Peter J Nelson

Summary

Publications

  1. doi request reprint The contentious ontogeny of fibrosis in the kidney
    Peter J Nelson
    Division of Nephrology and Kidney Research Institute, University of Washington, Medical Centre, Seattle, Washington 98195, USA
    Kidney Int 84:14-5. 2013
  2. doi request reprint Glomerular disease in 2011: New clues to environmental influences in glomerular disease
    Peter J Nelson
    Division of Nephrology, University of Washington Medical Center, 1959 NE Pacific Street, Seattle, WA 98195, USA
    Nat Rev Nephrol 8:65-6. 2012
  3. pmc The renal mononuclear phagocytic system
    Peter J Nelson
    University of Washington, Seattle, Washington, USA
    J Am Soc Nephrol 23:194-203. 2012
  4. doi request reprint Resident renal mononuclear phagocytes comprise five discrete populations with distinct phenotypes and functions
    Takahisa Kawakami
    Division of Nephrology, University of Washington, Seattle, WA 98109
    J Immunol 191:3358-72. 2013
  5. pmc Retinoids augment the expression of podocyte proteins by glomerular parietal epithelial cells in experimental glomerular disease
    Jiong Zhang
    Division of Nephrology, Department of Medicine, University of Washington School of Medicine, Seattle, Wash 98195 6521, USA
    Nephron Exp Nephrol 121:e23-37. 2012
  6. pmc Collapsing glomerulopathy: beyond serendipity in mouse genetics
    Peter J Nelson
    Division of Nephrology, Smilow Research Center, New York University School of Medicine, New York, NY 10016, USA
    Kidney Int 75:353-5. 2009
  7. doi request reprint SSeCKS sequesters cyclin D1 in glomerular parietal epithelial cells and influences proliferative injury in the glomerulus
    Bettina Burnworth
    Division of Nephrology and Kidney Research Institute, University of Washington, Health Sciences Center, Seattle, WA, USA
    Lab Invest 92:499-510. 2012
  8. ncbi request reprint Dendritic cells in the kidney
    Rohan John
    Division of Nephrology, New York University School of Medicine, New York, NY 10016, USA
    J Am Soc Nephrol 18:2628-35. 2007

Collaborators

Detail Information

Publications8

  1. doi request reprint The contentious ontogeny of fibrosis in the kidney
    Peter J Nelson
    Division of Nephrology and Kidney Research Institute, University of Washington, Medical Centre, Seattle, Washington 98195, USA
    Kidney Int 84:14-5. 2013
    ..Reich et al. now contend that circulating bone marrow-derived fibrocytes independent of monocyte lineages are an important source of fibrogenic cells recruited to the kidney following injury. ..
  2. doi request reprint Glomerular disease in 2011: New clues to environmental influences in glomerular disease
    Peter J Nelson
    Division of Nephrology, University of Washington Medical Center, 1959 NE Pacific Street, Seattle, WA 98195, USA
    Nat Rev Nephrol 8:65-6. 2012
    ..However, much less is known about environmental factors that may initiate or propagate podocyte injury. Seminal reports in 2011 provided new mechanistic insights into how this may occur...
  3. pmc The renal mononuclear phagocytic system
    Peter J Nelson
    University of Washington, Seattle, Washington, USA
    J Am Soc Nephrol 23:194-203. 2012
    ..Gaps and inconsistencies in current knowledge are used to create a roadmap of key questions to be answered in future research...
  4. doi request reprint Resident renal mononuclear phagocytes comprise five discrete populations with distinct phenotypes and functions
    Takahisa Kawakami
    Division of Nephrology, University of Washington, Seattle, WA 98109
    J Immunol 191:3358-72. 2013
    ..In summary, resident kidney MPCs comprise discrete subpopulations, which cannot be simply classified into the conventional entities, and they produce anti-inflammatory and tissue-homeostatic factors to differing degrees. ..
  5. pmc Retinoids augment the expression of podocyte proteins by glomerular parietal epithelial cells in experimental glomerular disease
    Jiong Zhang
    Division of Nephrology, Department of Medicine, University of Washington School of Medicine, Seattle, Wash 98195 6521, USA
    Nephron Exp Nephrol 121:e23-37. 2012
    ..Because retinoids improve many forms of experimental glomerular disease characterized by podocyte injury and loss, we asked if all-trans retinoic acid (ATRA) induces parietal epithelial cells to express podocyte proteins...
  6. pmc Collapsing glomerulopathy: beyond serendipity in mouse genetics
    Peter J Nelson
    Division of Nephrology, Smilow Research Center, New York University School of Medicine, New York, NY 10016, USA
    Kidney Int 75:353-5. 2009
    ..Chan et al. combine reverse and forward genetic techniques in mice toward understanding this complexity with HIV and identify candidate genetic modifiers of collapsing glomerulopathy...
  7. doi request reprint SSeCKS sequesters cyclin D1 in glomerular parietal epithelial cells and influences proliferative injury in the glomerulus
    Bettina Burnworth
    Division of Nephrology and Kidney Research Institute, University of Washington, Health Sciences Center, Seattle, WA, USA
    Lab Invest 92:499-510. 2012
    ..These results suggest that SSeCKS controls the localization and activity of cyclin D1 in PECs and influences proliferative injury in the glomerulus...
  8. ncbi request reprint Dendritic cells in the kidney
    Rohan John
    Division of Nephrology, New York University School of Medicine, New York, NY 10016, USA
    J Am Soc Nephrol 18:2628-35. 2007
    ..Together, these studies suggest that renal DC play a critical role in the health and disease of the kidney, opening the way to direct targeting of renal DC for therapeutic benefit...