rep 23:67-75. 2003
    ....
  5. doi Prooxidants prevent yeast cell death induced by genotoxic stress
    Dmitry A Knorre
    Belozersky Institute of Physico Chemical Biology and Institute of Mitoengineering, Moscow State University, Vorobyevy Gory 1, Moscow, Russia
    Cell Biol Int 35:431-5. 2011
    ..The comparison of the published expression profile responses to prooxidant and MMS treatments identifies a set of ROS-activated genes, which are likely to protect cells from the genotoxic stress...
  6. doi Amiodarone inhibits multiple drug resistance in yeast Saccharomyces cerevisiae
    Dmitry A Knorre
    A N Belozersky Institute of Physico Chemical Biology, Moscow State University, GSP 2, Leninskie Gory, 119992, Moscow, Russia
    Arch Microbiol 191:675-9. 2009
    ..Interestingly, the action of amiodarone is additive with the one of chloroquine, a known inhibitor of ABC-transporters. We speculate that these findings could help in the development of antifungal drug mixes...
  7. ncbi Natural conditions inducing programmed cell death in the yeast Saccharomyces cerevisiae
    D A Knorre
    Belozersky Institute of Physico Chemical Biology, Lomonosov Moscow State University, Moscow 119992, Russia
    Biochemistry (Mosc) 70:264-6. 2005
    ....
  8. ncbi Physiological scenarios of programmed loss of mitochondrial DNA function and death of yeast
    S A Kochmak
    Faculty of Bioengineering and Bioinformatics, Lomonosov Moscow State University, Russia
    Biochemistry (Mosc) 76:167-71. 2011
    ....
  9. doi Longevity and mitochondrial membrane potential
    D A Knorre
    Belozersky Institute of Physico Chemical Biology, Lomonosov Moscow State University, Moscow, 119991, Russia
    Biochemistry (Mosc) 77:793-4. 2012
    ..Low concentrations of protonophores can be beneficial by increasing the accuracy of the mitophagosomal degradation of mitochondria with deleterious mutations in their DNA...
  10. ncbi Mitochondrial matrix fragmentation as a protection mechanism of yeast Saccharomyces cerevisiae
    D A Knorre
    Belozersky Institute of Physico Chemical Biology, Lomonosov Moscow State University, Moscow, 119992, Russia
    Biochemistry (Mosc) 73:1254-9. 2008
    ..Mitochondria with fragmented matrixes were also detected in cells of the stationary phase. Our data suggest that such fragmentation acts as a cellular protective mechanism against stress...
  11. doi Does Mitochondrial Fusion Require Transmembrane Potential?
    I E Karavaeva
    Faculty of Bioengineering and Bioinformatics, Lomonosov Moscow State University, Moscow, 119992, Russia
    Biochemistry (Mosc) 80:549-58. 2015
    ..We speculate that transmembrane potential is not directly involved in regulation of mitochondrial fusion but affects mitochondrial NTP/NDP ratio, which in turn regulates their fusion. ..