Ralf Marienfeld

Summary

Publications

  1. ncbi request reprint RelB forms transcriptionally inactive complexes with RelA/p65
    Ralf Marienfeld
    Section of Immunobiology and Department of Molecular Biophysics and Biochemistry, Yale University Medical School, New Haven, Connecticut 06520, USA
    J Biol Chem 278:19852-60. 2003
  2. ncbi request reprint Critical role of RelB serine 368 for dimerization and p100 stabilization
    Harald Jakob Maier
    Department of Physiological Chemistry, Ulm University, Ulm 89081, Germany
    J Biol Chem 278:39242-50. 2003
  3. ncbi request reprint Sustained JNK activation in response to tumor necrosis factor is mediated by caspases in a cell type-specific manner
    Andreas Wicovsky
    Department of Molecular Internal Medicine, Medical Clinic and Polyclinic II, University of Wurzburg, Rontgenring 11, 97070 Wurzburg, Germany
    J Biol Chem 282:2174-83. 2007
  4. doi request reprint Identification of lysine residues critical for the transcriptional activity and polyubiquitination of the NF-kappaB family member RelB
    Julia Leidner
    Institute of Physiological Chemistry, Ulm University, Ulm, Germany
    Biochem J 416:117-27. 2008

Collaborators

Detail Information

Publications4

  1. ncbi request reprint RelB forms transcriptionally inactive complexes with RelA/p65
    Ralf Marienfeld
    Section of Immunobiology and Department of Molecular Biophysics and Biochemistry, Yale University Medical School, New Haven, Connecticut 06520, USA
    J Biol Chem 278:19852-60. 2003
    ..Taken together, our findings demonstrate that sequestration of RelA in transcriptionally inactive RelA.RelB complexes provides a molecular mechanism that may explain the repressive role of RelB on NF-kappaB-dependent gene expression...
  2. ncbi request reprint Critical role of RelB serine 368 for dimerization and p100 stabilization
    Harald Jakob Maier
    Department of Physiological Chemistry, Ulm University, Ulm 89081, Germany
    J Biol Chem 278:39242-50. 2003
    ..Wild type RelB, but not mutant RelB, prolonged p100 half-life. We therefore suggest an inhibitory effect of RelB on p100 processing, which is possibly regulated in a signal-dependent manner...
  3. ncbi request reprint Sustained JNK activation in response to tumor necrosis factor is mediated by caspases in a cell type-specific manner
    Andreas Wicovsky
    Department of Molecular Internal Medicine, Medical Clinic and Polyclinic II, University of Wurzburg, Rontgenring 11, 97070 Wurzburg, Germany
    J Biol Chem 282:2174-83. 2007
    ..Thus, sustained JNK activation by TNF has no obligate role in TNF-induced cell death and is mediated by caspases and reactive oxygen species in a cell type-specific manner...
  4. doi request reprint Identification of lysine residues critical for the transcriptional activity and polyubiquitination of the NF-kappaB family member RelB
    Julia Leidner
    Institute of Physiological Chemistry, Ulm University, Ulm, Germany
    Biochem J 416:117-27. 2008
    ..Collectively, these results imply a dual role of RelB polyubiquitination for the stability and activity of this transcription factor...