Y Endoh

Summary

Affiliation: Yamagata University
Country: Japan

Publications

  1. ncbi request reprint Esophageal adenocarcinoma that probably originated in the esophageal gland duct: a case report
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Japan
    Pathol Int 49:156-9. 1999
  2. ncbi request reprint Apocrine adenosis of the breast: clonal evidence of neoplasia
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    Histopathology 38:221-4. 2001
  3. pmc Frequent hypermethylation of the hMLH1 gene promoter in differentiated-type tumors of the stomach with the gastric foveolar phenotype
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Yamagata Niigata University School of Medicine, Niigata, Japan
    Am J Pathol 157:717-22. 2000
  4. ncbi request reprint Cellular phenotypes of differentiated-type adenocarcinomas and precancerous lesions of the stomach are dependent on the genetic pathways
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Japan
    J Pathol 191:257-63. 2000
  5. ncbi request reprint The common 18-base pair deletion at codons 418-423 of the E-cadherin gene in differentiated-type adenocarcinomas and intramucosal precancerous lesions of the stomach with the features of gastric foveolar epithelium
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    J Pathol 189:201-6. 1999
  6. pmc Hypermethylation of the hMLH1 gene promoter in solitary and multiple gastric cancers with microsatellite instability
    K Sakata
    Department of Pathology, Yamagata University School of Medicine, Iida nishi 2 2 2, Yamagata 990 9585, Japan
    Br J Cancer 86:564-7. 2002
  7. ncbi request reprint Molecular characterization of undifferentiated-type gastric carcinoma
    G Tamura
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    Lab Invest 81:593-8. 2001
  8. ncbi request reprint Mutations in mitochondrial control region DNA in gastric tumours of Japanese patients
    G Tamura
    Department of Pathology, Yamagata University School of Medicine, Japan
    Eur J Cancer 35:316-9. 1999
  9. ncbi request reprint Microsatellite alterations and target gene mutations in the early stages of multiple gastric cancer
    S Ogata
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    J Pathol 194:334-40. 2001
  10. ncbi request reprint Absence of BAT-26 instability in gastric intestinal metaplasia
    Z Jin
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    Pathol Int 51:473-5. 2001

Collaborators

Detail Information

Publications11

  1. ncbi request reprint Esophageal adenocarcinoma that probably originated in the esophageal gland duct: a case report
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Japan
    Pathol Int 49:156-9. 1999
    ..Barrett's metaplastic epithelium or ectopic gastric mucosa was not found around the tumor. It is strongly suggested that this unique carcinoma is derived from the esophageal gland ducts...
  2. ncbi request reprint Apocrine adenosis of the breast: clonal evidence of neoplasia
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    Histopathology 38:221-4. 2001
    ..We report here a case of apocrine adenosis of the breast in a 66-year-old woman. To clarify the nature of this lesion, we examined it by clonal analysis...
  3. pmc Frequent hypermethylation of the hMLH1 gene promoter in differentiated-type tumors of the stomach with the gastric foveolar phenotype
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Yamagata Niigata University School of Medicine, Niigata, Japan
    Am J Pathol 157:717-22. 2000
    ..Hypermethylation of hMLH1 promoter is an initial, vital event in the development of foveolar-type tumors of the stomach...
  4. ncbi request reprint Cellular phenotypes of differentiated-type adenocarcinomas and precancerous lesions of the stomach are dependent on the genetic pathways
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Japan
    J Pathol 191:257-63. 2000
    ..The 'suppressor pathway', represented by p53 alteration, could participate in the tumourigenesis of the CIM-type, but is rare in foveolar-type tumours...
  5. ncbi request reprint The common 18-base pair deletion at codons 418-423 of the E-cadherin gene in differentiated-type adenocarcinomas and intramucosal precancerous lesions of the stomach with the features of gastric foveolar epithelium
    Y Endoh
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    J Pathol 189:201-6. 1999
    ..This mutation was previously reported in an advanced carcinoma of undifferentiated type. The results suggest that these tumours progress to undifferentiated-type carcinomas through loss of E-cadherin function...
  6. pmc Hypermethylation of the hMLH1 gene promoter in solitary and multiple gastric cancers with microsatellite instability
    K Sakata
    Department of Pathology, Yamagata University School of Medicine, Iida nishi 2 2 2, Yamagata 990 9585, Japan
    Br J Cancer 86:564-7. 2002
    ..Hypermethylation of the hMLH1 promoter occurs in non-cancerous gastric mucosa of microsatellite instability-H tumours and may increase the risk of subsequent neoplasia...
  7. ncbi request reprint Molecular characterization of undifferentiated-type gastric carcinoma
    G Tamura
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    Lab Invest 81:593-8. 2001
    ....
  8. ncbi request reprint Mutations in mitochondrial control region DNA in gastric tumours of Japanese patients
    G Tamura
    Department of Pathology, Yamagata University School of Medicine, Japan
    Eur J Cancer 35:316-9. 1999
    ..In summary, mtDNA is mutated in a subset of benign and malignant gastric tumours, but, disruption of the mtDNA repair system appears not to be significantly involved in gastric tumours of Japanese patients...
  9. ncbi request reprint Microsatellite alterations and target gene mutations in the early stages of multiple gastric cancer
    S Ogata
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    J Pathol 194:334-40. 2001
    ..Mutations of E2F-4 are early events in multiple gastric cancer development, occurring even in the intestinal metaplastic mucosa, with mutations of other target genes to follow during cancer progression...
  10. ncbi request reprint Absence of BAT-26 instability in gastric intestinal metaplasia
    Z Jin
    Department of Pathology, Yamagata University School of Medicine, Yamagata, Japan
    Pathol Int 51:473-5. 2001
    ..Although all MSI-H gastric cancer samples showed BAT-26 instability, as assessed using 12 dinucleotide microsatellite markers, BAT-26 instability was not found in the adjacent intestinal metaplastic mucosa in any of the samples...
  11. pmc Frequent loss of expression without sequence mutations of the DCC gene in primary gastric cancer
    K Sato
    Departments of Pathology, Surgery, Yamagata University School of Medicine, 2 2 2 Iida nishi, Yamagata, 990 9585, Japan
    Br J Cancer 85:199-203. 2001
    ..01). These results indicate that DCC is frequently silenced, probably by epigenetic mechanisms instead of sequence mutations in gastric cancer...