A E Berman

Summary

Affiliation: Institute of Biomedical Chemistry
Country: Russia

Publications

  1. ncbi Integrins: structure and functions
    A E Berman
    Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow
    Membr Cell Biol 13:207-44. 2000
  2. ncbi Role of integrin alphavbeta3 in substrate-dependent apoptosis of human intestinal carcinoma cells
    G E Morozevich
    Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow, 119992, Russia
    Biochemistry (Mosc) 68:416-23. 2003
  3. ncbi Integrins: structure and signaling
    A E Berman
    Orekchovich Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow 119121, Russia
    Biochemistry (Mosc) 68:1284-99. 2003
  4. ncbi Expression and role of integrins in invasive activity of oncotransformed fibroblasts differing in spontaneous metastasizing
    G E Morozevich
    Orekhovich Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow 119121, Russia
    Biochemistry (Mosc) 69:665-73. 2004
  5. ncbi Implication of alpha5beta1 integrin in invasion of drug-resistant MCF-7/ADR breast carcinoma cells: a role for MMP-2 collagenase
    G E Morozevich
    Orekhovich Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow, 119121, Russia
    Biochemistry (Mosc) 73:791-6. 2008
  6. ncbi The role of beta1 integrin subfamily in anchorage-dependent apoptosis of breast carcinoma cells differing in multidrug resistance
    G E Morozevich
    Orekhovich Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow, Russia
    Biochemistry (Mosc) 71:489-95. 2006
  7. ncbi Integrin alphavbeta3 promotes anchorage-dependent apoptosis in human intestinal carcinoma cells
    N I Kozlova
    Institute of Biomedical Chemistry RAMS, Pogodinskaya str. 10, 119832 Moscow, Russia
    Oncogene 20:4710-7. 2001
  8. ncbi [The role of alphavbeta3 integrin in changes of the invasive phenotype in CP-transformed fibroblasts with multiple drug resistance]
    G E Morozevich
    Institute of Biomedical Chemistry by V N Orekchovich, RAMS, Moscow
    Vopr Med Khim 48:111-20. 2002
  9. doi C-MYC overexpression is required for continuous suppression of oncogene-induced senescence in melanoma cells
    D Zhuang
    Department of Cell Stress Biology, Roswell Park Cancer Institute, Buffalo, NY 14263, USA
    Oncogene 27:6623-34. 2008

Collaborators

  • I B Cheglakov
  • G E Morozevich
  • N I Kozlova
  • A N Chubukina
  • D Zhuang
  • M E Preobrazhenskaya
  • N A Ushakova
  • I A Eltsov
  • J A Wawrzyniak
  • W H Tang
  • M S Soengas
  • T J Giordano
  • V Grachtchouk
  • S Patil
  • E V Prochownik
  • S Mannava
  • M A Nikiforov
  • A A Shtil

Detail Information

Publications9

  1. ncbi Integrins: structure and functions
    A E Berman
    Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow
    Membr Cell Biol 13:207-44. 2000
    ..Biochemical mechanisms, by which the signal functions of integrins are realized, are discussed. The data on the participation of integrins in oncogenic cell transformation, metastasis, and apoptosis are analyzed...
  2. ncbi Role of integrin alphavbeta3 in substrate-dependent apoptosis of human intestinal carcinoma cells
    G E Morozevich
    Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow, 119992, Russia
    Biochemistry (Mosc) 68:416-23. 2003
    ..The data provide the first evidence that alphavbeta3 integrin can generate apoptosis-stimulating signals...
  3. ncbi Integrins: structure and signaling
    A E Berman
    Orekchovich Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow 119121, Russia
    Biochemistry (Mosc) 68:1284-99. 2003
    ..This review summarizes current views on the structure of integrins, integrin associated proteins, and biochemical mechanisms underlying their signaling functions...
  4. ncbi Expression and role of integrins in invasive activity of oncotransformed fibroblasts differing in spontaneous metastasizing
    G E Morozevich
    Orekhovich Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow 119121, Russia
    Biochemistry (Mosc) 69:665-73. 2004
    ..The role of specific properties of the extracellular matrix surrounding tumor cells and of specific surface integrins expressed in these cells in developing of the malignant phenotype is discussed...
  5. ncbi Implication of alpha5beta1 integrin in invasion of drug-resistant MCF-7/ADR breast carcinoma cells: a role for MMP-2 collagenase
    G E Morozevich
    Orekhovich Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow, 119121, Russia
    Biochemistry (Mosc) 73:791-6. 2008
    ..These data suggest that alpha5beta1 controls invasion ability of these cells by regulating expression of MMP-2, which involves PI-3K and Erk1/2 protein kinase signaling...
  6. ncbi The role of beta1 integrin subfamily in anchorage-dependent apoptosis of breast carcinoma cells differing in multidrug resistance
    G E Morozevich
    Orekhovich Institute of Biomedical Chemistry, Russian Academy of Medical Sciences, Moscow, Russia
    Biochemistry (Mosc) 71:489-95. 2006
    ..These data show that activation of beta1-integrin signaling markedly upregulated anoikis of the adenocarcinoma cells...
  7. ncbi Integrin alphavbeta3 promotes anchorage-dependent apoptosis in human intestinal carcinoma cells
    N I Kozlova
    Institute of Biomedical Chemistry RAMS, Pogodinskaya str. 10, 119832 Moscow, Russia
    Oncogene 20:4710-7. 2001
    ..Thus, for the first time, our data prove that alphavbeta3 integrin can be an active transducer of apoptosis-stimulating signals generated in response to disruption of the cell-matrix contacts...
  8. ncbi [The role of alphavbeta3 integrin in changes of the invasive phenotype in CP-transformed fibroblasts with multiple drug resistance]
    G E Morozevich
    Institute of Biomedical Chemistry by V N Orekchovich, RAMS, Moscow
    Vopr Med Khim 48:111-20. 2002
    ..The role of integrin alpha v beta 3 in malignant phenotype and multiple drug resistance of tumor cells is discussed...
  9. doi C-MYC overexpression is required for continuous suppression of oncogene-induced senescence in melanoma cells
    D Zhuang
    Department of Cell Stress Biology, Roswell Park Cancer Institute, Buffalo, NY 14263, USA
    Oncogene 27:6623-34. 2008
    ..Our data suggest that one of the major functions of C-MYC overexpression in melanoma progression is to continuous suppress BRAF(V600E)- or NRAS(Q61R)-dependent senescence programs...