Sidhartha D Ray

Summary

Publications

  1. ncbi request reprint Curcumin exposure modulates multiple pro-apoptotic and anti-apoptotic signaling pathways to antagonize acetaminophen-induced toxicity
    Elida Bulku
    Division of Pharmaceutical Sciences, Manchester College School of Pharmacy, Fort Wayne, IN 46845, USA
    Curr Neurovasc Res 9:58-71. 2012
  2. ncbi request reprint Doxorubicin-induced in vivo nephrotoxicity involves oxidative stress- mediated multiple pro- and anti-apoptotic signaling pathways
    Tejas S Lahoti
    The Huck Institute of Life Sciences, Penn State University, University Park, PA 16802, USA
    Curr Neurovasc Res 9:282-95. 2012
  3. doi request reprint Long term exposure effect of a unique metabolic nutrition system containing a diverse group of phytochemicals on serum chemistry and genomic and non-genomic changes in the liver of female B6C3F1 mice
    Sidhartha D Ray
    Molecular Toxicology Laboratories, Division of Pharmaceutical Sciences, Arnold and Marie Schwartz College of Pharmacy and Health Sciences, Long Island University, Brooklyn, New York 11201, USA
    Phytother Res 22:458-71. 2008
  4. pmc A novel dietary supplement containing multiple phytochemicals and vitamins elevates hepatorenal and cardiac antioxidant enzymes in the absence of significant serum chemistry and genomic changes
    Elida Bulku
    Molecular Toxicology Laboratories, Division of Pharmaceutical Sciences, A and M Schwartz College of Pharmacy and Health Sciences, Long Island University, Brooklyn, NY, USA
    Oxid Med Cell Longev 3:129-44. 2010
  5. doi request reprint Silymarin modulates doxorubicin-induced oxidative stress, Bcl-xL and p53 expression while preventing apoptotic and necrotic cell death in the liver
    Nirav Patel
    Molecular Toxicology Laboratories, Division of Pharmaceutical Sciences, Arnold and Marie Schwartz College of Pharmacy and Health Sciences, Long Island University, Brooklyn, NY 11201, USA
    Toxicol Appl Pharmacol 245:143-52. 2010
  6. ncbi request reprint Pre-exposure to a novel nutritional mixture containing a series of phytochemicals prevents acetaminophen-induced programmed and unprogrammed cell deaths by enhancing BCL-XL expression and minimizing oxidative stress in the liver
    Sidhartha D Ray
    Molecular Toxicology Laboratories, Division of Pharmaceutical Sciences, Arnold and Marie Schwartz College of Pharmacy and Health Sciences, Long Island University, Brooklyn, NY 11201, USA
    Mol Cell Biochem 293:119-36. 2006

Collaborators

  • Sidney J Stohs
  • Elida Bulku
  • Tejas S Lahoti
  • Nirav Patel
  • Herb Halley
  • Lorraine Cicero
  • Robert Beckett
  • Venkatesh Thekkemadom
  • Darshan Patel
  • Tracy Brooks
  • Vishal Javia
  • George B Corcoran
  • Tejas Lahoti
  • Cecil Joseph
  • Daniel Zinkovsky
  • Inna Khodos
  • Payal Patel

Detail Information

Publications6

  1. ncbi request reprint Curcumin exposure modulates multiple pro-apoptotic and anti-apoptotic signaling pathways to antagonize acetaminophen-induced toxicity
    Elida Bulku
    Division of Pharmaceutical Sciences, Manchester College School of Pharmacy, Fort Wayne, IN 46845, USA
    Curr Neurovasc Res 9:58-71. 2012
    ..Based on the available literature, and these results, it seems likely that CUR may impart global protection in vivo against drug-induced liver injury by opposing several crucial events instrumental to both apoptosis and necrosis...
  2. ncbi request reprint Doxorubicin-induced in vivo nephrotoxicity involves oxidative stress- mediated multiple pro- and anti-apoptotic signaling pathways
    Tejas S Lahoti
    The Huck Institute of Life Sciences, Penn State University, University Park, PA 16802, USA
    Curr Neurovasc Res 9:282-95. 2012
    ....
  3. doi request reprint Long term exposure effect of a unique metabolic nutrition system containing a diverse group of phytochemicals on serum chemistry and genomic and non-genomic changes in the liver of female B6C3F1 mice
    Sidhartha D Ray
    Molecular Toxicology Laboratories, Division of Pharmaceutical Sciences, Arnold and Marie Schwartz College of Pharmacy and Health Sciences, Long Island University, Brooklyn, New York 11201, USA
    Phytother Res 22:458-71. 2008
    ..This study indicates that a caffeine and ephedra containing metabolic nutrition system is non-toxic and non-hepatotoxic in mice at up to 10x the human consumption dose of ephedra...
  4. pmc A novel dietary supplement containing multiple phytochemicals and vitamins elevates hepatorenal and cardiac antioxidant enzymes in the absence of significant serum chemistry and genomic changes
    Elida Bulku
    Molecular Toxicology Laboratories, Division of Pharmaceutical Sciences, A and M Schwartz College of Pharmacy and Health Sciences, Long Island University, Brooklyn, NY, USA
    Oxid Med Cell Longev 3:129-44. 2010
    ..Additionally, this study reiterates the potential benefits of exposure to a pharmacologically relevant combination of phytochemicals compared to a single phytochemical entity...
  5. doi request reprint Silymarin modulates doxorubicin-induced oxidative stress, Bcl-xL and p53 expression while preventing apoptotic and necrotic cell death in the liver
    Nirav Patel
    Molecular Toxicology Laboratories, Division of Pharmaceutical Sciences, Arnold and Marie Schwartz College of Pharmacy and Health Sciences, Long Island University, Brooklyn, NY 11201, USA
    Toxicol Appl Pharmacol 245:143-52. 2010
    ..These observations demonstrate the protective actions of SMN in liver, and raise the possibility that such protection may extend to other organs during Dox treatment including the heart...
  6. ncbi request reprint Pre-exposure to a novel nutritional mixture containing a series of phytochemicals prevents acetaminophen-induced programmed and unprogrammed cell deaths by enhancing BCL-XL expression and minimizing oxidative stress in the liver
    Sidhartha D Ray
    Molecular Toxicology Laboratories, Division of Pharmaceutical Sciences, Arnold and Marie Schwartz College of Pharmacy and Health Sciences, Long Island University, Brooklyn, NY 11201, USA
    Mol Cell Biochem 293:119-36. 2006
    ....