Research Topics
Species | H F S NegaardSummaryAffiliation: University of Oslo Country: Norway Publications
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Detail Information
Publications
Angiogenesis and hemostasis in hematological neoplasiasH Negaard
Department of Hematology, Ullevaal University Hospital, Kirkeveien 166, 0407 Oslo, Norway
Curr Drug Targets 6:683-99. 2005..A better understanding of this shared biology may reveal new targets, and will probably increase the safety of targeting the blood supply...
Hypercoagulability in patients with haematological neoplasia: no apparent initiation by tissue factorHelene F S Negaard
Department of Haematology, Ulleval University Hospital Trust, Oslo, Norway
Thromb Haemost 99:1040-8. 2008..In conclusion we found a hypercoagulable state in patients with haematological malignancy that did not seem to be initiated by TF...
Increased bone marrow microvascular density in haematological malignancies is associated with differential regulation of angiogenic factorsH F S Negaard
Department of Haematology, Ulleval University Hospital Trust, Oslo, Norway
Leukemia 23:162-9. 2009..VEGF, IL-6 and IL-8 seem to contribute to the malignant phenotype...
Alterations in regulators of the extracellular matrix in non-Hodgkin lymphomasHelene F S Negaard
Department of Haematology, Oslo University Hospital Ulleval, Oslo, Norway
Leuk Lymphoma 50:998-1004. 2009..After therapy, serum MMP-2 increased while MMP-9 decreased (P < 0.05), the others being unchanged. Several compounds affecting ECM may be involved in angiogenic activity in NHL...
Increased acquired activated protein C resistance in unselected patients with hematological malignanciesH F S Negaard
Department of Hematology, Ulleval University Hospital Trust, Oslo, Norway
J Thromb Haemost 6:1482-7. 2008..Acquired activated protein C (APC) resistance may be another mechanism responsible for such hypercoagulation, and has been demonstrated in patients with solid tumors, but not in patients with hematological malignancy...
Decreased lung cancer survival with hormone-replacement therapy: caused by a decreased tissue factor pathway inhibitor level?Helene F S Negaard
J Clin Oncol 24:2683-4; author reply 2684. 2006
