Yuchi Li

Summary

Publications

  1. ncbi request reprint Inhibition of c-Jun N-terminal kinase pathway improves cell viability in response to oxidant injury
    Yuchi Li
    The CardioPulmonary Research Institute, Winthrop University Hospital, Suite 505, 222 Station Plaza N, Mineola, NY 11501, USA
    Am J Respir Cell Mol Biol 29:779-83. 2003
  2. doi request reprint Superoxide dismutase attenuates hyperoxia-induced interleukin-8 induction via AP-1
    Ansamma Joseph
    CardioPulmonary Research Institute, Department of Medicine, Winthrop University Hospital, State University of New York Stony Brook School of Medicine, Mineola, NY 11501, USA
    Free Radic Biol Med 45:1143-9. 2008
  3. ncbi request reprint Mitochondrial localization of catalase provides optimal protection from H2O2-induced cell death in lung epithelial cells
    Yuko Arita
    Department of Pediatrics, CardioPulmonary Research Institute, Winthrop University Hospital, SUNY Stony Brook School of Medicine, Suite 505, 222 Station Plaza North, Mineola, NY 11501, USA
    Am J Physiol Lung Cell Mol Physiol 290:L978-86. 2006
  4. pmc Antioxidants improve antibacterial function in hyperoxia-exposed macrophages
    Yuko Arita
    CardioPulmonary Research Institute, Winthrop University Hospital, State University of New York at Stony Brook School of Medicine, Mineola, NY 11501, USA
    Free Radic Biol Med 42:1517-23. 2007
  5. ncbi request reprint Effects of transgene expression of superoxide dismutase and glutathione peroxidase on pulmonary epithelial cell growth in hyperoxia
    Hshi Chi Koo
    CardioPulmonary Research Institute, Winthrop University Hospital, 222 Station Plaza North, Suite 604, Mineola, NY 11501, USA
    Am J Physiol Lung Cell Mol Physiol 288:L718-26. 2005
  6. ncbi request reprint Superoxide dismutase moderates basal and induced bacterial adherence and interleukin-8 expression in airway epithelial cells
    Yuko Arita
    CardioPulmonary Research Institute, Department of Medicine, Division of Pulmonary and Critical Care Medicine, Winthrop University Hospital, SUNY Stony Brook School of Medicine, Mineola, NY 11501, USA
    Am J Physiol Lung Cell Mol Physiol 287:L1199-206. 2004

Collaborators

  • Jonathan M Davis
  • A Chander
  • Hshi Chi Koo
  • Yuko Arita
  • Ansamma Joseph
  • Jeffrey A Kazzaz
  • Simcha Pollack
  • S Hella Harkness
  • J Andres Melendez
  • Marlene S Strayer
  • Dimitrios Hatzis
  • Harry Opsimos
  • Philip L Ballard
  • Thomas A Palaia

Detail Information

Publications6

  1. ncbi request reprint Inhibition of c-Jun N-terminal kinase pathway improves cell viability in response to oxidant injury
    Yuchi Li
    The CardioPulmonary Research Institute, Winthrop University Hospital, Suite 505, 222 Station Plaza N, Mineola, NY 11501, USA
    Am J Respir Cell Mol Biol 29:779-83. 2003
    ..Inhibition of the JNK pathway in this manner prevented hyperoxic and H2O2-induced cell death. These results demonstrate that hyperoxic cell death is pathway-driven and that both modes of death involve the JNK signaling pathway...
  2. doi request reprint Superoxide dismutase attenuates hyperoxia-induced interleukin-8 induction via AP-1
    Ansamma Joseph
    CardioPulmonary Research Institute, Department of Medicine, Winthrop University Hospital, State University of New York Stony Brook School of Medicine, Mineola, NY 11501, USA
    Free Radic Biol Med 45:1143-9. 2008
    ..JNK1 RNAi also moderated IL-8 induction. The data indicate that activation of JNK1/AP-1 and subsequent IL-8 induction in hyperoxia are mediated by intracellular ROS, with SOD having significant protective effects...
  3. ncbi request reprint Mitochondrial localization of catalase provides optimal protection from H2O2-induced cell death in lung epithelial cells
    Yuko Arita
    Department of Pediatrics, CardioPulmonary Research Institute, Winthrop University Hospital, SUNY Stony Brook School of Medicine, Suite 505, 222 Station Plaza North, Mineola, NY 11501, USA
    Am J Physiol Lung Cell Mol Physiol 290:L978-86. 2006
    ..This has important implications in antioxidant supplementation trials to prevent ROS-induced lung injury in critically ill patients...
  4. pmc Antioxidants improve antibacterial function in hyperoxia-exposed macrophages
    Yuko Arita
    CardioPulmonary Research Institute, Winthrop University Hospital, State University of New York at Stony Brook School of Medicine, Mineola, NY 11501, USA
    Free Radic Biol Med 42:1517-23. 2007
    ....
  5. ncbi request reprint Effects of transgene expression of superoxide dismutase and glutathione peroxidase on pulmonary epithelial cell growth in hyperoxia
    Hshi Chi Koo
    CardioPulmonary Research Institute, Winthrop University Hospital, 222 Station Plaza North, Suite 604, Mineola, NY 11501, USA
    Am J Physiol Lung Cell Mol Physiol 288:L718-26. 2005
    ..This has important implications for clinical trials in preterm infants receiving SOD supplementation to prevent acute and chronic lung injury...
  6. ncbi request reprint Superoxide dismutase moderates basal and induced bacterial adherence and interleukin-8 expression in airway epithelial cells
    Yuko Arita
    CardioPulmonary Research Institute, Department of Medicine, Division of Pulmonary and Critical Care Medicine, Winthrop University Hospital, SUNY Stony Brook School of Medicine, Mineola, NY 11501, USA
    Am J Physiol Lung Cell Mol Physiol 287:L1199-206. 2004
    ..Therapeutic intervention strategies designed to prevent accumulation of intracellular reactive oxygen species may reduce opportunistic pulmonary infections...