Masocha W, Amin D, Kristensson K, Rottenberg M. Differential invasion of Trypanosoma brucei brucei and lymphocytes into the brain of C57BL/6 and 129Sv/Ev mice. Scand J Immunol. 2008;68:484-91 pubmed publisher
..b. brucei into the brain parenchyma, which is paralleled by the severity of inflammation in the brain and course of the disease, but not by parasitaemia nor by antibody titres. ..
Masocha W. CD200 receptors are differentially expressed and modulated by minocycline in the brain during Trypanosoma brucei infection. J Neuroimmunol. 2010;226:59-65 pubmed publisher
..This suggests that CD200RLa might play a role in microglia/macrophage activation during trypanosome infection. ..
Masocha W, Kristensson K. Human African trypanosomiasis: How do the parasites enter and cause dysfunctions of the nervous system in murine models?. Brain Res Bull. 2018;: pubmed publisher
Masocha W, Parvathy S, Pavarthy S. Assessment of weight bearing changes and pharmacological antinociception in mice with LPS-induced monoarthritis using the Catwalk gait analysis system. Life Sci. 2009;85:462-9 pubmed publisher
..This study shows that the Catwalk gait analysis system can be used to objectively quantify LPS-induced monoarthritis weight bearing changes in all four limbs and evaluate pharmacological antinociception in freely moving mice. ..
Masocha W. Systemic lipopolysaccharide (LPS)-induced microglial activation results in different temporal reduction of CD200 and CD200 receptor gene expression in the brain. J Neuroimmunol. 2009;214:78-82 pubmed publisher
..Thus, LPS-induced changes in CD200-CD200R equilibrium might keep microglia chronically activated. Minocycline does not effectively inhibit microglia activation induced by high-dose LPS. ..