M Y Menshikov

Summary

Affiliation: Institute of Experimental Cardiology
Country: Russia

Publications

  1. doi request reprint Participation of the urokinase-type plasminogen activator receptor (uPAR) in neutrophil transendothelial migration
    Boris K Pliyev
    Department of Biological and Medical Chemistry, Faculty of Fundamental Medicine, Moscow State University, Lomonosovsky Pr, 31 5, Moscow 119192, Russia
    Mol Immunol 48:1168-77. 2011
  2. doi request reprint Extracellular acidosis promotes neutrophil transdifferentiation to MHC class II-expressing cells
    Boris K Pliyev
    Department of Biological and Medical Chemistry, Faculty of Fundamental Medicine, Moscow State University, Moscow, Russia
    Cell Immunol 271:214-8. 2011
  3. doi request reprint Alpha-fetoprotein contributes to THP-1 cell invasion and chemotaxis via protein kinase and Gi-protein-dependent pathways
    Ekaterina Zubkova
    Institute of Experimental Cardiology, Cardiology Research and Production Center, Moscow, Russia
    Mol Cell Biochem 379:283-93. 2013
  4. ncbi request reprint Plasmin-independent gelatinase B (matrix metalloproteinase-9) release by monocytes under the influence of urokinase
    M Y Menshikov
    Institute of Experimental Cardiology, Cardiology Research Center, 3 ya Cherepkovskaya ul 15A, Moscow, 121552 Russia
    Biochemistry (Mosc) 66:954-9. 2001
  5. pmc Urokinase upregulates matrix metalloproteinase-9 expression in THP-1 monocytes via gene transcription and protein synthesis
    Mikhail Menshikov
    Institute of Experimental Cardiology, Cardiology Research Center, 3rd Cherepkovskaya St, 15a, 121552, Moscow, Russia
    Biochem J 367:833-9. 2002
  6. ncbi request reprint Urokinase induces matrix metalloproteinase-9/gelatinase B expression in THP-1 monocytes via ERK1/2 and cytosolic phospholipase A2 activation and eicosanoid production
    Mikhail Menshikov
    Institute of Experimental Cardiology, Cardiology Research Center, Moscow, Russia
    J Vasc Res 43:482-90. 2006
  7. ncbi request reprint Urokinase plasminogen activator stimulates vascular smooth muscle cell proliferation via redox-dependent pathways
    Mikhail Menshikov
    Cardiology Research Center, Moscow, Russia
    Arterioscler Thromb Vasc Biol 26:801-7. 2006

Collaborators

  • Hua Cai
  • Ye V Parfyonova
  • Boris K Pliyev
  • Ekaterina Zubkova
  • Lidiya Semenkova
  • Elena Dudich
  • Igor Dudich
  • Olga A Antonova
  • Alexander B Sumarokov
  • Lyudmila I Buriachkovskaia

Detail Information

Publications7

  1. doi request reprint Participation of the urokinase-type plasminogen activator receptor (uPAR) in neutrophil transendothelial migration
    Boris K Pliyev
    Department of Biological and Medical Chemistry, Faculty of Fundamental Medicine, Moscow State University, Lomonosovsky Pr, 31 5, Moscow 119192, Russia
    Mol Immunol 48:1168-77. 2011
    ..Thus, uPAR expressed on neutrophil plasma membrane regulates transendothelial migration independently of uPA proteolytical activity and acting as a cofactor for integrin Mac-1...
  2. doi request reprint Extracellular acidosis promotes neutrophil transdifferentiation to MHC class II-expressing cells
    Boris K Pliyev
    Department of Biological and Medical Chemistry, Faculty of Fundamental Medicine, Moscow State University, Moscow, Russia
    Cell Immunol 271:214-8. 2011
    ..Thus, extracellular acidosis can represent a factor that promotes neutrophil transdifferentiation and potentiates the functional abilities of the transdifferentiated cells in inflammatory foci in vivo...
  3. doi request reprint Alpha-fetoprotein contributes to THP-1 cell invasion and chemotaxis via protein kinase and Gi-protein-dependent pathways
    Ekaterina Zubkova
    Institute of Experimental Cardiology, Cardiology Research and Production Center, Moscow, Russia
    Mol Cell Biochem 379:283-93. 2013
    ..The action of CCR5 inhibitor Maraviroc results in partial suppression of MMP9 up-regulation and calcium response suggesting that CCR5 might be involved in these effects...
  4. ncbi request reprint Plasmin-independent gelatinase B (matrix metalloproteinase-9) release by monocytes under the influence of urokinase
    M Y Menshikov
    Institute of Experimental Cardiology, Cardiology Research Center, 3 ya Cherepkovskaya ul 15A, Moscow, 121552 Russia
    Biochemistry (Mosc) 66:954-9. 2001
    ..The effect of urokinase is completely abolished by actinomycin D and cycloheximide, indicating the participation of transcription and translation processes in its development...
  5. pmc Urokinase upregulates matrix metalloproteinase-9 expression in THP-1 monocytes via gene transcription and protein synthesis
    Mikhail Menshikov
    Institute of Experimental Cardiology, Cardiology Research Center, 3rd Cherepkovskaya St, 15a, 121552, Moscow, Russia
    Biochem J 367:833-9. 2002
    ..These results suggest that urokinase upregulates MMP9 expression in monocytes via MMP9 gene transcription and protein biosynthesis...
  6. ncbi request reprint Urokinase induces matrix metalloproteinase-9/gelatinase B expression in THP-1 monocytes via ERK1/2 and cytosolic phospholipase A2 activation and eicosanoid production
    Mikhail Menshikov
    Institute of Experimental Cardiology, Cardiology Research Center, Moscow, Russia
    J Vasc Res 43:482-90. 2006
    ..We characterized the mechanisms by which uPA regulates matrix metalloproteinase-9 (MMP9) function in THP-1 monocytes...
  7. ncbi request reprint Urokinase plasminogen activator stimulates vascular smooth muscle cell proliferation via redox-dependent pathways
    Mikhail Menshikov
    Cardiology Research Center, Moscow, Russia
    Arterioscler Thromb Vasc Biol 26:801-7. 2006
    ..Because increases in VSMC reactive oxygen species (ROS) contribute to VSMC proliferation, we hypothesized that uPA increases ROS generation by regulating expression or activity of cellular oxidases...