Affiliation: College of Medicine
- MLN51 and GM-CSF involvement in the proliferation of fibroblast-like synoviocytes in the pathogenesis of rheumatoid arthritisJinah Jang
Department of Biological Science, Sungkyunkwan University, 300 Cheoncheon dong, Suwon, Gyeonggi 440 746, Korea
Arthritis Res Ther 8:R170. 2006..Taken together, our results imply that MLN51, induced by GM-CSF, is important in the proliferation of RA FLSs in the pathogenesis of RA...
- FLIP and MAPK play crucial roles in the MLN51-mediated hyperproliferation of fibroblast-like synoviocytes in the pathogenesis of rheumatoid arthritisJu Eun Ha
Department of Biological Science, Sungkyunkwan University, Suwon, Gyeonggi Do, South Korea
FEBS J 275:3546-55. 2008..These results imply that GM-CSF signaling activates mitogen-activated protein kinase, followed by the upregulation of MLN51 and FLICE-inhibitory protein, resulting in fibroblast-like synoviocyte hyperplasia in rheumatoid arthritis...
- CISH is induced during DC development and regulates DC-mediated CTL activationMohammad Alam Miah
Department of Biological Sciences, Sungkyunkwan University, Suwon, Gyeonggi Do, South Korea
Eur J Immunol 42:58-68. 2012..Taken together, our data suggest that CISH expression at the later stage of DC development triggers the shutdown of DC progenitor cell proliferation and facilitates DC differentiation into a potent stimulator of CTLs...