Kathrin Sabine Ellesat

Summary

Affiliation: University of Oslo
Country: Norway

Publications

  1. doi Cytotoxicity of atorvastatin and simvastatin on primary rainbow trout (Oncorhynchus mykiss) hepatocytes
    Kathrin Sabine Ellesat
    Department of Biology, University of Oslo, Oslo, Norway
    Toxicol In Vitro 24:1610-8. 2010
  2. doi Atorvastatin up-regulate toxicologically relevant genes in rainbow trout gills
    Kathrin Sabine Ellesat
    Department of Biology, University of Oslo, P O Box 1066, Blindern, 0316, Oslo, Norway
    Ecotoxicology 21:1841-56. 2012
  3. doi Species-dependent sensitivity to contaminants: an approach using primary hepatocyte cultures with three marine fish species
    Kathrin Sabine Ellesat
    Department of Biology, University of Oslo, P O Box 1066 Blindern, N 0316 Oslo, Norway
    Mar Environ Res 72:216-24. 2011

Detail Information

Publications3

  1. doi Cytotoxicity of atorvastatin and simvastatin on primary rainbow trout (Oncorhynchus mykiss) hepatocytes
    Kathrin Sabine Ellesat
    Department of Biology, University of Oslo, Oslo, Norway
    Toxicol In Vitro 24:1610-8. 2010
    ..Simvastatin was more toxic than atorvastatin and the lactone form more toxic than the acid form. Exposure time affected atorvastatin and ortho-hydroxy atorvastatin lactone but not simvastatin toxicity...
  2. doi Atorvastatin up-regulate toxicologically relevant genes in rainbow trout gills
    Kathrin Sabine Ellesat
    Department of Biology, University of Oslo, P O Box 1066, Blindern, 0316, Oslo, Norway
    Ecotoxicology 21:1841-56. 2012
    ..The pattern of differentially expressed genes in rainbow trout gills differ from responses previously observed in mammalian tissues following statin exposure...
  3. doi Species-dependent sensitivity to contaminants: an approach using primary hepatocyte cultures with three marine fish species
    Kathrin Sabine Ellesat
    Department of Biology, University of Oslo, P O Box 1066 Blindern, N 0316 Oslo, Norway
    Mar Environ Res 72:216-24. 2011
    ..Flatfish hepatocytes were more susceptible to copper and statin exposure than hepatocytes from cod. This study has shown that species-dependent differences in contaminant sensitivity can be investigated using primary hepatocyte cultures...