T Aarvak

Summary

Affiliation: University of Oslo
Country: Norway

Publications

  1. ncbi request reprint IL-17 is produced by some proinflammatory Th1/Th0 cells but not by Th2 cells
    T Aarvak
    Institute of Immunology, Laboratory of Rheumatology Research, The National Hospital, Oslo, Norway
    J Immunol 162:1246-51. 1999
  2. pmc Cell-cell interactions in synovitis: antigen presenting cells and T cell interaction in rheumatoid arthritis
    T Aarvak
    Institute of Immunology, Laboratory of Rheumatology Research, The National Hospital, Oslo, Norway
    Arthritis Res 3:13-7. 2001
  3. ncbi request reprint Switch in chemokine receptor phenotype on memory T cells without a change in the cytokine phenotype
    T Aarvak
    Institute of Immunology, Laboratory of Rheumatology Research and the Center for Rheumatic Diseases, The National Hospital, Oslo, Norway
    Scand J Immunol 54:100-8. 2001
  4. ncbi request reprint T-cell responses to viral, bacterial and protozoan antigens in rheumatoid inflammation. Selective migration of T cells to synovial tissue
    K R Shadidi
    The National Hospital, Institute of Immunology, Department of Rheumatology Research, Oslo, Norway
    Rheumatology (Oxford) 40:1120-5. 2001
  5. ncbi request reprint The chemokines CCL5, CCL2 and CXCL12 play significant roles in the migration of Th1 cells into rheumatoid synovial tissue
    K R Shadidi
    Rikshospitalet University Hospital, Institute of Immunology, Section of Rheumatology Research, Oslo, Norway
    Scand J Immunol 57:192-8. 2003
  6. ncbi request reprint Association of antigen specificity and migratory capacity of memory T cells in rheumatoid arthritis
    K R Shadidi
    Rikshospitalet University Hospital, Institute of Immunology, Department of Rheumatology Research, Oslo, Norway
    Scand J Immunol 55:274-83. 2002
  7. ncbi request reprint Potential contribution of IL-17-producing Th(1)cells to defective repair activity in joint inflammation: partial correction with Th(2)-promoting conditions
    M Chabaud
    Departments of Immunology and Rheumatology, , 69437 Lyon Cedex 03, France
    Cytokine 13:113-8. 2001

Collaborators

Detail Information

Publications7

  1. ncbi request reprint IL-17 is produced by some proinflammatory Th1/Th0 cells but not by Th2 cells
    T Aarvak
    Institute of Immunology, Laboratory of Rheumatology Research, The National Hospital, Oslo, Norway
    J Immunol 162:1246-51. 1999
    ..Thus, IL-17 may define a new subset of T cells, and IL-17 production appears to be a mechanism for Th1/Th0 cells, the most frequent Th subtype present in the rheumatoid synovium, to contribute to the local inflammatory reactions...
  2. pmc Cell-cell interactions in synovitis: antigen presenting cells and T cell interaction in rheumatoid arthritis
    T Aarvak
    Institute of Immunology, Laboratory of Rheumatology Research, The National Hospital, Oslo, Norway
    Arthritis Res 3:13-7. 2001
    ..Blocking of the signaling pathways involved in APC-T cell interactions might provide a specific immuno-therapeutic approach for the treatment of RA...
  3. ncbi request reprint Switch in chemokine receptor phenotype on memory T cells without a change in the cytokine phenotype
    T Aarvak
    Institute of Immunology, Laboratory of Rheumatology Research and the Center for Rheumatic Diseases, The National Hospital, Oslo, Norway
    Scand J Immunol 54:100-8. 2001
    ....
  4. ncbi request reprint T-cell responses to viral, bacterial and protozoan antigens in rheumatoid inflammation. Selective migration of T cells to synovial tissue
    K R Shadidi
    The National Hospital, Institute of Immunology, Department of Rheumatology Research, Oslo, Norway
    Rheumatology (Oxford) 40:1120-5. 2001
    ..CONCLUSIONS: The results indicate a selective migration of T cells of given specificities to the inflamed rheumatoid synovium...
  5. ncbi request reprint The chemokines CCL5, CCL2 and CXCL12 play significant roles in the migration of Th1 cells into rheumatoid synovial tissue
    K R Shadidi
    Rikshospitalet University Hospital, Institute of Immunology, Section of Rheumatology Research, Oslo, Norway
    Scand J Immunol 57:192-8. 2003
    ..This indicates that CCL5, CCL2 and CXCL12 play significant roles in attracting Th1 cells towards the RA ST, and may prove potent targets for obstructing T-cell migration to the synovium...
  6. ncbi request reprint Association of antigen specificity and migratory capacity of memory T cells in rheumatoid arthritis
    K R Shadidi
    Rikshospitalet University Hospital, Institute of Immunology, Department of Rheumatology Research, Oslo, Norway
    Scand J Immunol 55:274-83. 2002
    ..Blocking of single chemokines substantially reduced the migratory capacity of memory T cells to ST cell-derived supernatant indicating unique roles for each chemokine receptor in the process of T cell migration...
  7. ncbi request reprint Potential contribution of IL-17-producing Th(1)cells to defective repair activity in joint inflammation: partial correction with Th(2)-promoting conditions
    M Chabaud
    Departments of Immunology and Rheumatology, , 69437 Lyon Cedex 03, France
    Cytokine 13:113-8. 2001
    ..As rheumatoid synovium is infiltrated by Th(1)cells, local cell interactions result in a pro-inflammatory pattern with defective repair, which can be reversed at least in part, by a Th(2)pattern...