Yukiko Kagohashi

Summary

Publications

  1. doi request reprint Diet with a low n-6/n-3 essential fatty acid ratio when started immediately after the onset of overt diabetes prolongs survival of type 1 diabetes model NOD mice
    Yukiko Kagohashi
    Department of Health and Nutrition, The University of Shimane, Matsue, Shimane, Japan
    Congenit Anom (Kyoto) 50:226-31. 2010
  2. doi request reprint Maternal dietary n-6/n-3 fatty acid ratio affects type 1 diabetes development in the offspring of non-obese diabetic mice
    Yukiko Kagohashi
    Department of Health and Nutrition, The University of Shimane, Matsue, Shimane, Japan
    Congenit Anom (Kyoto) 50:212-20. 2010
  3. ncbi request reprint Maternal environment affects endogenous virus induction in the offspring of type 1 diabetes model non-obese diabetic mice
    Yukiko Kagohashi
    Department of Developmental Biology, Faculty of Medicine, Shimane University, Izumo, Shimane, Japan
    Congenit Anom (Kyoto) 45:80-4. 2005
  4. ncbi request reprint Maternal factors in a model of type 1 diabetes differentially affect the development of insulitis and overt diabetes in offspring
    Yukiko Kagohashi
    Department of Developmental Biology, Faculty of Medicine, Shimane University, Izumo, Shimane, 693 8501, Japan
    Diabetes 54:2026-31. 2005
  5. doi request reprint Individual variation in organ histogenesis as a causative factor in the developmental origins of health and disease: unnoticed congenital anomalies?
    Hiroki Otani
    Department of Developmental Biology, Faculty of Medicine, Shimane University, Izumo, Shimane, Japan
    Congenit Anom (Kyoto) 50:205-11. 2010

Detail Information

Publications5

  1. doi request reprint Diet with a low n-6/n-3 essential fatty acid ratio when started immediately after the onset of overt diabetes prolongs survival of type 1 diabetes model NOD mice
    Yukiko Kagohashi
    Department of Health and Nutrition, The University of Shimane, Matsue, Shimane, Japan
    Congenit Anom (Kyoto) 50:226-31. 2010
    ..These findings indicate that a diet with a low n-6/n-3 ratio prolongs the 'honeymoon' period by retaining the β cell mass, suggesting its potential therapeutic merit...
  2. doi request reprint Maternal dietary n-6/n-3 fatty acid ratio affects type 1 diabetes development in the offspring of non-obese diabetic mice
    Yukiko Kagohashi
    Department of Health and Nutrition, The University of Shimane, Matsue, Shimane, Japan
    Congenit Anom (Kyoto) 50:212-20. 2010
    ..The present findings suggest that n-6/n-3 ratio of the maternal diet during gestation and lactation rather than that of offspring after weaning strongly affects the development of overt diabetes in NOD mice...
  3. ncbi request reprint Maternal environment affects endogenous virus induction in the offspring of type 1 diabetes model non-obese diabetic mice
    Yukiko Kagohashi
    Department of Developmental Biology, Faculty of Medicine, Shimane University, Izumo, Shimane, Japan
    Congenit Anom (Kyoto) 45:80-4. 2005
    ..These findings suggest that the observed virus is endogenous and that maternal environmental factors, including hormone levels, affect the induction of endogenous viruses and cause the earlier onset of insulitis...
  4. ncbi request reprint Maternal factors in a model of type 1 diabetes differentially affect the development of insulitis and overt diabetes in offspring
    Yukiko Kagohashi
    Department of Developmental Biology, Faculty of Medicine, Shimane University, Izumo, Shimane, 693 8501, Japan
    Diabetes 54:2026-31. 2005
    ..The present study indicates that altered maternal factors modify the immune response to islets, which in turn might affect the pathogenic course from insulitis to overt diabetes...
  5. doi request reprint Individual variation in organ histogenesis as a causative factor in the developmental origins of health and disease: unnoticed congenital anomalies?
    Hiroki Otani
    Department of Developmental Biology, Faculty of Medicine, Shimane University, Izumo, Shimane, Japan
    Congenit Anom (Kyoto) 50:205-11. 2010
    ....