Torsten Waldminghaus

Summary

Affiliation: Institute for Cancer Research
Country: Norway

Publications

  1. pmc ChIP on Chip: surprising results are often artifacts
    Torsten Waldminghaus
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radium Hospital, Oslo University Hospital and University of Oslo, 0310 Oslo, Norway
    BMC Genomics 11:414. 2010
  2. pmc Replication fork movement and methylation govern SeqA binding to the Escherichia coli chromosome
    Torsten Waldminghaus
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radium Hospital, Oslo University Hospital and University of Oslo, 0310 Oslo, Norway
    Nucleic Acids Res 40:5465-76. 2012
  3. doi request reprint The Escherichia coli SeqA protein
    Torsten Waldminghaus
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radium Hospital, Rikshospitalet, University of Oslo, 0310 Oslo, Norway
    Plasmid 61:141-50. 2009
  4. doi request reprint The G157C mutation in the Escherichia coli sliding clamp specifically affects initiation of replication
    Line Johnsen
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radium Hospital, Oslo University Hospital, Oslo, Norway
    Mol Microbiol 79:433-46. 2011
  5. doi request reprint Replication patterns and organization of replication forks in Vibrio cholerae
    Caroline Stokke
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radiumhospital, Oslo University Hospital, Oslo, Norway
    Microbiology 157:695-708. 2011

Collaborators

Detail Information

Publications5

  1. pmc ChIP on Chip: surprising results are often artifacts
    Torsten Waldminghaus
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radium Hospital, Oslo University Hospital and University of Oslo, 0310 Oslo, Norway
    BMC Genomics 11:414. 2010
    ..The method of chromatin immunoprecipitation combined with microarrays (ChIP-Chip) is a powerful tool for genome-wide analysis of protein binding. However, a high background signal is a common phenomenon...
  2. pmc Replication fork movement and methylation govern SeqA binding to the Escherichia coli chromosome
    Torsten Waldminghaus
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radium Hospital, Oslo University Hospital and University of Oslo, 0310 Oslo, Norway
    Nucleic Acids Res 40:5465-76. 2012
    ..The finding suggests simultaneous origin de-sequestration and loss of SeqA from old replication forks...
  3. doi request reprint The Escherichia coli SeqA protein
    Torsten Waldminghaus
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radium Hospital, Rikshospitalet, University of Oslo, 0310 Oslo, Norway
    Plasmid 61:141-50. 2009
    ..SeqA can also bind, with less affinity, to fully methylated origins and affect timing of "primary" initiations. In addition to its roles in replication, the SeqA protein may also act in chromosome organization and gene regulation...
  4. doi request reprint The G157C mutation in the Escherichia coli sliding clamp specifically affects initiation of replication
    Line Johnsen
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radium Hospital, Oslo University Hospital, Oslo, Norway
    Mol Microbiol 79:433-46. 2011
    ..The result indicates that some aspects of β clamp activity are specific to the origin. It is possible that the origin specific activities of β contribute to regulation of initiation frequency...
  5. doi request reprint Replication patterns and organization of replication forks in Vibrio cholerae
    Caroline Stokke
    Department of Cell Biology, Institute for Cancer Research, The Norwegian Radiumhospital, Oslo University Hospital, Oslo, Norway
    Microbiology 157:695-708. 2011
    ..The increased degree of fork organization during rapid growth may be a means by which correct segregation of daughter molecules is facilitated...